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Results: 12

Cited In for PubMed (Select 17699523)

1.

Impact of RTN3 deficiency on expression of BACE1 and amyloid deposition.

Shi Q, Ge Y, Sharoar MG, He W, Xiang R, Zhang Z, Hu X, Yan R.

J Neurosci. 2014 Oct 15;34(42):13954-62. doi: 10.1523/JNEUROSCI.1588-14.2014.

2.

Glutamate provides a key structural contact between reticulon-4 (Nogo-66) and phosphocholine.

Alhoshani A, Vithayathil R, Bandong J, Chrunyk KM, Moreno GO, Weiss GA, Cocco MJ.

Biochim Biophys Acta. 2014 Sep;1838(9):2350-6. doi: 10.1016/j.bbamem.2014.05.021. Epub 2014 May 24.

PMID:
24863057
3.

The role of reticulons in neurodegenerative diseases.

Chiurchi├╣ V, Maccarrone M, Orlacchio A.

Neuromolecular Med. 2014 Mar;16(1):3-15. doi: 10.1007/s12017-013-8271-9. Epub 2013 Nov 12. Review.

4.

Increased expression of reticulon 3 in neurons leads to reduced axonal transport of ╬▓ site amyloid precursor protein-cleaving enzyme 1.

Deng M, He W, Tan Y, Han H, Hu X, Xia K, Zhang Z, Yan R.

J Biol Chem. 2013 Oct 18;288(42):30236-45. doi: 10.1074/jbc.M113.480079. Epub 2013 Sep 4.

5.

Preventing formation of reticulon 3 immunoreactive dystrophic neurites improves cognitive function in mice.

Shi Q, Prior M, Zhou X, Tang X, He W, Hu X, Yan R.

J Neurosci. 2013 Feb 13;33(7):3059-66. doi: 10.1523/JNEUROSCI.2445-12.2013.

6.

Interaction between amyloid precursor protein and Nogo receptors regulates amyloid deposition.

Zhou X, Hu X, He W, Tang X, Shi Q, Zhang Z, Yan R.

FASEB J. 2011 Sep;25(9):3146-56. doi: 10.1096/fj.11-184325. Epub 2011 Jun 13.

7.

Widespread distribution of reticulon-3 in various neurodegenerative diseases.

Heath JE, Siedlak SL, Zhu X, Lee HG, Thakur A, Yan R, Perry G, Smith MA, Castellani RJ.

Neuropathology. 2010 Dec;30(6):574-9. doi: 10.1111/j.1440-1789.2010.01107.x.

8.

RTN/Nogo in forming Alzheimer's neuritic plaques.

Prior M, Shi Q, Hu X, He W, Levey A, Yan R.

Neurosci Biobehav Rev. 2010 Jul;34(8):1201-6. doi: 10.1016/j.neubiorev.2010.01.017. Epub 2010 Feb 6.

9.

Reduced amyloid deposition in mice overexpressing RTN3 is adversely affected by preformed dystrophic neurites.

Shi Q, Prior M, He W, Tang X, Hu X, Yan R.

J Neurosci. 2009 Jul 22;29(29):9163-73. doi: 10.1523/JNEUROSCI.5741-08.2009.

10.

The occurrence of aging-dependent reticulon 3 immunoreactive dystrophic neurites decreases cognitive function.

Shi Q, Hu X, Prior M, Yan R.

J Neurosci. 2009 Apr 22;29(16):5108-15. doi: 10.1523/JNEUROSCI.5887-08.2009.

11.

Changes in gamma-secretase activity and specificity caused by the introduction of consensus aspartyl protease active motif in Presenilin 1.

Carter DB, Dunn E, Pauley AM, McKinley DD, Fleck TJ, Ellerbrook BR, Stratman NC, Zhou X, Himes CS, Nye JS, Tomasselli A, Yan R.

Mol Neurodegener. 2008 May 12;3:6. doi: 10.1186/1750-1326-3-6.

12.

The reticulon and DP1/Yop1p proteins form immobile oligomers in the tubular endoplasmic reticulum.

Shibata Y, Voss C, Rist JM, Hu J, Rapoport TA, Prinz WA, Voeltz GK.

J Biol Chem. 2008 Jul 4;283(27):18892-904. doi: 10.1074/jbc.M800986200. Epub 2008 Apr 28.

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