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Items: 1 to 20 of 95

1.

Nε-carboxymethyllysine-mediated endoplasmic reticulum stress promotes endothelial cell injury through Nox4/MKP-3 interaction.

Lee WJ, Sheu WH, Liu SH, Yi YC, Chen WC, Lin SY, Liang KW, Shen CC, Yeh HY, Lin LY, Tsai YC, Tien HR, Lee MR, Yang TJ, Sheu ML.

Free Radic Biol Med. 2014 Sep;74:294-306. doi: 10.1016/j.freeradbiomed.2014.06.015. Epub 2014 Jul 8.

PMID:
25014566
2.

Advanced glycation end product Nε-carboxymethyllysine induces endothelial cell injury: the involvement of SHP-1-regulated VEGFR-2 dephosphorylation.

Liu SH, Sheu WH, Lee MR, Lee WJ, Yi YC, Yang TJ, Jen JF, Pan HC, Shen CC, Chen WB, Tien HR, Sheu ML.

J Pathol. 2013 Jun;230(2):215-27. doi: 10.1002/path.4045. Epub 2013 Mar 12.

PMID:
22553146
3.

Sphingosine-1-phosphate inhibits high glucose-mediated ERK1/2 action in endothelium through induction of MAP kinase phosphatase-3.

Whetzel AM, Bolick DT, Hedrick CC.

Am J Physiol Cell Physiol. 2009 Feb;296(2):C339-45. doi: 10.1152/ajpcell.00293.2008. Epub 2008 Dec 17.

4.

Hepatic extracellular signal-regulated kinase 2 suppresses endoplasmic reticulum stress and protects from oxidative stress and endothelial dysfunction.

Kujiraoka T, Satoh Y, Ayaori M, Shiraishi Y, Arai-Nakaya Y, Hakuno D, Yada H, Kuwada N, Endo S, Isoda K, Adachi T.

J Am Heart Assoc. 2013 Aug 16;2(4):e000361. doi: 10.1161/JAHA.113.000361.

5.

Advanced glycation end-product Nε-carboxymethyl-Lysine accelerates progression of atherosclerotic calcification in diabetes.

Wang Z, Jiang Y, Liu N, Ren L, Zhu Y, An Y, Chen D.

Atherosclerosis. 2012 Apr;221(2):387-96. doi: 10.1016/j.atherosclerosis.2012.01.019. Epub 2012 Jan 13.

PMID:
22305260
6.

Inhibitory cross-talk between the AMPK and ERK pathways mediates endoplasmic reticulum stress-induced insulin resistance in skeletal muscle.

Hwang SL, Jeong YT, Li X, Kim YD, Lu Y, Chang YC, Lee IK, Chang HW.

Br J Pharmacol. 2013 May;169(1):69-81. doi: 10.1111/bph.12124.

7.

A novel mechanism by which SDF-1β protects cardiac cells from palmitate-induced endoplasmic reticulum stress and apoptosis via CXCR7 and AMPK/p38 MAPK-mediated interleukin-6 generation.

Zhao Y, Tan Y, Xi S, Li Y, Li C, Cui J, Yan X, Li X, Wang G, Li W, Cai L.

Diabetes. 2013 Jul;62(7):2545-58. doi: 10.2337/db12-1233. Epub 2013 Feb 19.

8.

Nox4 NADPH oxidase-derived reactive oxygen species, via endogenous carbon monoxide, promote survival of brain endothelial cells during TNF-α-induced apoptosis.

Basuroy S, Tcheranova D, Bhattacharya S, Leffler CW, Parfenova H.

Am J Physiol Cell Physiol. 2011 Feb;300(2):C256-65. doi: 10.1152/ajpcell.00272.2010. Epub 2010 Dec 1.

9.

Modulation of albumin-induced endoplasmic reticulum stress in renal proximal tubule cells by upregulation of mapk phosphatase-1.

Gorostizaga A, Mori Sequeiros García MM, Acquier A, Gomez NV, Maloberti PM, Mendez CF, Paz C.

Chem Biol Interact. 2013 Oct 25;206(1):47-54. doi: 10.1016/j.cbi.2013.08.009. Epub 2013 Aug 29.

PMID:
23994741
10.

Metformin protects endothelial function in diet-induced obese mice by inhibition of endoplasmic reticulum stress through 5' adenosine monophosphate-activated protein kinase-peroxisome proliferator-activated receptor δ pathway.

Cheang WS, Tian XY, Wong WT, Lau CW, Lee SS, Chen ZY, Yao X, Wang N, Huang Y.

Arterioscler Thromb Vasc Biol. 2014 Apr;34(4):830-6. doi: 10.1161/ATVBAHA.113.301938. Epub 2014 Jan 30.

11.

MAPK phosphatase-3 promotes hepatic gluconeogenesis through dephosphorylation of forkhead box O1 in mice.

Wu Z, Jiao P, Huang X, Feng B, Feng Y, Yang S, Hwang P, Du J, Nie Y, Xiao G, Xu H.

J Clin Invest. 2010 Nov;120(11):3901-11. doi: 10.1172/JCI43250.

13.

The alternative crosstalk between RAGE and nitrative thioredoxin inactivation during diabetic myocardial ischemia-reperfusion injury.

Liu Y, Qu Y, Wang R, Ma Y, Xia C, Gao C, Liu J, Lian K, Xu A, Lu X, Sun L, Yang L, Lau WB, Gao E, Koch W, Wang H, Tao L.

Am J Physiol Endocrinol Metab. 2012 Oct 1;303(7):E841-52. doi: 10.1152/ajpendo.00075.2012. Epub 2012 Jul 24.

14.
15.

Critical role of Nox4-based NADPH oxidase in glucose-induced oxidative stress in the kidney: implications in type 2 diabetic nephropathy.

Sedeek M, Callera G, Montezano A, Gutsol A, Heitz F, Szyndralewiez C, Page P, Kennedy CR, Burns KD, Touyz RM, Hébert RL.

Am J Physiol Renal Physiol. 2010 Dec;299(6):F1348-58. doi: 10.1152/ajprenal.00028.2010. Epub 2010 Jul 14.

16.

Nox4 is a protective reactive oxygen species generating vascular NADPH oxidase.

Schröder K, Zhang M, Benkhoff S, Mieth A, Pliquett R, Kosowski J, Kruse C, Luedike P, Michaelis UR, Weissmann N, Dimmeler S, Shah AM, Brandes RP.

Circ Res. 2012 Apr 27;110(9):1217-25. doi: 10.1161/CIRCRESAHA.112.267054. Epub 2012 Mar 27.

17.

CIKS (Act1 or TRAF3IP2) mediates high glucose-induced endothelial dysfunction.

Venkatesan B, Valente AJ, Das NA, Carpenter AJ, Yoshida T, Delafontaine JL, Siebenlist U, Chandrasekar B.

Cell Signal. 2013 Jan;25(1):359-71. doi: 10.1016/j.cellsig.2012.10.009. Epub 2012 Oct 17.

18.

MEK/ERK pathway mediates insulin-promoted degradation of MKP-3 protein in liver cells.

Feng B, Jiao P, Yang Z, Xu H.

Mol Cell Endocrinol. 2012 Sep 25;361(1-2):116-23. doi: 10.1016/j.mce.2012.03.025. Epub 2012 Apr 12.

19.

Ameliorative effect of berberine on endothelial dysfunction in diabetic rats induced by high-fat diet and streptozotocin.

Wang C, Li J, Lv X, Zhang M, Song Y, Chen L, Liu Y.

Eur J Pharmacol. 2009 Oct 12;620(1-3):131-7. doi: 10.1016/j.ejphar.2009.07.027. Epub 2009 Aug 15.

PMID:
19686728
20.

Nox4 acts as a switch between differentiation and proliferation in preadipocytes.

Schröder K, Wandzioch K, Helmcke I, Brandes RP.

Arterioscler Thromb Vasc Biol. 2009 Feb;29(2):239-45. doi: 10.1161/ATVBAHA.108.174219. Epub 2008 Dec 4.

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