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Items: 1 to 20 of 110

1.

Progress and outlooks in a genetic absence epilepsy model (WAG/Rij).

van Luijtelaar G, Zobeiri M.

Curr Med Chem. 2014;21(6):704-21. Review.

PMID:
24251564
2.

Genetic models of absence epilepsy, with emphasis on the WAG/Rij strain of rats.

Coenen AM, Drinkenburg WH, Inoue M, van Luijtelaar EL.

Epilepsy Res. 1992 Jul;12(2):75-86. Review.

PMID:
1396543
3.

Protective role for type-1 metabotropic glutamate receptors against spike and wave discharges in the WAG/Rij rat model of absence epilepsy.

Ngomba RT, Santolini I, Biagioni F, Molinaro G, Simonyi A, van Rijn CM, D'Amore V, Mastroiacovo F, Olivieri G, Gradini R, Ferraguti F, Battaglia G, Bruno V, Puliti A, van Luijtelaar G, Nicoletti F.

Neuropharmacology. 2011 Jun;60(7-8):1281-91. doi: 10.1016/j.neuropharm.2011.01.007. Epub 2011 Jan 26.

PMID:
21277877
4.

Spike-wave discharges in adult Sprague-Dawley rats and their implications for animal models of temporal lobe epilepsy.

Pearce PS, Friedman D, Lafrancois JJ, Iyengar SS, Fenton AA, Maclusky NJ, Scharfman HE.

Epilepsy Behav. 2014 Mar;32:121-31. doi: 10.1016/j.yebeh.2014.01.004. Epub 2014 Feb 15. Review.

5.

Amygdala kindling in the WAG/Rij rat model of absence epilepsy.

Aker RG, Yananli HR, Gurbanova AA, Ozkaynakçi AE, Ateş N, van Luijtelaar G, Onat FY.

Epilepsia. 2006 Jan;47(1):33-40.

6.

Time-frequency characteristics and dynamics of sleep spindles in WAG/Rij rats with absence epilepsy.

Sitnikova E, Hramov AE, Grubov V, Koronovsky AA.

Brain Res. 2014 Jan 16;1543:290-9. doi: 10.1016/j.brainres.2013.11.001. Epub 2013 Nov 11.

PMID:
24231550
7.

On the origin and suddenness of absences in genetic absence models.

van Luijtelaar G, Sitnikova E, Littjohann A.

Clin EEG Neurosci. 2011 Apr;42(2):83-97. Erratum in: Clin EEG Neurosci. 2014 Jan;45(1):61.

PMID:
21675598
8.

Thalamic lesions in a genetic rat model of absence epilepsy: dissociation between spike-wave discharges and sleep spindles.

Meeren HK, Veening JG, Möderscheim TA, Coenen AM, van Luijtelaar G.

Exp Neurol. 2009 May;217(1):25-37. doi: 10.1016/j.expneurol.2009.01.009. Epub 2009 Jan 24.

PMID:
19416679
9.

The effect of generalized absence seizures on the progression of kindling in the rat.

Onat FY, Aker RG, Gurbanova AA, Ateş N, van Luijtelaar G.

Epilepsia. 2007;48 Suppl 5:150-6. Erratum in: Epilepsia. 2007 Dec;48(12):2381.

10.

Electroencephalographic differences between WAG/Rij and GAERS rat models of absence epilepsy.

Akman O, Demiralp T, Ates N, Onat FY.

Epilepsy Res. 2010 May;89(2-3):185-93. doi: 10.1016/j.eplepsyres.2009.12.005. Epub 2010 Jan 25.

PMID:
20092980
11.

Genetic animal models for absence epilepsy: a review of the WAG/Rij strain of rats.

Coenen AM, Van Luijtelaar EL.

Behav Genet. 2003 Nov;33(6):635-55. Review.

PMID:
14574120
12.

Effect of systemic and intracortical administration of phenytoin in two genetic models of absence epilepsy.

Gurbanova AA, Aker R, Berkman K, Onat FY, van Rijn CM, van Luijtelaar G.

Br J Pharmacol. 2006 Aug;148(8):1076-82. Epub 2006 Jul 24.

13.

The WAG/Rij strain: a genetic animal model of absence epilepsy with comorbidity of depression [corrected].

Sarkisova K, van Luijtelaar G.

Prog Neuropsychopharmacol Biol Psychiatry. 2011 Jun 1;35(4):854-76. doi: 10.1016/j.pnpbp.2010.11.010. Epub 2010 Nov 17. Review. Erratum in: Prog Neuropsychopharmacol Biol Psychiatry. 2012 Jan 10;36(1):212.

PMID:
21093520
14.

Chromosomal mapping of genetic loci controlling absence epilepsy phenotypes in the WAG/Rij rat.

Gauguier D, van Luijtelaar G, Bihoreau MT, Wilder SP, Godfrey RF, Vossen J, Coenen A, Cox RD.

Epilepsia. 2004 Aug;45(8):908-15.

15.

The preferential mGlu2/3 receptor antagonist, LY341495, reduces the frequency of spike-wave discharges in the WAG/Rij rat model of absence epilepsy.

Ngomba RT, Biagioni F, Casciato S, Willems-van Bree E, Battaglia G, Bruno V, Nicoletti F, van Luijtelaar EL.

Neuropharmacology. 2005;49 Suppl 1:89-103.

PMID:
16043198
16.

Global and focal aspects of absence epilepsy: the contribution of genetic models.

van Luijtelaar G, Sitnikova E.

Neurosci Biobehav Rev. 2006;30(7):983-1003. Epub 2006 May 24. Review.

PMID:
16725200
17.

Neuroactive steroids and GABAA receptor plasticity in the brain of the WAG/Rij rat, a model of absence epilepsy.

Pisu MG, Mostallino MC, Dore R, Mura ML, Maciocco E, Russo E, De Sarro G, Serra M.

J Neurochem. 2008 Sep;106(6):2502-14. doi: 10.1111/j.1471-4159.2008.05538.x. Epub 2008 Jul 4.

18.

Stress, glucocorticoids and absences in a genetic epilepsy model.

Tolmacheva EA, Oitzl MS, van Luijtelaar G.

Horm Behav. 2012 May;61(5):706-10. doi: 10.1016/j.yhbeh.2012.03.004. Epub 2012 Mar 23.

PMID:
22465594
19.

WAG/Rij rats show a reduced expression of CB₁ receptors in thalamic nuclei and respond to the CB₁ receptor agonist, R(+)WIN55,212-2, with a reduced incidence of spike-wave discharges.

van Rijn CM, Gaetani S, Santolini I, Badura A, Gabova A, Fu J, Watanabe M, Cuomo V, van Luijtelaar G, Nicoletti F, Ngomba RT.

Epilepsia. 2010 Aug;51(8):1511-21. doi: 10.1111/j.1528-1167.2009.02510.x. Epub 2010 Feb 3.

20.

Increased resting functional connectivity in spike-wave epilepsy in WAG/Rij rats.

Mishra AM, Bai X, Motelow JE, Desalvo MN, Danielson N, Sanganahalli BG, Hyder F, Blumenfeld H.

Epilepsia. 2013 Jul;54(7):1214-22. doi: 10.1111/epi.12227.

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