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Results: 1 to 20 of 249

Cited In for PubMed (Select 15454078)


Wrecked regulation of intrinsically disordered proteins in diseases: pathogenicity of deregulated regulators.

Uversky VN.

Front Mol Biosci. 2014 Jul 25;1:6. doi: 10.3389/fmolb.2014.00006. eCollection 2014. Review.


Cav1.2, cell proliferation, and new target in atherosclerosis.

Soldatov NM.

ISRN Biochem. 2013 May 12;2013:463527. doi: 10.1155/2013/463527. eCollection 2013. Review.


Rhythm genes sing more than one tune: noncanonical functions of cardiac ion channels.

Akhirome E, Jay PY.

Circ Arrhythm Electrophysiol. 2015 Apr;8(2):261-2. doi: 10.1161/CIRCEP.115.002834. No abstract available.


Graded Ca²⁺/calmodulin-dependent coupling of voltage-gated CaV1.2 channels.

Dixon RE, Moreno CM, Yuan C, Opitz-Araya X, Binder MD, Navedo MF, Santana LF.

Elife. 2015 Feb 25;4. doi: 10.7554/eLife.05608.


CACNA1D de novo mutations in autism spectrum disorders activate Cav1.3 L-type calcium channels.

Pinggera A, Lieb A, Benedetti B, Lampert M, Monteleone S, Liedl KR, Tuluc P, Striessnig J.

Biol Psychiatry. 2015 May 1;77(9):816-22. doi: 10.1016/j.biopsych.2014.11.020. Epub 2014 Dec 8.


Control of cardiac repolarization by phosphoinositide 3-kinase signaling to ion channels.

Ballou LM, Lin RZ, Cohen IS.

Circ Res. 2015 Jan 2;116(1):127-37. doi: 10.1161/CIRCRESAHA.116.303975. Review.


Calcium signaling regulates ventricular hypertrophy during development independent of contraction or blood flow.

Andersen ND, Ramachandran KV, Bao MM, Kirby ML, Pitt GS, Hutson MR.

J Mol Cell Cardiol. 2015 Mar;80:1-9. doi: 10.1016/j.yjmcc.2014.12.016. Epub 2014 Dec 20.


Functional characterization of CaVα2δ mutations associated with sudden cardiac death.

Bourdin B, Shakeri B, Tétreault MP, Sauvé R, Lesage S, Parent L.

J Biol Chem. 2015 Jan 30;290(5):2854-69. doi: 10.1074/jbc.M114.597930. Epub 2014 Dec 19.


Genetics studies indicate that neural induction and early neuronal maturation are disturbed in autism.

Casanova EL, Casanova MF.

Front Cell Neurosci. 2014 Nov 19;8:397. doi: 10.3389/fncel.2014.00397. eCollection 2014.


Recessive cardiac phenotypes in induced pluripotent stem cell models of Jervell and Lange-Nielsen syndrome: disease mechanisms and pharmacological rescue.

Zhang M, D'Aniello C, Verkerk AO, Wrobel E, Frank S, Ward-van Oostwaard D, Piccini I, Freund C, Rao J, Seebohm G, Atsma DE, Schulze-Bahr E, Mummery CL, Greber B, Bellin M.

Proc Natl Acad Sci U S A. 2014 Dec 16;111(50):E5383-92. doi: 10.1073/pnas.1419553111. Epub 2014 Dec 1.


MiRNA-1/133a clusters regulate adrenergic control of cardiac repolarization.

Besser J, Malan D, Wystub K, Bachmann A, Wietelmann A, Sasse P, Fleischmann BK, Braun T, Boettger T.

PLoS One. 2014 Nov 21;9(11):e113449. doi: 10.1371/journal.pone.0113449. eCollection 2014.


DUSP4 regulates neuronal differentiation and calcium homeostasis by modulating ERK1/2 phosphorylation.

Kim SY, Han YM, Oh M, Kim WK, Oh KJ, Lee SC, Bae KH, Han BS.

Stem Cells Dev. 2015 Mar 15;24(6):686-700. doi: 10.1089/scd.2014.0434. Epub 2014 Dec 23.


Modeling non-syndromic autism and the impact of TRPC6 disruption in human neurons.

Griesi-Oliveira K, Acab A, Gupta AR, Sunaga DY, Chailangkarn T, Nicol X, Nunez Y, Walker MF, Murdoch JD, Sanders SJ, Fernandez TV, Ji W, Lifton RP, Vadasz E, Dietrich A, Pradhan D, Song H, Ming GL, Gu X, Haddad G, Marchetto MC, Spitzer N, Passos-Bueno MR, State MW, Muotri AR.

Mol Psychiatry. 2014 Nov 11. doi: 10.1038/mp.2014.141. [Epub ahead of print]


Alteration in basal and depolarization induced transcriptional network in iPSC derived neurons from Timothy syndrome.

Tian Y, Voineagu I, Paşca SP, Won H, Chandran V, Horvath S, Dolmetsch RE, Geschwind DH.

Genome Med. 2014 Oct 10;6(10):75. doi: 10.1186/s13073-014-0075-5. eCollection 2014.


Association between autism spectrum disorder in individuals with velocardiofacial (22q11.2 deletion) syndrome and PRODH and COMT genotypes.

Radoeva PD, Coman IL, Salazar CA, Gentile KL, Higgins AM, Middleton FA, Antshel KM, Fremont W, Shprintzen RJ, Morrow BE, Kates WR.

Psychiatr Genet. 2014 Dec;24(6):269-72. doi: 10.1097/YPG.0000000000000062.


Anchored protein kinase A signalling in cardiac cellular electrophysiology.

Soni S, Scholten A, Vos MA, van Veen TA.

J Cell Mol Med. 2014 Nov;18(11):2135-46. doi: 10.1111/jcmm.12365. Epub 2014 Sep 12.


Ion channel engineering: perspectives and strategies.

Subramanyam P, Colecraft HM.

J Mol Biol. 2015 Jan 16;427(1):190-204. doi: 10.1016/j.jmb.2014.09.001. Epub 2014 Sep 7. Review.


A CACNA1C variant associated with reduced voltage-dependent inactivation, increased CaV1.2 channel window current, and arrhythmogenesis.

Hennessey JA, Boczek NJ, Jiang YH, Miller JD, Patrick W, Pfeiffer R, Sutphin BS, Tester DJ, Barajas-Martinez H, Ackerman MJ, Antzelevitch C, Kanter R, Pitt GS.

PLoS One. 2014 Sep 3;9(9):e106982. doi: 10.1371/journal.pone.0106982. eCollection 2014.


Toward a hierarchy of mechanisms in CaMKII-mediated arrhythmia.

Vincent KP, McCulloch AD, Edwards AG.

Front Pharmacol. 2014 May 16;5:110. doi: 10.3389/fphar.2014.00110. eCollection 2014. Review.


Ca(2+) current facilitation is CaMKII-dependent and has arrhythmogenic consequences.

Bers DM, Morotti S.

Front Pharmacol. 2014 Jun 17;5:144. doi: 10.3389/fphar.2014.00144. eCollection 2014. Review.

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