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Results: 1 to 20 of 94

Similar articles for PubMed (Select 23922721)

1.

Mitochondrial dysfunction promotes breast cancer cell migration and invasion through HIF1α accumulation via increased production of reactive oxygen species.

Ma J, Zhang Q, Chen S, Fang B, Yang Q, Chen C, Miele L, Sarkar FH, Xia J, Wang Z.

PLoS One. 2013 Jul 29;8(7):e69485. doi: 10.1371/journal.pone.0069485. Print 2013.

2.

Mitochondrial dysfunction promotes cell migration via reactive oxygen species-enhanced β5-integrin expression in human gastric cancer SC-M1 cells.

Hung WY, Huang KH, Wu CW, Chi CW, Kao HL, Li AF, Yin PH, Lee HC.

Biochim Biophys Acta. 2012 Jul;1820(7):1102-10. doi: 10.1016/j.bbagen.2012.04.016. Epub 2012 Apr 26.

PMID:
22561002
3.

Regulation of VEGF-induced endothelial cell migration by mitochondrial reactive oxygen species.

Wang Y, Zang QS, Liu Z, Wu Q, Maass D, Dulan G, Shaul PW, Melito L, Frantz DE, Kilgore JA, Williams NS, Terada LS, Nwariaku FE.

Am J Physiol Cell Physiol. 2011 Sep;301(3):C695-704. doi: 10.1152/ajpcell.00322.2010. Epub 2011 Jun 8.

4.

Mitochondrial dysfunction and reactive oxygen species imbalance promote breast cancer cell motility through a CXCL14-mediated mechanism.

Pelicano H, Lu W, Zhou Y, Zhang W, Chen Z, Hu Y, Huang P.

Cancer Res. 2009 Mar 15;69(6):2375-83. doi: 10.1158/0008-5472.CAN-08-3359. Epub 2009 Mar 10.

5.

Mitochondrial production of reactive oxygen species mediate dicumarol-induced cytotoxicity in cancer cells.

Du J, Daniels DH, Asbury C, Venkataraman S, Liu J, Spitz DR, Oberley LW, Cullen JJ.

J Biol Chem. 2006 Dec 8;281(49):37416-26. Epub 2006 Oct 13.

6.

Mitochondrial mutations contribute to HIF1alpha accumulation via increased reactive oxygen species and up-regulated pyruvate dehydrogenease kinase 2 in head and neck squamous cell carcinoma.

Sun W, Zhou S, Chang SS, McFate T, Verma A, Califano JA.

Clin Cancer Res. 2009 Jan 15;15(2):476-84. doi: 10.1158/1078-0432.CCR-08-0930.

7.

Reactive oxygen species are generated by the respiratory complex II--evidence for lack of contribution of the reverse electron flow in complex I.

Moreno-Sánchez R, Hernández-Esquivel L, Rivero-Segura NA, Marín-Hernández A, Neuzil J, Ralph SJ, Rodríguez-Enríquez S.

FEBS J. 2013 Feb;280(3):927-38. doi: 10.1111/febs.12086. Epub 2013 Jan 7.

PMID:
23206332
8.

Cyanide preconditioning protects brain endothelial and NT2 neuron-like cells against glucotoxicity: role of mitochondrial reactive oxygen species and HIF-1α.

Correia SC, Santos RX, Cardoso SM, Santos MS, Oliveira CR, Moreira PI.

Neurobiol Dis. 2012 Jan;45(1):206-18. doi: 10.1016/j.nbd.2011.08.005. Epub 2011 Aug 10.

PMID:
21854848
9.

Mitochondrial DNA depletion promotes impaired oxidative status and adaptive resistance to apoptosis in T47D breast cancer cells.

Yu M, Shi Y, Wei X, Yang Y, Zang F, Niu R.

Eur J Cancer Prev. 2009 Nov;18(6):445-57. doi: 10.1097/CEJ.0b013e32832f9bd6.

PMID:
19609211
10.

TNF-α mediates mitochondrial uncoupling and enhances ROS-dependent cell migration via NF-κB activation in liver cells.

Kastl L, Sauer SW, Ruppert T, Beissbarth T, Becker MS, Süss D, Krammer PH, Gülow K.

FEBS Lett. 2014 Jan 3;588(1):175-83. doi: 10.1016/j.febslet.2013.11.033. Epub 2013 Dec 4.

11.

Depletion of end-binding protein 1 (EB1) promotes apoptosis of human non-small-cell lung cancer cells via reactive oxygen species and Bax-mediated mitochondrial dysfunction.

Kim MJ, Yun HS, Hong EH, Lee SJ, Baek JH, Lee CW, Yim JH, Kim JS, Park JK, Um HD, Hwang SG.

Cancer Lett. 2013 Oct 1;339(1):15-24. doi: 10.1016/j.canlet.2013.07.027. Epub 2013 Jul 27.

12.

Metabolic reprogramming and two-compartment tumor metabolism: opposing role(s) of HIF1α and HIF2α in tumor-associated fibroblasts and human breast cancer cells.

Chiavarina B, Martinez-Outschoorn UE, Whitaker-Menezes D, Howell A, Tanowitz HB, Pestell RG, Sotgia F, Lisanti MP.

Cell Cycle. 2012 Sep 1;11(17):3280-9. doi: 10.4161/cc.21643. Epub 2012 Aug 16.

13.

HIV antiretroviral drug combination induces endothelial mitochondrial dysfunction and reactive oxygen species production, but not apoptosis.

Jiang B, Hebert VY, Li Y, Mathis JM, Alexander JS, Dugas TR.

Toxicol Appl Pharmacol. 2007 Oct 1;224(1):60-71. Epub 2007 Jun 30.

PMID:
17669453
14.

Mitochondrial complex III is required for hypoxia-induced ROS production and cellular oxygen sensing.

Guzy RD, Hoyos B, Robin E, Chen H, Liu L, Mansfield KD, Simon MC, Hammerling U, Schumacker PT.

Cell Metab. 2005 Jun;1(6):401-8.

15.

Reactive oxygen species generated at mitochondrial complex III stabilize hypoxia-inducible factor-1alpha during hypoxia: a mechanism of O2 sensing.

Chandel NS, McClintock DS, Feliciano CE, Wood TM, Melendez JA, Rodriguez AM, Schumacker PT.

J Biol Chem. 2000 Aug 18;275(33):25130-8.

16.

Redox regulation of p53 during hypoxia.

Chandel NS, Vander Heiden MG, Thompson CB, Schumacker PT.

Oncogene. 2000 Aug 10;19(34):3840-8.

17.

Disruption of endothelial adherens junction by invasive breast cancer cells is mediated by reactive oxygen species and is attenuated by AHCC.

Haidari M, Zhang W, Wakame K.

Life Sci. 2013 Dec 18;93(25-26):994-1003. doi: 10.1016/j.lfs.2013.10.027. Epub 2013 Nov 6.

PMID:
24211779
18.

Reactive oxygen species mediate caspase activation and apoptosis induced by lipoic acid in human lung epithelial cancer cells through Bcl-2 down-regulation.

Moungjaroen J, Nimmannit U, Callery PS, Wang L, Azad N, Lipipun V, Chanvorachote P, Rojanasakul Y.

J Pharmacol Exp Ther. 2006 Dec;319(3):1062-9. Epub 2006 Sep 21.

19.

Mitochondrial ROS-K+ channel signaling pathway regulated secretion of human pulmonary artery endothelial cells.

Ouyang JS, Li YP, Li CY, Cai C, Chen CS, Chen SX, Chen YF, Yang L, Xie YP.

Free Radic Res. 2012 Dec;46(12):1437-45. doi: 10.3109/10715762.2012.724532. Epub 2012 Sep 27.

PMID:
22928487
20.

Carbon monoxide activates autophagy via mitochondrial reactive oxygen species formation.

Lee SJ, Ryter SW, Xu JF, Nakahira K, Kim HP, Choi AM, Kim YS.

Am J Respir Cell Mol Biol. 2011 Oct;45(4):867-73. doi: 10.1165/rcmb.2010-0352OC. Epub 2011 Mar 25.

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