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Items: 1 to 20 of 176

1.

The γH2AX assay for genotoxic and nongenotoxic agents: comparison of H2AX phosphorylation with cell death response.

Nikolova T, Dvorak M, Jung F, Adam I, Krämer E, Gerhold-Ay A, Kaina B.

Toxicol Sci. 2014 Jul;140(1):103-17. doi: 10.1093/toxsci/kfu066. Epub 2014 Apr 17.

2.

The loss of gammaH2AX signal is a marker of DNA double strand breaks repair only at low levels of DNA damage.

Bouquet F, Muller C, Salles B.

Cell Cycle. 2006 May;5(10):1116-22. Epub 2006 May 15.

PMID:
16721046
3.

Assessment of DNA double-strand breaks and gammaH2AX induced by the topoisomerase II poisons etoposide and mitoxantrone.

Smart DJ, Halicka HD, Schmuck G, Traganos F, Darzynkiewicz Z, Williams GM.

Mutat Res. 2008 May 10;641(1-2):43-7. doi: 10.1016/j.mrfmmm.2008.03.005. Epub 2008 Mar 25.

4.

Genotoxicity screening via the γH2AX by flow assay.

Smart DJ, Ahmedi KP, Harvey JS, Lynch AM.

Mutat Res. 2011 Oct 1;715(1-2):25-31. doi: 10.1016/j.mrfmmm.2011.07.001. Epub 2011 Jul 28.

PMID:
21824484
5.

Highly sensitive automated method for DNA damage assessment: gamma-H2AX foci counting and cell cycle sorting.

Hernández L, Terradas M, Martín M, Tusell L, Genescà A.

Int J Mol Sci. 2013 Jul 30;14(8):15810-26. doi: 10.3390/ijms140815810.

6.

Performance of in vitro γH2AX assay in HepG2 cells to predict in vivo genotoxicity.

Tsamou M, Jennen DG, Claessen SM, Magkoufopoulou C, Kleinjans JC, van Delft JH.

Mutagenesis. 2012 Nov;27(6):645-52. doi: 10.1093/mutage/ges030. Epub 2012 Aug 21.

7.

A minority of foci or pan-nuclear apoptotic staining of gammaH2AX in the S phase after UV damage contain DNA double-strand breaks.

de Feraudy S, Revet I, Bezrookove V, Feeney L, Cleaver JE.

Proc Natl Acad Sci U S A. 2010 Apr 13;107(15):6870-5. doi: 10.1073/pnas.1002175107. Epub 2010 Mar 29.

8.

Histone H2AX phosphorylation after cell irradiation with UV-B: relationship to cell cycle phase and induction of apoptosis.

Halicka HD, Huang X, Traganos F, King MA, Dai W, Darzynkiewicz Z.

Cell Cycle. 2005 Feb;4(2):339-45. Epub 2005 Feb 21.

PMID:
15655354
9.

DNA damage evaluated by gammaH2AX foci formation by a selective group of chemical/physical stressors.

Zhou C, Li Z, Diao H, Yu Y, Zhu W, Dai Y, Chen FF, Yang J.

Mutat Res. 2006 Apr 30;604(1-2):8-18. Epub 2006 Jan 19.

10.

Most hydrogen peroxide-induced histone H2AX phosphorylation is mediated by ATR and is not dependent on DNA double-strand breaks.

Katsube T, Mori M, Tsuji H, Shiomi T, Wang B, Liu Q, Nenoi M, Onoda M.

J Biochem. 2014 Aug;156(2):85-95. doi: 10.1093/jb/mvu021. Epub 2014 Mar 28.

PMID:
24682951
11.

Whole cell-ELISA to measure the gammaH2AX response of six aneugens and eight DNA-damaging chemicals.

Matsuzaki K, Harada A, Takeiri A, Tanaka K, Mishima M.

Mutat Res. 2010 Jul 19;700(1-2):71-9. doi: 10.1016/j.mrgentox.2010.05.009. Epub 2010 May 24.

PMID:
20580854
12.

Induction and persistence of large γH2AX foci by high linear energy transfer radiation in DNA-dependent protein kinase-deficient cells.

Bracalente C, Ibañez IL, Molinari B, Palmieri M, Kreiner A, Valda A, Davidson J, Durán H.

Int J Radiat Oncol Biol Phys. 2013 Nov 15;87(4):785-94. doi: 10.1016/j.ijrobp.2013.07.014. Epub 2013 Aug 22.

PMID:
23972723
13.

The complexity of phosphorylated H2AX foci formation and DNA repair assembly at DNA double-strand breaks.

Nakamura AJ, Rao VA, Pommier Y, Bonner WM.

Cell Cycle. 2010 Jan 15;9(2):389-97. Epub 2010 Jan 29.

14.

Induction and rejoining of DNA double strand breaks assessed by H2AX phosphorylation in melanoma cells irradiated with proton and lithium beams.

Ibañez IL, Bracalente C, Molinari BL, Palmieri MA, Policastro L, Kreiner AJ, Burlón AA, Valda A, Navalesi D, Davidson J, Davidson M, Vázquez M, Ozafrán M, Durán H.

Int J Radiat Oncol Biol Phys. 2009 Jul 15;74(4):1226-35. doi: 10.1016/j.ijrobp.2009.02.070.

PMID:
19545788
15.

Bleomycin-induced γH2AX foci map preferentially to replicating domains in CHO9 interphase nuclei.

Liddle P, Lafon-Hughes L, Di Tomaso MV, Reyes-Ábalos AL, Jara J, Cerda M, Härtel S, Folle GA.

Chromosome Res. 2014 Dec;22(4):463-81. doi: 10.1007/s10577-014-9433-9. Epub 2014 Jul 18.

PMID:
25035135
16.

Preferential localization of γH2AX foci in euchromatin of retina rod cells after DNA damage induction.

Lafon-Hughes L, Di Tomaso MV, Liddle P, Toledo A, Reyes-Ábalos AL, Folle GA.

Chromosome Res. 2013 Dec;21(8):789-803. doi: 10.1007/s10577-013-9395-3. Epub 2013 Dec 10.

PMID:
24323064
17.

γH2AX as a marker of DNA double strand breaks and genomic instability in human population studies.

Valdiglesias V, Giunta S, Fenech M, Neri M, Bonassi S.

Mutat Res. 2013 Jul-Sep;753(1):24-40. doi: 10.1016/j.mrrev.2013.02.001. Epub 2013 Feb 13. Review.

PMID:
23416207
18.

Cytometric analysis of DNA damage: phosphorylation of histone H2AX as a marker of DNA double-strand breaks (DSBs).

Tanaka T, Halicka D, Traganos F, Darzynkiewicz Z.

Methods Mol Biol. 2009;523:161-8. doi: 10.1007/978-1-59745-190-1_11.

19.

Discordance between phosphorylation and recruitment of 53BP1 in response to DNA double-strand breaks.

Harding SM, Bristow RG.

Cell Cycle. 2012 Apr 1;11(7):1432-44. doi: 10.4161/cc.19824. Epub 2012 Apr 1.

PMID:
22421153
20.

Chloroethylnitrosourea-induced cell death and genotoxicity: cell cycle dependence and the role of DNA double-strand breaks, HR and NHEJ.

Nikolova T, Hennekes F, Bhatti A, Kaina B.

Cell Cycle. 2012 Jul 15;11(14):2606-19. doi: 10.4161/cc.20862. Epub 2012 Jul 15.

PMID:
22751442
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