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Items: 1 to 20 of 78

1.

ABRAXAS (FAM175A) and Breast Cancer Susceptibility: No Evidence of Association in the Breast Cancer Family Registry.

Renault AL, Lesueur F, Coulombe Y, Gobeil S, Soucy P, Hamdi Y, Desjardins S, Le Calvez-Kelm F, Vallée M, Voegele C; Breast Cancer Family Registry, Hopper JL, Andrulis IL, Southey MC, John EM, Masson JY, Tavtigian SV, Simard J.

PLoS One. 2016 Jun 7;11(6):e0156820. doi: 10.1371/journal.pone.0156820. eCollection 2016.

2.

Genetic evaluation of BRCA1 associated a complex genes with triple-negative breast cancer susceptibility in Chinese women.

Ling H, Li S, Wu Y, Zheng YZ, Qiao F, Yao L, Cao ZG, Ye FG, Wu J, Hu X, Wang B, Shao ZM.

Oncotarget. 2016 Mar 1;7(9):9759-72. doi: 10.18632/oncotarget.7112.

3.

Tankyrases Promote Homologous Recombination and Check Point Activation in Response to DSBs.

Nagy Z, Kalousi A, Furst A, Koch M, Fischer B, Soutoglou E.

PLoS Genet. 2016 Feb 4;12(2):e1005791. doi: 10.1371/journal.pgen.1005791. eCollection 2016 Feb.

4.

TRAIP/RNF206 is required for recruitment of RAP80 to sites of DNA damage.

Soo Lee N, Jin Chung H, Kim HJ, Yun Lee S, Ji JH, Seo Y, Hun Han S, Choi M, Yun M, Lee SG, Myung K, Kim Y, Chul Kang H, Kim H.

Nat Commun. 2016 Jan 19;7:10463. doi: 10.1038/ncomms10463.

5.

Structure of BRCA1-BRCT/Abraxas Complex Reveals Phosphorylation-Dependent BRCT Dimerization at DNA Damage Sites.

Wu Q, Paul A, Su D, Mehmood S, Foo TK, Ochi T, Bunting EL, Xia B, Robinson CV, Wang B, Blundell TL.

Mol Cell. 2016 Feb 4;61(3):434-48. doi: 10.1016/j.molcel.2015.12.017. Epub 2016 Jan 14.

6.

RAP80 regulates epithelial-mesenchymal transition related with metastasis and malignancy of cancer.

Park SY, Korm S, Chung HJ, Choi SJ, Jang JJ, Cho S, Lim YT, Kim H, Lee JY.

Cancer Sci. 2016 Mar;107(3):267-73. doi: 10.1111/cas.12877. Epub 2016 Feb 10.

7.

Role of Deubiquitinating Enzymes in DNA Repair.

Kee Y, Huang TT.

Mol Cell Biol. 2015 Dec 7;36(4):524-44. doi: 10.1128/MCB.00847-15. Review.

8.

BRCA1 Is Required for Maintenance of Phospho-Chk1 and G2/M Arrest during DNA Cross-Link Repair in DT40 Cells.

Draga M, Madgett EB, Vandenberg CJ, du Plessis D, Kaufmann A, Werler P, Chakraborty P, Lowndes NF, Hiom K.

Mol Cell Biol. 2015 Nov;35(22):3829-40. doi: 10.1128/MCB.01497-14. Epub 2015 Aug 31.

9.

Mutation of the BRCA1 SQ-cluster results in aberrant mitosis, reduced homologous recombination, and a compensatory increase in non-homologous end joining.

Beckta JM, Dever SM, Gnawali N, Khalil A, Sule A, Golding SE, Rosenberg E, Narayanan A, Kehn-Hall K, Xu B, Povirk LF, Valerie K.

Oncotarget. 2015 Sep 29;6(29):27674-87. doi: 10.18632/oncotarget.4876.

10.

CCDC34 is up-regulated in bladder cancer and regulates bladder cancer cell proliferation, apoptosis and migration.

Gong Y, Qiu W, Ning X, Yang X, Liu L, Wang Z, Lin J, Li X, Guo Y.

Oncotarget. 2015 Sep 22;6(28):25856-67. doi: 10.18632/oncotarget.4624.

11.

Irreversible binding of an anticancer compound (BI-94) to plasma proteins.

Gautam N, Thakare R, Rana S, Natarajan A, Alnouti Y.

Xenobiotica. 2015;45(10):858-73. doi: 10.3109/00498254.2015.1025250. Epub 2015 Apr 14.

12.

Structural maintenance of chromosomes flexible hinge domain containing 1 (SMCHD1) promotes non-homologous end joining and inhibits homologous recombination repair upon DNA damage.

Tang M, Li Y, Zhang X, Deng T, Zhou Z, Ma W, Songyang Z.

J Biol Chem. 2014 Dec 5;289(49):34024-32. doi: 10.1074/jbc.M114.601179. Epub 2014 Oct 7.

13.

ABRO1 suppresses tumourigenesis and regulates the DNA damage response by stabilizing p53.

Zhang J, Cao M, Dong J, Li C, Xu W, Zhan Y, Wang X, Yu M, Ge C, Ge Z, Yang X.

Nat Commun. 2014 Oct 6;5:5059. doi: 10.1038/ncomms6059.

14.

PARP1-driven poly-ADP-ribosylation regulates BRCA1 function in homologous recombination-mediated DNA repair.

Hu Y, Petit SA, Ficarro SB, Toomire KJ, Xie A, Lim E, Cao SA, Park E, Eck MJ, Scully R, Brown M, Marto JA, Livingston DM.

Cancer Discov. 2014 Dec;4(12):1430-47. doi: 10.1158/2159-8290.CD-13-0891. Epub 2014 Sep 24.

15.

The role of poly(ADP-ribosyl)ation in DNA damage response and cancer chemotherapy.

Li M, Yu X.

Oncogene. 2015 Jun;34(26):3349-56. doi: 10.1038/onc.2014.295. Epub 2014 Sep 15. Review.

16.

The BRCA1-interacting protein Abraxas is required for genomic stability and tumor suppression.

Castillo A, Paul A, Sun B, Huang TH, Wang Y, Yazinski SA, Tyler J, Li L, You MJ, Zou L, Yao J, Wang B.

Cell Rep. 2014 Aug 7;8(3):807-17. doi: 10.1016/j.celrep.2014.06.050. Epub 2014 Jul 24.

17.

PALB2: the hub of a network of tumor suppressors involved in DNA damage responses.

Park JY, Zhang F, Andreassen PR.

Biochim Biophys Acta. 2014 Aug;1846(1):263-75. doi: 10.1016/j.bbcan.2014.06.003. Epub 2014 Jul 3. Review.

18.

BRCA1 and Its Network of Interacting Partners.

Christou CM, Kyriacou K.

Biology (Basel). 2013 Jan 2;2(1):40-63. doi: 10.3390/biology2010040.

19.

NUSAP1 influences the DNA damage response by controlling BRCA1 protein levels.

Kotian S, Banerjee T, Lockhart A, Huang K, Catalyurek UV, Parvin JD.

Cancer Biol Ther. 2014 May;15(5):533-43. doi: 10.4161/cbt.28019. Epub 2014 Feb 12.

20.

The breast cancer susceptibility genes (BRCA) in breast and ovarian cancers.

Paul A, Paul S.

Front Biosci (Landmark Ed). 2014 Jan 1;19:605-18. Review.

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