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Items: 1 to 20 of 116

1.

Tafazzin knockdown interrupts cell cycle progression in cultured neonatal ventricular fibroblasts.

He Q, Wang M, Harris N, Han X.

Am J Physiol Heart Circ Physiol. 2013 Nov 1;305(9):H1332-43. doi: 10.1152/ajpheart.00084.2013. Epub 2013 Aug 30.

2.

Tafazzin knockdown causes hypertrophy of neonatal ventricular myocytes.

He Q.

Am J Physiol Heart Circ Physiol. 2010 Jul;299(1):H210-6. doi: 10.1152/ajpheart.00098.2010. Epub 2010 Mar 26.

3.

Mitochondria-targeted antioxidant prevents cardiac dysfunction induced by tafazzin gene knockdown in cardiac myocytes.

He Q, Harris N, Ren J, Han X.

Oxid Med Cell Longev. 2014;2014:654198. doi: 10.1155/2014/654198. Epub 2014 Aug 27.

4.

Barth syndrome: cellular compensation of mitochondrial dysfunction and apoptosis inhibition due to changes in cardiolipin remodeling linked to tafazzin (TAZ) gene mutation.

Gonzalvez F, D'Aurelio M, Boutant M, Moustapha A, Puech JP, Landes T, Arnauné-Pelloquin L, Vial G, Taleux N, Slomianny C, Wanders RJ, Houtkooper RH, Bellenguer P, Møller IM, Gottlieb E, Vaz FM, Manfredi G, Petit PX.

Biochim Biophys Acta. 2013 Aug;1832(8):1194-206. doi: 10.1016/j.bbadis.2013.03.005. Epub 2013 Mar 20.

5.

Cardiac and skeletal muscle defects in a mouse model of human Barth syndrome.

Acehan D, Vaz F, Houtkooper RH, James J, Moore V, Tokunaga C, Kulik W, Wansapura J, Toth MJ, Strauss A, Khuchua Z.

J Biol Chem. 2011 Jan 14;286(2):899-908. doi: 10.1074/jbc.M110.171439. Epub 2010 Nov 9.

6.

Cardiolipin remodeling by TAZ/tafazzin is selectively required for the initiation of mitophagy.

Hsu P, Liu X, Zhang J, Wang HG, Ye JM, Shi Y.

Autophagy. 2015 Apr 3;11(4):643-52. doi: 10.1080/15548627.2015.1023984.

7.

Intrafamilial variability for novel TAZ gene mutation: Barth syndrome with dilated cardiomyopathy and heart failure in an infant and left ventricular noncompaction in his great-uncle.

Ronvelia D, Greenwood J, Platt J, Hakim S, Zaragoza MV.

Mol Genet Metab. 2012 Nov;107(3):428-32. doi: 10.1016/j.ymgme.2012.09.013. Epub 2012 Sep 18.

8.

A Drosophila model of Barth syndrome.

Xu Y, Condell M, Plesken H, Edelman-Novemsky I, Ma J, Ren M, Schlame M.

Proc Natl Acad Sci U S A. 2006 Aug 1;103(31):11584-8. Epub 2006 Jul 19.

9.

Cardiolipin deficiency affects respiratory chain function and organization in an induced pluripotent stem cell model of Barth syndrome.

Dudek J, Cheng IF, Balleininger M, Vaz FM, Streckfuss-Bömeke K, Hübscher D, Vukotic M, Wanders RJ, Rehling P, Guan K.

Stem Cell Res. 2013 Sep;11(2):806-19. doi: 10.1016/j.scr.2013.05.005. Epub 2013 May 28.

10.

A zebrafish model of human Barth syndrome reveals the essential role of tafazzin in cardiac development and function.

Khuchua Z, Yue Z, Batts L, Strauss AW.

Circ Res. 2006 Jul 21;99(2):201-8. Epub 2006 Jun 22.

12.

Adiponectin induces interleukin-6 production and its underlying mechanism in adult rat cardiac fibroblasts.

Fan D, Li L, Wang C, Cui XB, Zhou Y, Wu LL.

J Cell Physiol. 2011 Jul;226(7):1793-802. doi: 10.1002/jcp.22512.

PMID:
21069809
14.

Atrial natriuretic peptide suppresses endothelin gene expression and proliferation in cardiac fibroblasts through a GATA4-dependent mechanism.

Glenn DJ, Rahmutula D, Nishimoto M, Liang F, Gardner DG.

Cardiovasc Res. 2009 Nov 1;84(2):209-17. doi: 10.1093/cvr/cvp208. Epub 2009 Jun 22.

15.

Advances in the understanding of Barth syndrome.

Aprikyan AA, Khuchua Z.

Br J Haematol. 2013 May;161(3):330-8. doi: 10.1111/bjh.12271. Epub 2013 Feb 25. Review.

PMID:
23432031
16.

Modeling the mitochondrial cardiomyopathy of Barth syndrome with induced pluripotent stem cell and heart-on-chip technologies.

Wang G, McCain ML, Yang L, He A, Pasqualini FS, Agarwal A, Yuan H, Jiang D, Zhang D, Zangi L, Geva J, Roberts AE, Ma Q, Ding J, Chen J, Wang DZ, Li K, Wang J, Wanders RJ, Kulik W, Vaz FM, Laflamme MA, Murry CE, Chien KR, Kelley RI, Church GM, Parker KK, Pu WT.

Nat Med. 2014 Jun;20(6):616-23. doi: 10.1038/nm.3545. Epub 2014 May 11.

17.

Characterization of a transgenic short hairpin RNA-induced murine model of Tafazzin deficiency.

Soustek MS, Falk DJ, Mah CS, Toth MJ, Schlame M, Lewin AS, Byrne BJ.

Hum Gene Ther. 2011 Jul;22(7):865-71. doi: 10.1089/hum.2010.199. Epub 2011 May 19.

18.

Cardiac metabolic pathways affected in the mouse model of barth syndrome.

Huang Y, Powers C, Madala SK, Greis KD, Haffey WD, Towbin JA, Purevjav E, Javadov S, Strauss AW, Khuchua Z.

PLoS One. 2015 Jun 1;10(6):e0128561. doi: 10.1371/journal.pone.0128561. eCollection 2015.

19.

Nicotinamide adenine dinucleotide phosphate oxidase 4 mediates the differential responsiveness of atrial versus ventricular fibroblasts to transforming growth factor-β.

Yeh YH, Kuo CT, Chang GJ, Qi XY, Nattel S, Chen WJ.

Circ Arrhythm Electrophysiol. 2013 Aug;6(4):790-8. doi: 10.1161/CIRCEP.113.000338. Epub 2013 Jul 24.

20.

Regulation of collagen synthesis by inhibitory Smad7 in cardiac myofibroblasts.

Wang B, Omar A, Angelovska T, Drobic V, Rattan SG, Jones SC, Dixon IM.

Am J Physiol Heart Circ Physiol. 2007 Aug;293(2):H1282-90. Epub 2007 May 18.

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