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Items: 18


Pharmacogenetics of analgesic drugs.

Cregg R, Russo G, Gubbay A, Branford R, Sato H.

Br J Pain. 2013 Nov;7(4):189-208. doi: 10.1177/2049463713507439.


Primary erythromelalgia: a review.

Tang Z, Chen Z, Tang B, Jiang H.

Orphanet J Rare Dis. 2015 Sep 30;10:127. doi: 10.1186/s13023-015-0347-1.


Carbamazepine potentiates the effectiveness of morphine in a rodent model of neuropathic pain.

Due MR, Yang XF, Allette YM, Randolph AL, Ripsch MS, Wilson SM, Dustrude ET, Khanna R, White FA.

PLoS One. 2014 Sep 15;9(9):e107399. doi: 10.1371/journal.pone.0107399. eCollection 2014.


Mexiletine as a treatment for primary erythromelalgia: normalization of biophysical properties of mutant L858F NaV 1.7 sodium channels.

Cregg R, Cox JJ, Bennett DL, Wood JN, Werdehausen R.

Br J Pharmacol. 2014 Oct;171(19):4455-63. doi: 10.1111/bph.12788. Epub 2014 Aug 29.


An Egyptian child with erythromelalgia responding to a new line of treatment: a case report and review of the literature.

Al-Minshawy SM, El-Mazary AA.

J Med Case Rep. 2014 Feb 25;8:69. doi: 10.1186/1752-1947-8-69.


Guest editorial: Opportunities in rehabilitation research.

Ommaya AK, Adams KM, Allman RM, Collins EG, Cooper RA, Dixon CE, Fishman PS, Henry JA, Kardon R, Kerns RD, Kupersmith J, Lo A, Macko R, McArdle R, McGlinchey RE, McNeil MR, O'Toole TP, Peckham PH, Tuszynski MH, Waxman SG, Wittenberg GF.

J Rehabil Res Dev. 2013;50(6):vii-xxxii. doi: 10.1682/JRRD.2012.09.0167. No abstract available.


Genomics of pain in osteoarthritis.

Thakur M, Dawes JM, McMahon SB.

Osteoarthritis Cartilage. 2013 Sep;21(9):1374-82. doi: 10.1016/j.joca.2013.06.010. Review.


A residue in the transmembrane segment 6 of domain I in insect and mammalian sodium channels regulate differential sensitivities to pyrethroid insecticides.

Oliveira EE, Du Y, Nomura Y, Dong K.

Neurotoxicology. 2013 Sep;38:42-50. doi: 10.1016/j.neuro.2013.06.001. Epub 2013 Jun 10.


A novel SCN9A mutation responsible for primary erythromelalgia and is resistant to the treatment of sodium channel blockers.

Wu MT, Huang PY, Yen CT, Chen CC, Lee MJ.

PLoS One. 2013;8(1):e55212. doi: 10.1371/journal.pone.0055212. Epub 2013 Jan 31.


Structural modelling and mutant cycle analysis predict pharmacoresponsiveness of a Na(V)1.7 mutant channel.

Yang Y, Dib-Hajj SD, Zhang J, Zhang Y, Tyrrell L, Estacion M, Waxman SG.

Nat Commun. 2012;3:1186. doi: 10.1038/ncomms2184.


Novel insights on diagnosis, cause and treatment of diabetic neuropathy: focus on painful diabetic neuropathy.

Tavakoli M, Asghar O, Alam U, Petropoulos IN, Fadavi H, Malik RA.

Ther Adv Endocrinol Metab. 2010 Apr;1(2):69-88. doi: 10.1177/2042018810370954.


Gain-of-function Nav1.8 mutations in painful neuropathy.

Faber CG, Lauria G, Merkies IS, Cheng X, Han C, Ahn HS, Persson AK, Hoeijmakers JG, Gerrits MM, Pierro T, Lombardi R, Kapetis D, Dib-Hajj SD, Waxman SG.

Proc Natl Acad Sci U S A. 2012 Nov 20;109(47):19444-9. doi: 10.1073/pnas.1216080109. Epub 2012 Oct 31.


Neurological perspectives on voltage-gated sodium channels.

Eijkelkamp N, Linley JE, Baker MD, Minett MS, Cregg R, Werdehausen R, Rugiero F, Wood JN.

Brain. 2012 Sep;135(Pt 9):2585-612. doi: 10.1093/brain/aws225. Review.


Splice variants of Na(V)1.7 sodium channels have distinct β subunit-dependent biophysical properties.

Farmer C, Cox JJ, Fletcher EV, Woods CG, Wood JN, Schorge S.

PLoS One. 2012;7(7):e41750. doi: 10.1371/journal.pone.0041750. Epub 2012 Jul 24.


Sodium channels, the electrogenisome and the electrogenistat: lessons and questions from the clinic.

Waxman SG.

J Physiol. 2012 Jun 1;590(Pt 11):2601-12. doi: 10.1113/jphysiol.2012.228460. Epub 2012 Mar 12. Review.


Extending the clinical spectrum of pain channelopathies.

Houlden H.

Brain. 2012 Feb;135(Pt 2):313-6. doi: 10.1093/brain/aws007. No abstract available.


Inhibition of Navβ4 peptide-mediated resurgent sodium currents in Nav1.7 channels by carbamazepine, riluzole, and anandamide.

Theile JW, Cummins TR.

Mol Pharmacol. 2011 Oct;80(4):724-34. doi: 10.1124/mol.111.072751. Epub 2011 Jul 25.


Mutations at opposite ends of the DIII/S4-S5 linker of sodium channel Na V 1.7 produce distinct pain disorders.

Cheng X, Dib-Hajj SD, Tyrrell L, Wright DA, Fischer TZ, Waxman SG.

Mol Pain. 2010 Apr 29;6:24. doi: 10.1186/1744-8069-6-24.

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