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Items: 1 to 20 of 107

1.

A Shigella effector dampens inflammation by regulating epithelial release of danger signal ATP through production of the lipid mediator PtdIns5P.

Puhar A, Tronchère H, Payrastre B, Nhieu GT, Sansonetti PJ.

Immunity. 2013 Dec 12;39(6):1121-31. doi: 10.1016/j.immuni.2013.11.013.

2.

Two msbB genes encoding maximal acylation of lipid A are required for invasive Shigella flexneri to mediate inflammatory rupture and destruction of the intestinal epithelium.

D'Hauteville H, Khan S, Maskell DJ, Kussak A, Weintraub A, Mathison J, Ulevitch RJ, Wuscher N, Parsot C, Sansonetti PJ.

J Immunol. 2002 May 15;168(10):5240-51.

3.

PtdIns5P activates the host cell PI3-kinase/Akt pathway during Shigella flexneri infection.

Pendaries C, Tronchère H, Arbibe L, Mounier J, Gozani O, Cantley L, Fry MJ, Gaits-Iacovoni F, Sansonetti PJ, Payrastre B.

EMBO J. 2006 Mar 8;25(5):1024-34. Epub 2006 Feb 16.

4.

PtdIns5P protects Akt from dephosphorylation through PP2A inhibition.

Ramel D, Lagarrigue F, Dupuis-Coronas S, Chicanne G, Leslie N, Gaits-Iacovoni F, Payrastre B, Tronchère H.

Biochem Biophys Res Commun. 2009 Sep 11;387(1):127-31. doi: 10.1016/j.bbrc.2009.06.139. Epub 2009 Jul 1.

PMID:
19576174
5.

The ShiA protein encoded by the Shigella flexneri SHI-2 pathogenicity island attenuates inflammation.

Ingersoll MA, Moss JE, Weinrauch Y, Fisher PE, Groisman EA, Zychlinsky A.

Cell Microbiol. 2003 Nov;5(11):797-807.

PMID:
14531895
6.

Secretory IgA-mediated neutralization of Shigella flexneri prevents intestinal tissue destruction by down-regulating inflammatory circuits.

Boullier S, Tanguy M, Kadaoui KA, Caubet C, Sansonetti P, Corthésy B, Phalipon A.

J Immunol. 2009 Nov 1;183(9):5879-85. doi: 10.4049/jimmunol.0901838. Epub 2009 Oct 14.

7.

Intracellular Shigella remodels its LPS to dampen the innate immune recognition and evade inflammasome activation.

Paciello I, Silipo A, Lembo-Fazio L, Curcurù L, Zumsteg A, Noël G, Ciancarella V, Sturiale L, Molinaro A, Bernardini ML.

Proc Natl Acad Sci U S A. 2013 Nov 12;110(46):E4345-54. doi: 10.1073/pnas.1303641110. Epub 2013 Oct 28. Erratum in: Proc Natl Acad Sci U S A. 2013 Dec 17;110(51):20843.

8.

Sumoylation controls host anti-bacterial response to the gut invasive pathogen Shigella flexneri.

Fritah S, Lhocine N, Golebiowski F, Mounier J, Andrieux A, Jouvion G, Hay RT, Sansonetti P, Dejean A.

EMBO Rep. 2014 Sep;15(9):965-72. doi: 10.15252/embr.201338386. Epub 2014 Aug 5.

9.

The type III effectors NleE and NleB from enteropathogenic E. coli and OspZ from Shigella block nuclear translocation of NF-kappaB p65.

Newton HJ, Pearson JS, Badea L, Kelly M, Lucas M, Holloway G, Wagstaff KM, Dunstone MA, Sloan J, Whisstock JC, Kaper JB, Robins-Browne RM, Jans DA, Frankel G, Phillips AD, Coulson BS, Hartland EL.

PLoS Pathog. 2010 May 13;6(5):e1000898. doi: 10.1371/journal.ppat.1000898.

10.

Shigella flexneri type III secreted effector OspF reveals new crosstalks of proinflammatory signaling pathways during bacterial infection.

Reiterer V, Grossniklaus L, Tschon T, Kasper CA, Sorg I, Arrieumerlou C.

Cell Signal. 2011 Jul;23(7):1188-96. doi: 10.1016/j.cellsig.2011.03.006. Epub 2011 Mar 21.

PMID:
21402152
11.

Connexin-dependent inter-cellular communication increases invasion and dissemination of Shigella in epithelial cells.

Tran Van Nhieu G, Clair C, Bruzzone R, Mesnil M, Sansonetti P, Combettes L.

Nat Cell Biol. 2003 Aug;5(8):720-6.

PMID:
12844145
12.

The regulatory protein PhoP controls susceptibility to the host inflammatory response in Shigella flexneri.

Moss JE, Fisher PE, Vick B, Groisman EA, Zychlinsky A.

Cell Microbiol. 2000 Dec;2(6):443-52.

PMID:
11207599
13.
14.

Protective role of mouse MBL-C on intestinal mucosa during Shigella flexneri invasion.

Zuo DM, Zhang LY, Lu X, Liu Y, Chen ZL.

Int Immunol. 2009 Oct;21(10):1125-34. doi: 10.1093/intimm/dxp078. Epub 2009 Aug 14.

15.

The Shigella flexneri type three secretion system effector IpgD inhibits T cell migration by manipulating host phosphoinositide metabolism.

Konradt C, Frigimelica E, Nothelfer K, Puhar A, Salgado-Pabon W, di Bartolo V, Scott-Algara D, Rodrigues CD, Sansonetti PJ, Phalipon A.

Cell Host Microbe. 2011 Apr 21;9(4):263-72. doi: 10.1016/j.chom.2011.03.010.

16.

Shigella flexneri infection generates the lipid PI5P to alter endocytosis and prevent termination of EGFR signaling.

Ramel D, Lagarrigue F, Pons V, Mounier J, Dupuis-Coronas S, Chicanne G, Sansonetti PJ, Gaits-Iacovoni F, Tronchère H, Payrastre B.

Sci Signal. 2011 Sep 20;4(191):ra61. doi: 10.1126/scisignal.2001619.

17.

Tyrosine kinases, drugs, and Shigella flexneri dissemination.

Dragoi AM, Agaisse H.

Gut Microbes. 2014 Jan-Feb;5(1):44-7. doi: 10.4161/gmic.26523. Epub 2013 Sep 19.

18.

Roles for T and NK cells in the innate immune response to Shigella flexneri.

Le-Barillec K, Magalhaes JG, Corcuff E, Thuizat A, Sansonetti PJ, Phalipon A, Di Santo JP.

J Immunol. 2005 Aug 1;175(3):1735-40.

19.

Shigella gets captured to gain entry.

McCormick BA.

Cell Host Microbe. 2011 Jun 16;9(6):449-50. doi: 10.1016/j.chom.2011.06.001.

20.

Altered expression of MUC2 and MUC5AC in response to Shigella infection, an in vivo study.

Radhakrishnan P, Halagowder D, Devaraj SN.

Biochim Biophys Acta. 2007 Jun;1770(6):884-9. Epub 2007 Feb 23.

PMID:
17395379
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