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Mice lacking α1,3-fucosyltransferase 9 exhibit modulation of in vivo immune responses against pathogens.

Kashiwazaki H, Kakizaki M, Ikehara Y, Togayachi A, Narimatsu H, Watanabe R.

Pathol Int. 2014 May;64(5):199-208. doi: 10.1111/pin.12159.


Mice lacking alpha1,3-fucosyltransferase IX demonstrate disappearance of Lewis x structure in brain and increased anxiety-like behaviors.

Kudo T, Fujii T, Ikegami S, Inokuchi K, Takayama Y, Ikehara Y, Nishihara S, Togayachi A, Takahashi S, Tachibana K, Yuasa S, Narimatsu H.

Glycobiology. 2007 Jan;17(1):1-9. Epub 2006 Sep 14. Erratum in: Glycobiology. 2007 Oct;17(10):1030.


Differential roles of CCL2 and CCR2 in host defense to coronavirus infection.

Held KS, Chen BP, Kuziel WA, Rollins BJ, Lane TE.

Virology. 2004 Nov 24;329(2):251-60.


Differential regulation of innate and adaptive immune responses in viral encephalitis.

Rempel JD, Murray SJ, Meisner J, Buchmeier MJ.

Virology. 2004 Jan 5;318(1):381-92.


Murine coronavirus mouse hepatitis virus is recognized by MDA5 and induces type I interferon in brain macrophages/microglia.

Roth-Cross JK, Bender SJ, Weiss SR.

J Virol. 2008 Oct;82(20):9829-38. doi: 10.1128/JVI.01199-08. Epub 2008 Jul 30.


Expression of B and T lymphocyte attenuator (BTLA) in macrophages contributes to the fulminant hepatitis caused by murine hepatitis virus strain-3.

Yang C, Chen Y, Guo G, Li H, Cao D, Xu H, Guo S, Fei L, Yan W, Ning Q, Zheng L, Wu Y.

Gut. 2013 Aug;62(8):1204-13. doi: 10.1136/gutjnl-2012-302239. Epub 2012 May 25.


Protein interferon-stimulated gene 15 conjugation delays but does not overcome coronavirus proliferation in a model of fulminant hepatitis.

Ma XZ, Bartczak A, Zhang J, He W, Shalev I, Smil D, Chen L, Phillips J, Feld JJ, Selzner N, Levy G, McGilvray I.

J Virol. 2014 Jun;88(11):6195-204. doi: 10.1128/JVI.03801-13. Epub 2014 Mar 19.


The Lewis X-related α1,3-fucosyltransferase, Fut10, is required for the maintenance of stem cell populations.

Kumar A, Torii T, Ishino Y, Muraoka D, Yoshimura T, Togayachi A, Narimatsu H, Ikenaka K, Hitoshi S.

J Biol Chem. 2013 Oct 4;288(40):28859-68. doi: 10.1074/jbc.M113.469403. Epub 2013 Aug 28.


Insertion of the CXC chemokine ligand 9 (CXCL9) into the mouse hepatitis virus genome results in protection from viral-induced encephalitis and hepatitis.

Muse M, Kane JA, Carr DJ, Farber JM, Lane TE.

Virology. 2008 Dec 20;382(2):132-44. doi: 10.1016/j.virol.2008.09.032. Epub 2008 Oct 29.


Resistance of naive mice to murine hepatitis virus strain 3 requires development of a Th1, but not a Th2, response, whereas pre-existing antibody partially protects against primary infection.

Liu MF, Ning Q, Pope M, Mosmann T, Leibowitz J, Ding JW, Fung LS, Rotstein O, Gorczynski R, Levy GA.

Adv Exp Med Biol. 1998;440:415-23.


Expression of hemagglutinin/esterase by a mouse hepatitis virus coronavirus defective-interfering RNA alters viral pathogenesis.

Zhang X, Hinton DR, Park S, Parra B, Liao CL, Lai MM, Stohlman SA.

Virology. 1998 Mar 1;242(1):170-83.


Role of spleen macrophages in innate and acquired immune responses against mouse hepatitis virus strain A59.

Wijburg OL, Heemskerk MH, Boog CJ, Van Rooijen N.

Immunology. 1997 Oct;92(2):252-8.


Alpha1,3-fucosyltransferase IX (Fut9) determines Lewis X expression in brain.

Nishihara S, Iwasaki H, Nakajima K, Togayachi A, Ikehara Y, Kudo T, Kushi Y, Furuya A, Shitara K, Narimatsu H.

Glycobiology. 2003 Jun;13(6):445-55. Epub 2003 Feb 20.


Both spike and background genes contribute to murine coronavirus neurovirulence.

Iacono KT, Kazi L, Weiss SR.

J Virol. 2006 Jul;80(14):6834-43.


T cell epitope specificity and pathogenesis of mouse hepatitis virus-1-induced disease in susceptible and resistant hosts.

Khanolkar A, Fulton RB, Epping LL, Pham NL, Tifrea D, Varga SM, Harty JT.

J Immunol. 2010 Jul 15;185(2):1132-41. doi: 10.4049/jimmunol.0902749. Epub 2010 Jun 16.


Type I interferon receptor-independent and -dependent host transcriptional responses to mouse hepatitis coronavirus infection in vivo.

Raaben M, Groot Koerkamp MJ, Rottier PJ, de Haan CA.

BMC Genomics. 2009 Aug 3;10:350. doi: 10.1186/1471-2164-10-350.


The spike glycoprotein of murine coronavirus MHV-JHM mediates receptor-independent infection and spread in the central nervous systems of Ceacam1a-/- Mice.

Miura TA, Travanty EA, Oko L, Bielefeldt-Ohmann H, Weiss SR, Beauchemin N, Holmes KV.

J Virol. 2008 Jan;82(2):755-63. Epub 2007 Nov 14.

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