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Items: 1 to 20 of 129


Toll-like receptors and CD40 modulate each other's expression affecting Leishmania major infection.

Chandel HS, Pandey SP, Shukla D, Lalsare K, Selvaraj SK, Jha MK, Saha B.

Clin Exp Immunol. 2014 May;176(2):283-90. doi: 10.1111/cei.12264.


Leishmania expressed lipophosphoglycan interacts with Toll-like receptor (TLR)-2 to decrease TLR-9 expression and reduce anti-leishmanial responses.

Srivastava S, Pandey SP, Jha MK, Chandel HS, Saha B.

Clin Exp Immunol. 2013 Jun;172(3):403-9. doi: 10.1111/cei.12074.


B cell TLR1/2, TLR4, TLR7 and TLR9 interact in induction of class switch DNA recombination: modulation by BCR and CD40, and relevance to T-independent antibody responses.

Pone EJ, Lou Z, Lam T, Greenberg ML, Wang R, Xu Z, Casali P.

Autoimmunity. 2015 Feb;48(1):1-12. doi: 10.3109/08916934.2014.993027.


Inhibition of CD40-induced N-Ras activation reduces leishmania major infection.

Chakraborty S, Srivastava A, Jha MK, Nair A, Pandey SP, Srivastava N, Kumari S, Singh S, Krishnasastry MV, Saha B.

J Immunol. 2015 Apr 15;194(8):3852-60. doi: 10.4049/jimmunol.1401996. Epub 2015 Mar 18.


CD40-modulated dual-specificity phosphatases MAPK phosphatase (MKP)-1 and MKP-3 reciprocally regulate Leishmania major infection.

Srivastava N, Sudan R, Saha B.

J Immunol. 2011 May 15;186(10):5863-72. doi: 10.4049/jimmunol.1003957. Epub 2011 Apr 6.


TLR9-deficiency reduces TLR1, TLR2 and TLR3 expressions in Leishmania major-infected macrophages.

Pandey SP, Doyen N, Mishra GC, Saha B, Chandel HS.

Exp Parasitol. 2015 Jul;154:82-6. doi: 10.1016/j.exppara.2015.04.005. Epub 2015 Apr 21.


SHP-1 plays a crucial role in CD40 signaling reciprocity.

Khan TH, Srivastava N, Srivastava A, Sareen A, Mathur RK, Chande AG, Musti KV, Roy S, Mukhopadhyaya R, Saha B.

J Immunol. 2014 Oct 1;193(7):3644-53. doi: 10.4049/jimmunol.1400620. Epub 2014 Sep 3.


Colony-stimulating factor-1 suppresses responses to CpG DNA and expression of toll-like receptor 9 but enhances responses to lipopolysaccharide in murine macrophages.

Sweet MJ, Campbell CC, Sester DP, Xu D, McDonald RC, Stacey KJ, Hume DA, Liew FY.

J Immunol. 2002 Jan 1;168(1):392-9.


CD40 signaling is impaired in L. major-infected macrophages and is rescued by a p38MAPK activator establishing a host-protective memory T cell response.

Awasthi A, Mathur R, Khan A, Joshi BN, Jain N, Sawant S, Boppana R, Mitra D, Saha B.

J Exp Med. 2003 Apr 21;197(8):1037-43. Epub 2003 Apr 14.


Combined CpG and poly I:C stimulation of monocytes results in unique signaling activation not observed with the individual ligands.

Arsenault RJ, Kogut MH, He H.

Cell Signal. 2013 Nov;25(11):2246-54. doi: 10.1016/j.cellsig.2013.07.014. Epub 2013 Jul 19.


Pegylated bisacycloxypropylcysteine, a diacylated lipopeptide ligand of TLR6, plays a host-protective role against experimental Leishmania major infection.

Pandey SP, Chandel HS, Srivastava S, Selvaraj S, Jha MK, Shukla D, Ebensen T, Guzman CA, Saha B.

J Immunol. 2014 Oct 1;193(7):3632-43. doi: 10.4049/jimmunol.1400672. Epub 2014 Sep 5.


UNC93B1 and nucleic acid-sensing Toll-like receptors mediate host resistance to infection with Leishmania major.

Schamber-Reis BL, Petritus PM, Caetano BC, Martinez ER, Okuda K, Golenbock D, Scott P, Gazzinelli RT.

J Biol Chem. 2013 Mar 8;288(10):7127-36. doi: 10.1074/jbc.M112.407684. Epub 2013 Jan 16.


TLR activation pathways in HIV-1-exposed seronegative individuals.

Biasin M, Piacentini L, Lo Caputo S, Naddeo V, Pierotti P, Borelli M, Trabattoni D, Mazzotta F, Shearer GM, Clerici M.

J Immunol. 2010 Mar 1;184(5):2710-7. doi: 10.4049/jimmunol.0902463. Epub 2010 Feb 1.


Cholesterol depletion associated with Leishmania major infection alters macrophage CD40 signalosome composition and effector function.

Rub A, Dey R, Jadhav M, Kamat R, Chakkaramakkil S, Majumdar S, Mukhopadhyaya R, Saha B.

Nat Immunol. 2009 Mar;10(3):273-80. doi: 10.1038/ni.1705. Epub 2009 Feb 8. Erratum in: Nat Immunol. 2009 Jun;10(6):665.


CD40 signaling induces reciprocal outcomes in Leishmania-infected macrophages; roles of host genotype and cytokine milieu.

Nunes MP, Cysne-Finkelstein L, Monteiro BC, de Souza DM, Gomes NA, DosReis GA.

Microbes Infect. 2005 Jan;7(1):78-85. Epub 2004 Nov 18.


Signaling crosstalk during sequential TLR4 and TLR9 activation amplifies the inflammatory response of mouse macrophages.

De Nardo D, De Nardo CM, Nguyen T, Hamilton JA, Scholz GM.

J Immunol. 2009 Dec 15;183(12):8110-8. doi: 10.4049/jimmunol.0901031. Epub .


Quantitative expression of Toll-like receptor-2, -4, and -9 in dendritic cells generated from blasts of patients with acute myeloid leukemia.

Schmitt A, Li L, Giannopoulos K, Greiner J, Reinhardt P, Wiesneth M, Schmitt M.

Transfusion. 2008 May;48(5):861-70. doi: 10.1111/j.1537-2995.2007.01616.x. Epub 2008 Jan 15.


IRF-8/interferon (IFN) consensus sequence-binding protein is involved in Toll-like receptor (TLR) signaling and contributes to the cross-talk between TLR and IFN-gamma signaling pathways.

Zhao J, Kong HJ, Li H, Huang B, Yang M, Zhu C, Bogunovic M, Zheng F, Mayer L, Ozato K, Unkeless J, Xiong H.

J Biol Chem. 2006 Apr 14;281(15):10073-80. Epub 2006 Feb 16.


Zinc finger protein 64 promotes Toll-like receptor-triggered proinflammatory and type I interferon production in macrophages by enhancing p65 subunit activation.

Wang C, Liu X, Liu Y, Zhang Q, Yao Z, Huang B, Zhang P, Li N, Cao X.

J Biol Chem. 2013 Aug 23;288(34):24600-8. doi: 10.1074/jbc.M113.473397. Epub 2013 Jul 15.

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