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Items: 1 to 20 of 43

1.

Mitochondrial Dysfunction Plus High-Sugar Diet Provokes a Metabolic Crisis That Inhibits Growth.

Kemppainen E, George J, Garipler G, Tuomela T, Kiviranta E, Soga T, Dunn CD, Jacobs HT.

PLoS One. 2016 Jan 26;11(1):e0145836. doi: 10.1371/journal.pone.0145836. eCollection 2016. Erratum in: PLoS One. 2016;11(3):e0151421.

2.

Drosophila sodium channel mutations: Contributions to seizure-susceptibility.

Kroll JR, Saras A, Tanouye MA.

Exp Neurol. 2015 Dec;274(Pt A):80-7. doi: 10.1016/j.expneurol.2015.06.018. Epub 2015 Jun 18. Review.

PMID:
26093037
3.

The alternative oxidase AOX does not rescue the phenotype of tko25t mutant flies.

Kemppainen KK, Kemppainen E, Jacobs HT.

G3 (Bethesda). 2014 Aug 21;4(10):2013-21. doi: 10.1534/g3.114.013946.

4.

Phenotypic rescue of a Drosophila model of mitochondrial ANT1 disease.

Vartiainen S, Chen S, George J, Tuomela T, Luoto KR, O'Dell KM, Jacobs HT.

Dis Model Mech. 2014 Jun;7(6):635-48. doi: 10.1242/dmm.016527. Epub 2014 May 8.

5.

A low-cost method for analyzing seizure-like activity and movement in Drosophila.

Stone B, Burke B, Pathakamuri J, Coleman J, Kuebler D.

J Vis Exp. 2014 Feb 19;(84):e51460. doi: 10.3791/51460.

6.

Drosophila as a model for intractable epilepsy: gilgamesh suppresses seizures in para(bss1) heterozygote flies.

Howlett IC, Rusan ZM, Parker L, Tanouye MA.

G3 (Bethesda). 2013 Aug 7;3(8):1399-407. doi: 10.1534/g3.113.006130.

7.

Drosophila clueless is highly expressed in larval neuroblasts, affects mitochondrial localization and suppresses mitochondrial oxidative damage.

Sen A, Damm VT, Cox RT.

PLoS One. 2013;8(1):e54283. doi: 10.1371/journal.pone.0054283. Epub 2013 Jan 16.

8.

A knock-in model of human epilepsy in Drosophila reveals a novel cellular mechanism associated with heat-induced seizure.

Sun L, Gilligan J, Staber C, Schutte RJ, Nguyen V, O'Dowd DK, Reenan R.

J Neurosci. 2012 Oct 10;32(41):14145-55. doi: 10.1523/JNEUROSCI.2932-12.2012.

9.

Probing mechanisms that underlie human neurodegenerative diseases in Drosophila.

Jaiswal M, Sandoval H, Zhang K, Bayat V, Bellen HJ.

Annu Rev Genet. 2012;46:371-96. doi: 10.1146/annurev-genet-110711-155456. Epub 2012 Sep 4. Review.

10.

A novel Drosophila SOD2 mutant demonstrates a role for mitochondrial ROS in neurodevelopment and disease.

Celotto AM, Liu Z, Vandemark AP, Palladino MJ.

Brain Behav. 2012 Jul;2(4):424-34. doi: 10.1002/brb3.73. Epub 2012 Jun 25.

11.

Mechanical and temperature stressor-induced seizure-and-paralysis behaviors in Drosophila bang-sensitive mutants.

Burg MG, Wu CF.

J Neurogenet. 2012 Jun;26(2):189-97. doi: 10.3109/01677063.2012.690011. Epub 2012 Jun 20.

12.

Voltage-gated potassium channels and the diversity of electrical signalling.

Jan LY, Jan YN.

J Physiol. 2012 Jun 1;590(11):2591-9. doi: 10.1113/jphysiol.2011.224212. Epub 2012 Mar 19. Review.

13.

Seizure and epilepsy: studies of seizure disorders in Drosophila.

Parker L, Howlett IC, Rusan ZM, Tanouye MA.

Int Rev Neurobiol. 2011;99:1-21. doi: 10.1016/B978-0-12-387003-2.00001-X. Review.

14.

Drosophila as a model for epilepsy: bss is a gain-of-function mutation in the para sodium channel gene that leads to seizures.

Parker L, Padilla M, Du Y, Dong K, Tanouye MA.

Genetics. 2011 Feb;187(2):523-34. doi: 10.1534/genetics.110.123299. Epub 2010 Nov 29.

15.

Bacteria, yeast, worms, and flies: exploiting simple model organisms to investigate human mitochondrial diseases.

Rea SL, Graham BH, Nakamaru-Ogiso E, Kar A, Falk MJ.

Dev Disabil Res Rev. 2010;16(2):200-18. doi: 10.1002/ddrr.114. Review.

16.

Modeling mitochondrial encephalomyopathy in Drosophila.

Palladino MJ.

Neurobiol Dis. 2010 Oct;40(1):40-5. doi: 10.1016/j.nbd.2010.05.009. Epub 2010 May 21. Review.

17.

Neurologic dysfunction and male infertility in Drosophila porin mutants: a new model for mitochondrial dysfunction and disease.

Graham BH, Li Z, Alesii EP, Versteken P, Lee C, Wang J, Craigen WJ.

J Biol Chem. 2010 Apr 9;285(15):11143-53. doi: 10.1074/jbc.M109.080317. Epub 2010 Jan 28.

18.

Gene expression in a Drosophila model of mitochondrial disease.

Fernández-Ayala DJ, Chen S, Kemppainen E, O'Dell KM, Jacobs HT.

PLoS One. 2010 Jan 6;5(1):e8549. doi: 10.1371/journal.pone.0008549.

19.

Clueless, a conserved Drosophila gene required for mitochondrial subcellular localization, interacts genetically with parkin.

Cox RT, Spradling AC.

Dis Model Mech. 2009 Sep-Oct;2(9-10):490-9. doi: 10.1242/dmm.002378. Epub 2009 Jul 28.

20.

Mutation of Drosophila focal adhesion kinase induces bang-sensitive behavior and disrupts glial function, axonal conduction and synaptic transmission.

Ueda A, Grabbe C, Lee J, Lee J, Palmer RH, Wu CF.

Eur J Neurosci. 2008 Jun;27(11):2860-70. doi: 10.1111/j.1460-9568.2008.06252.x. Epub 2008 Jun 6.

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