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Results: 1 to 20 of 109

Similar articles for PubMed (Select 24856820)

1.

Direct observations of amyloid β self-assembly in live cells provide insights into differences in the kinetics of Aβ(1-40) and Aβ(1-42) aggregation.

Esbjörner EK, Chan F, Rees E, Erdelyi M, Luheshi LM, Bertoncini CW, Kaminski CF, Dobson CM, Kaminski Schierle GS.

Chem Biol. 2014 Jun 19;21(6):732-42. doi: 10.1016/j.chembiol.2014.03.014. Epub 2014 May 22.

2.

Timing protein assembly in neurons.

Sauer M.

Chem Biol. 2014 Jun 19;21(6):703-4. doi: 10.1016/j.chembiol.2014.06.001.

3.

Inhibition of amyloid-β aggregation in Alzheimer's disease.

Wang Q, Yu X, Li L, Zheng J.

Curr Pharm Des. 2014;20(8):1223-43. Review.

PMID:
23713775
4.

Photo-induced inhibition of Alzheimer's β-amyloid aggregation in vitro by rose bengal.

Lee JS, Lee BI, Park CB.

Biomaterials. 2015 Jan;38:43-9. doi: 10.1016/j.biomaterials.2014.10.058. Epub 2014 Nov 11.

PMID:
25457982
5.

Genome-wide analysis of Saccharomyces cerevisiae identifies cellular processes affecting intracellular aggregation of Alzheimer's amyloid-β42: importance of lipid homeostasis.

Nair S, Traini M, Dawes IW, Perrone GG.

Mol Biol Cell. 2014 Aug 1;25(15):2235-49. doi: 10.1091/mbc.E13-04-0216. Epub 2014 May 28.

6.

Amyloid-β-induced action potential desynchronization and degradation of hippocampal gamma oscillations is prevented by interference with peptide conformation change and aggregation.

Kurudenkandy FR, Zilberter M, Biverstål H, Presto J, Honcharenko D, Strömberg R, Johansson J, Winblad B, Fisahn A.

J Neurosci. 2014 Aug 20;34(34):11416-25. doi: 10.1523/JNEUROSCI.1195-14.2014.

7.

Endogenous polyamines reduce the toxicity of soluble aβ peptide aggregates associated with Alzheimer's disease.

Luo J, Mohammed I, Wärmländer SK, Hiruma Y, Gräslund A, Abrahams JP.

Biomacromolecules. 2014 Jun 9;15(6):1985-91. doi: 10.1021/bm401874j. Epub 2014 May 2.

PMID:
24750033
8.

[The role of zinc ions and structural polymorphism of β-amyloid in the Alzheimer's disease initiation].

Kulikova AA, Makarov AA, Kozin SA.

Mol Biol (Mosk). 2015 Mar-Apr;49(2):249-63. Review. Russian.

PMID:
26065253
9.

Kinetics of protein fibrillation controlled by fibril elongation.

Kashchiev D.

Proteins. 2014 Sep;82(9):2229-39. doi: 10.1002/prot.24586. Epub 2014 May 2.

PMID:
24753319
10.

Aggregation pathways of the amyloid β(1-42) peptide depend on its colloidal stability and ordered β-sheet stacking.

Jiang D, Rauda I, Han S, Chen S, Zhou F.

Langmuir. 2012 Sep 4;28(35):12711-21. doi: 10.1021/la3021436. Epub 2012 Aug 22.

11.

Specific binding of a β-cyclodextrin dimer to the amyloid β peptide modulates the peptide aggregation process.

Wahlström A, Cukalevski R, Danielsson J, Jarvet J, Onagi H, Rebek J Jr, Linse S, Gräslund A.

Biochemistry. 2012 May 29;51(21):4280-9. doi: 10.1021/bi300341j. Epub 2012 May 17.

PMID:
22554145
12.

Location and conformation of amyloid β(25-35) peptide and its sequence-shuffled peptides within membranes: implications for aggregation and toxicity in PC12 cells.

Tsai HH, Lee JB, Shih YC, Wan L, Shieh FK, Chen CY.

ChemMedChem. 2014 May;9(5):1002-11. doi: 10.1002/cmdc.201400062. Epub 2014 Apr 11.

PMID:
24729535
13.

Naturally occurring polyphenolic inhibitors of amyloid beta aggregation.

Churches QI, Caine J, Cavanagh K, Epa VC, Waddington L, Tranberg CE, Meyer AG, Varghese JN, Streltsov V, Duggan PJ.

Bioorg Med Chem Lett. 2014 Jul 15;24(14):3108-12. doi: 10.1016/j.bmcl.2014.05.008. Epub 2014 May 13.

PMID:
24878198
14.

Interaction between amyloid beta peptide and an aggregation blocker peptide mimicking islet amyloid polypeptide.

Rezaei-Ghaleh N, Andreetto E, Yan LM, Kapurniotu A, Zweckstetter M.

PLoS One. 2011;6(5):e20289. doi: 10.1371/journal.pone.0020289. Epub 2011 May 25.

15.

Cellular polyamines promote amyloid-beta (Aβ) peptide fibrillation and modulate the aggregation pathways.

Luo J, Yu CH, Yu H, Borstnar R, Kamerlin SC, Gräslund A, Abrahams JP, Wärmländer SK.

ACS Chem Neurosci. 2013 Mar 20;4(3):454-62. doi: 10.1021/cn300170x. Epub 2013 Jan 16.

16.

Heparan sulphate proteoglycan and the low-density lipoprotein receptor-related protein 1 constitute major pathways for neuronal amyloid-beta uptake.

Kanekiyo T, Zhang J, Liu Q, Liu CC, Zhang L, Bu G.

J Neurosci. 2011 Feb 2;31(5):1644-51. doi: 10.1523/JNEUROSCI.5491-10.2011.

17.

Polymorphic structures of Alzheimer's β-amyloid globulomers.

Yu X, Zheng J.

PLoS One. 2011;6(6):e20575. doi: 10.1371/journal.pone.0020575. Epub 2011 Jun 7.

18.

Alzheimer's disease amyloid β-protein mutations and deletions that define neuronal binding/internalization as early stage nonfibrillar/fibrillar aggregates and late stage fibrils.

Poduslo JF, Howell KG, Olson NC, Ramirez-Alvarado M, Kandimalla KK.

Biochemistry. 2012 May 15;51(19):3993-4003. doi: 10.1021/bi300275g. Epub 2012 May 7.

PMID:
22545812
19.

Structure-activity relationships in peptide modulators of β-amyloid protein aggregation: variation in α,α-disubstitution results in altered aggregate size and morphology.

Bett CK, Ngunjiri JN, Serem WK, Fontenot KR, Hammer RP, McCarley RL, Garno JC.

ACS Chem Neurosci. 2010 Sep 15;1(9):608-26. doi: 10.1021/cn100045q. Epub 2010 Jul 8.

20.

Tanshinones inhibit amyloid aggregation by amyloid-β peptide, disaggregate amyloid fibrils, and protect cultured cells.

Wang Q, Yu X, Patal K, Hu R, Chuang S, Zhang G, Zheng J.

ACS Chem Neurosci. 2013 Jun 19;4(6):1004-15. doi: 10.1021/cn400051e. Epub 2013 Mar 29.

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