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Cited In for PubMed (Select 18001293)


Conditional Knock-out Reveals a Requirement for O-Linked N-Acetylglucosaminase (O-GlcNAcase) in Metabolic Homeostasis.

Keembiyehetty C, Love DC, Harwood KR, Gavrilova O, Comly ME, Hanover JA.

J Biol Chem. 2015 Mar 13;290(11):7097-113. doi: 10.1074/jbc.M114.617779. Epub 2015 Jan 16.


Mitohormesis: Promoting Health and Lifespan by Increased Levels of Reactive Oxygen Species (ROS).

Ristow M, Schmeisser K.

Dose Response. 2014 Jan 31;12(2):288-341. doi: 10.2203/dose-response.13-035.Ristow. eCollection 2014 May.


Targeting tissue-specific metabolic signaling pathways in aging: the promise and limitations.

Hu F, Liu F.

Protein Cell. 2014 Jan;5(1):21-35. doi: 10.1007/s13238-013-0002-3. Epub 2014 Jan 29. Review.


Insulin signaling, resistance, and the metabolic syndrome: insights from mouse models into disease mechanisms.

Guo S.

J Endocrinol. 2014 Jan 8;220(2):T1-T23. doi: 10.1530/JOE-13-0327. Print 2014 Feb. Review.


Aging on a different scale--chronological versus pathology-related aging.

Melis JP, Jonker MJ, Vijg J, Hoeijmakers JH, Breit TM, van Steeg H.

Aging (Albany NY). 2013 Oct;5(10):782-8.


FGT-1 is the major glucose transporter in C. elegans and is central to aging pathways.

Feng Y, Williams BG, Koumanov F, Wolstenholme AJ, Holman GD.

Biochem J. 2013 Dec 1;456(2):219-29. doi: 10.1042/BJ20131101.


The role of mitochondria in aging.

Bratic A, Larsson NG.

J Clin Invest. 2013 Mar;123(3):951-7. doi: 10.1172/JCI64125. Epub 2013 Mar 1. Review.


Metabolic characteristics of long-lived mice.

Bartke A, Westbrook R.

Front Genet. 2012 Dec 13;3:288. doi: 10.3389/fgene.2012.00288. eCollection 2012.


Increased mitochondrial activity in BMP7-treated brown adipocytes, due to increased CPT1- and CD36-mediated fatty acid uptake.

Townsend KL, An D, Lynes MD, Huang TL, Zhang H, Goodyear LJ, Tseng YH.

Antioxid Redox Signal. 2013 Jul 20;19(3):243-57. doi: 10.1089/ars.2012.4536. Epub 2012 Oct 9.


Mitochondrial energy metabolism and redox signaling in brain aging and neurodegeneration.

Yin F, Boveris A, Cadenas E.

Antioxid Redox Signal. 2014 Jan 10;20(2):353-71. doi: 10.1089/ars.2012.4774. Epub 2012 Sep 5. Review.


Impaired thermogenesis and adipose tissue development in mice with fat-specific disruption of insulin and IGF-1 signalling.

Boucher J, Mori MA, Lee KY, Smyth G, Liew CW, Macotela Y, Rourk M, Bluher M, Russell SJ, Kahn CR.

Nat Commun. 2012 Jun 12;3:902. doi: 10.1038/ncomms1905.


Comparative endocrinology of aging and longevity regulation.

Allard JB, Duan C.

Front Endocrinol (Lausanne). 2011 Nov 23;2:75. doi: 10.3389/fendo.2011.00075. eCollection 2011.


GIT2 acts as a potential keystone protein in functional hypothalamic networks associated with age-related phenotypic changes in rats.

Chadwick W, Martin B, Chapter MC, Park SS, Wang L, Daimon CM, Brenneman R, Maudsley S.

PLoS One. 2012;7(5):e36975. doi: 10.1371/journal.pone.0036975. Epub 2012 May 14.


Insulin, IGF-1 and longevity.

van Heemst D.

Aging Dis. 2010 Oct;1(2):147-57. Epub 2010 Aug 26.


Deficiency of the lipid synthesis enzyme, DGAT1, extends longevity in mice.

Streeper RS, Grueter CA, Salomonis N, Cases S, Levin MC, Koliwad SK, Zhou P, Hirschey MD, Verdin E, Farese RV Jr.

Aging (Albany NY). 2012 Jan;4(1):13-27.


Modulation of longevity and tissue homeostasis by the Drosophila PGC-1 homolog.

Rera M, Bahadorani S, Cho J, Koehler CL, Ulgherait M, Hur JH, Ansari WS, Lo T Jr, Jones DL, Walker DW.

Cell Metab. 2011 Nov 2;14(5):623-34. doi: 10.1016/j.cmet.2011.09.013.


The differential role of Hif1β/Arnt and the hypoxic response in adipose function, fibrosis, and inflammation.

Lee KY, Gesta S, Boucher J, Wang XL, Kahn CR.

Cell Metab. 2011 Oct 5;14(4):491-503. doi: 10.1016/j.cmet.2011.08.006.


Dissociation of diabetes and obesity in mice lacking orphan nuclear receptor small heterodimer partner.

Park YJ, Kim SC, Kim J, Anakk S, Lee JM, Tseng HT, Yechoor V, Park J, Choi JS, Jang HC, Lee KU, Novak CM, Moore DD, Lee YK.

J Lipid Res. 2011 Dec;52(12):2234-44. doi: 10.1194/jlr.M016048. Epub 2011 Sep 23.

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