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Items: 1 to 20 of 180

1.

Transplanted perivascular adipose tissue accelerates injury-induced neointimal hyperplasia: role of monocyte chemoattractant protein-1.

Manka D, Chatterjee TK, Stoll LL, Basford JE, Konaniah ES, Srinivasan R, Bogdanov VY, Tang Y, Blomkalns AL, Hui DY, Weintraub NL.

Arterioscler Thromb Vasc Biol. 2014 Aug;34(8):1723-30. doi: 10.1161/ATVBAHA.114.303983. Epub 2014 Jun 19.

2.

Endovascular injury induces rapid phenotypic changes in perivascular adipose tissue.

Takaoka M, Suzuki H, Shioda S, Sekikawa K, Saito Y, Nagai R, Sata M.

Arterioscler Thromb Vasc Biol. 2010 Aug;30(8):1576-82. doi: 10.1161/ATVBAHA.110.207175. Epub 2010 May 20.

3.

Differential effects of chlorinated and oxidized phospholipids in vascular tissue: implications for neointima formation.

Greig FH, Hutchison L, Spickett CM, Kennedy S.

Clin Sci (Lond). 2015 May;128(9):579-92. doi: 10.1042/CS20140578.

PMID:
25524654
4.

Deletion of Krüppel-like factor 4 in endothelial and hematopoietic cells enhances neointimal formation following vascular injury.

Yoshida T, Yamashita M, Horimai C, Hayashi M.

J Am Heart Assoc. 2014 Jan 27;3(1):e000622. doi: 10.1161/JAHA.113.000622.

5.

Nitric oxide prevents aortic neointimal hyperplasia by controlling macrophage polarization.

Lavin B, Gómez M, Pello OM, Castejon B, Piedras MJ, Saura M, Zaragoza C.

Arterioscler Thromb Vasc Biol. 2014 Aug;34(8):1739-46. doi: 10.1161/ATVBAHA.114.303866. Epub 2014 Jun 12.

6.

MFAP4 Promotes Vascular Smooth Muscle Migration, Proliferation and Accelerates Neointima Formation.

Schlosser A, Pilecki B, Hemstra LE, Kejling K, Kristmannsdottir GB, Wulf-Johansson H, Moeller JB, Füchtbauer EM, Nielsen O, Kirketerp-Møller K, Dubey LK, Hansen PB, Stubbe J, Wrede C, Hegermann J, Ochs M, Rathkolb B, Schrewe A, Bekeredjian R, Wolf E, Gailus-Durner V, Fuchs H, Hrabě de Angelis M, Lindholt JS, Holmskov U, Sorensen GL.

Arterioscler Thromb Vasc Biol. 2016 Jan;36(1):122-33. doi: 10.1161/ATVBAHA.115.306672. Epub 2015 Nov 12.

PMID:
26564819
7.

Crucial role of the CCL2/CCR2 axis in neointimal hyperplasia after arterial injury in hyperlipidemic mice involves early monocyte recruitment and CCL2 presentation on platelets.

Schober A, Zernecke A, Liehn EA, von Hundelshausen P, Knarren S, Kuziel WA, Weber C.

Circ Res. 2004 Nov 26;95(11):1125-33. Epub 2004 Nov 4.

8.

Inhibition of neointimal hyperplasia in the rat carotid artery injury model by a HMGB1 inhibitor.

Chen J, Zhang J, Xu L, Xu C, Chen S, Yang J, Jiang H.

Atherosclerosis. 2012 Oct;224(2):332-9. doi: 10.1016/j.atherosclerosis.2012.07.020. Epub 2012 Jul 24.

PMID:
22857898
9.

HMGB1-Driven Inflammation and Intimal Hyperplasia After Arterial Injury Involves Cell-Specific Actions Mediated by TLR4.

Cai J, Yuan H, Wang Q, Yang H, Al-Abed Y, Hua Z, Wang J, Chen D, Wu J, Lu B, Pribis JP, Jiang W, Yang K, Hackam DJ, Tracey KJ, Billiar TR, Chen AF.

Arterioscler Thromb Vasc Biol. 2015 Dec;35(12):2579-93. doi: 10.1161/ATVBAHA.115.305789. Epub 2015 Oct 29.

10.

Protein kinase C-delta mediates adventitial cell migration through regulation of monocyte chemoattractant protein-1 expression in a rat angioplasty model.

Si Y, Ren J, Wang P, Rateri DL, Daugherty A, Shi XD, Kent KC, Liu B.

Arterioscler Thromb Vasc Biol. 2012 Apr;32(4):943-54. doi: 10.1161/ATVBAHA.111.244921. Epub 2012 Feb 9.

11.

Soluble Flt-1 gene transfer ameliorates neointima formation after wire injury in flt-1 tyrosine kinase-deficient mice.

Koga J, Matoba T, Egashira K, Kubo M, Miyagawa M, Iwata E, Sueishi K, Shibuya M, Sunagawa K.

Arterioscler Thromb Vasc Biol. 2009 Apr;29(4):458-64. doi: 10.1161/ATVBAHA.109.183772. Epub 2009 Jan 22.

12.

Protein Z-deficiency is associated with enhanced neointima formation and inflammatory response after vascular injury in mice.

Butschkau A, Wagner NM, Bierhansl L, Genz B, Vollmar B.

Int J Clin Exp Pathol. 2014 Aug 15;7(9):6064-71. eCollection 2014.

13.

Smooth muscle LDL receptor-related protein-1 inactivation reduces vascular reactivity and promotes injury-induced neointima formation.

Basford JE, Moore ZW, Zhou L, Herz J, Hui DY.

Arterioscler Thromb Vasc Biol. 2009 Nov;29(11):1772-8. doi: 10.1161/ATVBAHA.109.194357. Epub 2009 Sep 3.

14.

The Ca(v)3.1 T-type calcium channel is required for neointimal formation in response to vascular injury in mice.

Tzeng BH, Chen YH, Huang CH, Lin SS, Lee KR, Chen CC.

Cardiovasc Res. 2012 Dec 1;96(3):533-42. doi: 10.1093/cvr/cvs257. Epub 2012 Aug 10.

15.

Mindin regulates vascular smooth muscle cell phenotype and prevents neointima formation.

Zhu LH, Huang L, Zhang X, Zhang P, Zhang SM, Guan H, Zhang Y, Zhu XY, Tian S, Deng K, Li H.

Clin Sci (Lond). 2015 Jul;129(2):129-45. doi: 10.1042/CS20140679.

PMID:
25751394
16.

Adenosine A₂B receptor agonism inhibits neointimal lesion development after arterial injury in apolipoprotein E-deficient mice.

Bot I, de Vries H, Korporaal SJ, Foks AC, Bot M, van Veldhoven J, Ter Borg MN, van Santbrink PJ, van Berkel TJ, Kuiper J, Ijzerman AP.

Arterioscler Thromb Vasc Biol. 2012 Sep;32(9):2197-205. doi: 10.1161/ATVBAHA.112.252924. Epub 2012 Jun 28.

17.

Kalirin promotes neointimal hyperplasia by activating Rac in smooth muscle cells.

Wu JH, Fanaroff AC, Sharma KC, Smith LS, Brian L, Eipper BA, Mains RE, Freedman NJ, Zhang L.

Arterioscler Thromb Vasc Biol. 2013 Apr;33(4):702-8. doi: 10.1161/ATVBAHA.112.300234. Epub 2013 Jan 3.

18.

Deficiency of MAPK-activated protein kinase 2 (MK2) prevents adverse remodelling and promotes endothelial healing after arterial injury.

Kapopara PR, von Felden J, Soehnlein O, Wang Y, Napp LC, Sonnenschein K, Wollert KC, Schieffer B, Gaestel M, Bauersachs J, Bavendiek U.

Thromb Haemost. 2014 Dec;112(6):1264-76. doi: 10.1160/TH14-02-0174. Epub 2014 Aug 14.

PMID:
25120198
19.

Mixed-lineage kinase 3 deficiency promotes neointima formation through increased activation of the RhoA pathway in vascular smooth muscle cells.

Gadang V, Konaniah E, Hui DY, Jaeschke A.

Arterioscler Thromb Vasc Biol. 2014 Jul;34(7):1429-36. doi: 10.1161/ATVBAHA.114.303439. Epub 2014 May 1.

20.

Vascular smooth muscle cell-derived transforming growth factor-β promotes maturation of activated, neointima lesion-like macrophages.

Ostriker A, Horita HN, Poczobutt J, Weiser-Evans MC, Nemenoff RA.

Arterioscler Thromb Vasc Biol. 2014 Apr;34(4):877-86. doi: 10.1161/ATVBAHA.114.303214. Epub 2014 Feb 13.

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