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Items: 1 to 20 of 138

1.

Metabolic abnormalities and hypoleptinemia in α-synuclein A53T mutant mice.

Rothman SM, Griffioen KJ, Fishbein KW, Spencer RG, Makrogiannis S, Cong WN, Martin B, Mattson MP.

Neurobiol Aging. 2014 May;35(5):1153-61. doi: 10.1016/j.neurobiolaging.2013.10.088. Epub 2013 Oct 22.

2.

Neuronal expression of familial Parkinson's disease A53T α-synuclein causes early motor impairment, reduced anxiety and potential sleep disturbances in mice.

Rothman SM, Griffioen KJ, Vranis N, Ladenheim B, Cong WN, Cadet JL, Haran J, Martin B, Mattson MP.

J Parkinsons Dis. 2013;3(2):215-29. doi: 10.3233/JPD-120130.

PMID:
23938351
3.

Augmentation of phenotype in a transgenic Parkinson mouse heterozygous for a Gaucher mutation.

Fishbein I, Kuo YM, Giasson BI, Nussbaum RL.

Brain. 2014 Dec;137(Pt 12):3235-47. doi: 10.1093/brain/awu291. Epub 2014 Oct 27.

4.
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Extensive enteric nervous system abnormalities in mice transgenic for artificial chromosomes containing Parkinson disease-associated alpha-synuclein gene mutations precede central nervous system changes.

Kuo YM, Li Z, Jiao Y, Gaborit N, Pani AK, Orrison BM, Bruneau BG, Giasson BI, Smeyne RJ, Gershon MD, Nussbaum RL.

Hum Mol Genet. 2010 May 1;19(9):1633-50. doi: 10.1093/hmg/ddq038. Epub 2010 Jan 27.

6.

Behavioral characterization of A53T mice reveals early and late stage deficits related to Parkinson's disease.

Paumier KL, Sukoff Rizzo SJ, Berger Z, Chen Y, Gonzales C, Kaftan E, Li L, Lotarski S, Monaghan M, Shen W, Stolyar P, Vasilyev D, Zaleska M, D Hirst W, Dunlop J.

PLoS One. 2013 Aug 1;8(8):e70274. doi: 10.1371/journal.pone.0070274. Print 2013.

7.

Higher vulnerability and stress sensitivity of neuronal precursor cells carrying an alpha-synuclein gene triplication.

Flierl A, Oliveira LM, Falomir-Lockhart LJ, Mak SK, Hesley J, Soldner F, Arndt-Jovin DJ, Jaenisch R, Langston JW, Jovin TM, Schüle B.

PLoS One. 2014 Nov 12;9(11):e112413. doi: 10.1371/journal.pone.0112413. eCollection 2014.

8.

Exacerbated synucleinopathy in mice expressing A53T SNCA on a Snca null background.

Cabin DE, Gispert-Sanchez S, Murphy D, Auburger G, Myers RR, Nussbaum RL.

Neurobiol Aging. 2005 Jan;26(1):25-35.

PMID:
15585343
9.

Region-specific deficits in dopamine, but not norepinephrine, signaling in a novel A30P α-synuclein BAC transgenic mouse.

Taylor TN, Potgieter D, Anwar S, Senior SL, Janezic S, Threlfell S, Ryan B, Parkkinen L, Deltheil T, Cioroch M, Livieratos A, Oliver PL, Jennings KA, Davies KE, Ansorge O, Bannerman DM, Cragg SJ, Wade-Martins R.

Neurobiol Dis. 2014 Feb;62:193-207. doi: 10.1016/j.nbd.2013.10.005. Epub 2013 Oct 10.

10.

Modulation of central leptin sensitivity and energy balance in a rat model of diet-induced obesity.

Fam BC, Morris MJ, Hansen MJ, Kebede M, Andrikopoulos S, Proietto J, Thorburn AW.

Diabetes Obes Metab. 2007 Nov;9(6):840-52.

PMID:
17924866
11.

Divergent effects of the H50Q and G51D SNCA mutations on the aggregation of α-synuclein.

Rutherford NJ, Moore BD, Golde TE, Giasson BI.

J Neurochem. 2014 Dec;131(6):859-67. doi: 10.1111/jnc.12806. Epub 2014 Jul 21.

12.

Alpha-synuclein A30P point-mutation generates age-dependent nigrostriatal deficiency in mice.

Plaas M, Karis A, Innos J, Rebane E, Baekelandt V, Vaarmann A, Luuk H, Vasar E, Koks S.

J Physiol Pharmacol. 2008 Jun;59(2):205-16.

13.

Increased energy expenditure and leptin sensitivity account for low fat mass in myostatin-deficient mice.

Choi SJ, Yablonka-Reuveni Z, Kaiyala KJ, Ogimoto K, Schwartz MW, Wisse BE.

Am J Physiol Endocrinol Metab. 2011 Jun;300(6):E1031-7. doi: 10.1152/ajpendo.00656.2010. Epub 2011 Mar 22.

14.

Expansion of the Parkinson disease-associated SNCA-Rep1 allele upregulates human alpha-synuclein in transgenic mouse brain.

Cronin KD, Ge D, Manninger P, Linnertz C, Rossoshek A, Orrison BM, Bernard DJ, El-Agnaf OM, Schlossmacher MG, Nussbaum RL, Chiba-Falek O.

Hum Mol Genet. 2009 Sep 1;18(17):3274-85. doi: 10.1093/hmg/ddp265. Epub 2009 Jun 4.

15.

Mitochondrial associated metabolic proteins are selectively oxidized in A30P alpha-synuclein transgenic mice--a model of familial Parkinson's disease.

Poon HF, Frasier M, Shreve N, Calabrese V, Wolozin B, Butterfield DA.

Neurobiol Dis. 2005 Apr;18(3):492-8.

PMID:
15755676
16.

Alpha-synuclein A53T mutation is not frequent on a sample of Brazilian Parkinson's disease patients.

Longo GS, Pinhel MA, Gregório ML, Oliveira BA, Quinhoneiro DC, Tognola WA, Oliveira FN, Martins DP, Cezario SM, Sado CL, Nakazone MA, Calastri MC, Souza DR.

Arq Neuropsiquiatr. 2015 Jun;73(6):506-9. doi: 10.1590/0004-282X20150032. Erratum in: Arq Neuropsiquiatr. 2015 Nov;73(11):I. Martins, Denise Poltronieri [added].

17.

Dietary energy intake modifies brainstem autonomic dysfunction caused by mutant α-synuclein.

Griffioen KJ, Rothman SM, Ladenheim B, Wan R, Vranis N, Hutchison E, Okun E, Cadet JL, Mattson MP.

Neurobiol Aging. 2013 Mar;34(3):928-35. doi: 10.1016/j.neurobiolaging.2012.07.008. Epub 2012 Aug 9.

18.

The carbohydrate sensitive rat as a model of obesity.

Nadkarni NA, Chaumontet C, Azzout-Marniche D, Piedcoq J, Fromentin G, Tomé D, Even PC.

PLoS One. 2013 Jul 30;8(7):e68436. doi: 10.1371/journal.pone.0068436. Print 2013.

19.

Tauopathic changes in the striatum of A53T α-synuclein mutant mouse model of Parkinson's disease.

Wills J, Credle J, Haggerty T, Lee JH, Oaks AW, Sidhu A.

PLoS One. 2011 Mar 21;6(3):e17953. doi: 10.1371/journal.pone.0017953.

20.
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