Similar articles for PMID: 8713451

Year	Number of Results
1985	1
1987	3
1988	5
1989	3
1990	8
1991	6
1992	8
1993	7
1994	8
1995	20
1996	18
1997	17
1998	15
1999	22
2000	14
2001	15
2002	7
2003	13
2004	4
2005	7
2006	5
2007	3
2008	12
2009	15
2010	12
2011	9
2012	7
2013	6
2014	7
2015	2
2017	3
2018	3
2019	8
2020	8
2021	10
2022	7
2024	0

1

Characterization of excitatory amino acid neurotoxicity in N-methyl-D-aspartate receptor-deficient mouse cortical neuronal cells.

Tokita Y, Bessho Y, Masu M, Nakamura K, Nakao K, Katsuki M, Nakanishi S. Tokita Y, et al. Eur J Neurosci. 1996 Jan;8(1):69-78. doi: 10.1111/j.1460-9568.1996.tb01168.x. Eur J Neurosci. 1996. PMID: 8713451

2

Cortical and striatal neuronal cultures of the same embryonic origin show intrinsic differences in glutamate receptor expression and vulnerability to excitotoxicity.

Kovács AD, Cebers G, Cebere A, Moreira T, Liljequist S. Kovács AD, et al. Exp Neurol. 2001 Mar;168(1):47-62. doi: 10.1006/exnr.2000.7576. Exp Neurol. 2001. PMID: 11170720

3

Neurotoxicity of acute glutamate transport blockade depends on coactivation of both NMDA and AMPA/Kainate receptors in organotypic hippocampal cultures.

Vornov JJ, Tasker RC, Park J. Vornov JJ, et al. Exp Neurol. 1995 May;133(1):7-17. doi: 10.1006/exnr.1995.1002. Exp Neurol. 1995. PMID: 7541369 Free article.

4

NMDA and non-NMDA receptor-mediated excitotoxicity are potentiated in cultured striatal neurons by prior chronic depolarization.

Chen Q, Surmeier DJ, Reiner A. Chen Q, et al. Exp Neurol. 1999 Sep;159(1):283-96. doi: 10.1006/exnr.1999.7135. Exp Neurol. 1999. PMID: 10486197

5

Involvement of NMDA-receptor in kainate-induced neurotoxicity in cultured fetal retinal neurons.

Zhang S, Kashii S, Yasuyoshi H, Honda Y, Ujihara H, Sasa M, Tamura Y, Akaike A. Zhang S, et al. Graefes Arch Clin Exp Ophthalmol. 2000 Mar;238(3):243-8. doi: 10.1007/s004170050351. Graefes Arch Clin Exp Ophthalmol. 2000. PMID: 10796040

6

Characterization of neuroprotection from excitotoxicity by moderate and profound hypothermia in cultured cortical neurons unmasks a temperature-insensitive component of glutamate neurotoxicity.

Tymianski M, Sattler R, Zabramski JM, Spetzler RF. Tymianski M, et al. J Cereb Blood Flow Metab. 1998 Aug;18(8):848-67. doi: 10.1097/00004647-199808000-00005. J Cereb Blood Flow Metab. 1998. PMID: 9701346

7

N-methyl-D-aspartate receptor-mediated mitochondrial Ca(2+) overload in acute excitotoxic motor neuron death: a mechanism distinct from chronic neurotoxicity after Ca(2+) influx.

Urushitani M, Nakamizo T, Inoue R, Sawada H, Kihara T, Honda K, Akaike A, Shimohama S. Urushitani M, et al. J Neurosci Res. 2001 Mar 1;63(5):377-87. doi: 10.1002/1097-4547(20010301)63:5<377::AID-JNR1032>3.0.CO;2-#. J Neurosci Res. 2001. PMID: 11223912

8

Xenon reduces glutamate-, AMPA-, and kainate-induced membrane currents in cortical neurones.

Dinse A, Föhr KJ, Georgieff M, Beyer C, Bulling A, Weigt HU. Dinse A, et al. Br J Anaesth. 2005 Apr;94(4):479-85. doi: 10.1093/bja/aei080. Epub 2005 Feb 4. Br J Anaesth. 2005. PMID: 15695547 Free article.

9

Evidence that functional glutamate receptors are not expressed on rat or human cerebromicrovascular endothelial cells.

Morley P, Small DL, Murray CL, Mealing GA, Poulter MO, Durkin JP, Stanimirovic DB. Morley P, et al. J Cereb Blood Flow Metab. 1998 Apr;18(4):396-406. doi: 10.1097/00004647-199804000-00008. J Cereb Blood Flow Metab. 1998. PMID: 9538905

10

Domoic acid neurotoxicity in cultured cerebellar granule neurons is mediated predominantly by NMDA receptors that are activated as a consequence of excitatory amino acid release.

Berman FW, Murray TF. Berman FW, et al. J Neurochem. 1997 Aug;69(2):693-703. doi: 10.1046/j.1471-4159.1997.69020693.x. J Neurochem. 1997. PMID: 9231729 Free article.

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