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Items: 1 to 20 of 166

1.

Mechanisms of HIV entry into the CNS: increased sensitivity of HIV infected CD14+CD16+ monocytes to CCL2 and key roles of CCR2, JAM-A, and ALCAM in diapedesis.

Williams DW, Calderon TM, Lopez L, Carvallo-Torres L, Gaskill PJ, Eugenin EA, Morgello S, Berman JW.

PLoS One. 2013 Jul 26;8(7):e69270. doi: 10.1371/journal.pone.0069270. Print 2013.

2.

JAM-A and ALCAM are therapeutic targets to inhibit diapedesis across the BBB of CD14+CD16+ monocytes in HIV-infected individuals.

Williams DW, Anastos K, Morgello S, Berman JW.

J Leukoc Biol. 2015 Feb;97(2):401-12. doi: 10.1189/jlb.5A0714-347R. Epub 2014 Nov 24.

3.

Characterization of monocyte maturation/differentiation that facilitates their transmigration across the blood-brain barrier and infection by HIV: implications for NeuroAIDS.

Buckner CM, Calderon TM, Willams DW, Belbin TJ, Berman JW.

Cell Immunol. 2011;267(2):109-23. doi: 10.1016/j.cellimm.2010.12.004. Epub 2010 Dec 25.

4.

Dopamine increases CD14+CD16+ monocyte migration and adhesion in the context of substance abuse and HIV neuropathogenesis.

Coley JS, Calderon TM, Gaskill PJ, Eugenin EA, Berman JW.

PLoS One. 2015 Feb 3;10(2):e0117450. doi: 10.1371/journal.pone.0117450. eCollection 2015.

5.

Monocyte maturation, HIV susceptibility, and transmigration across the blood brain barrier are critical in HIV neuropathogenesis.

Williams DW, Eugenin EA, Calderon TM, Berman JW.

J Leukoc Biol. 2012 Mar;91(3):401-15. doi: 10.1189/jlb.0811394. Epub 2012 Jan 6. Review.

6.

Monocytes mediate HIV neuropathogenesis: mechanisms that contribute to HIV associated neurocognitive disorders.

Williams DW, Veenstra M, Gaskill PJ, Morgello S, Calderon TM, Berman JW.

Curr HIV Res. 2014;12(2):85-96. Review.

7.

Buprenorphine decreases the CCL2-mediated chemotactic response of monocytes.

Carvallo L, Lopez L, Che FY, Lim J, Eugenin EA, Williams DW, Nieves E, Calderon TM, Madrid-Aliste C, Fiser A, Weiss L, Angeletti RH, Berman JW.

J Immunol. 2015 Apr 1;194(7):3246-58. doi: 10.4049/jimmunol.1302647. Epub 2015 Feb 25.

8.

Expansion of a subset of CD14highCD16negCCR2low/neg monocytes functionally similar to myeloid-derived suppressor cells during SIV and HIV infection.

Gama L, Shirk EN, Russell JN, Carvalho KI, Li M, Queen SE, Kalil J, Zink MC, Clements JE, Kallas EG.

J Leukoc Biol. 2012 May;91(5):803-16. doi: 10.1189/jlb.1111579. Epub 2012 Feb 24.

9.

CCR2 on CD14(+)CD16(+) monocytes is a biomarker of HIV-associated neurocognitive disorders.

Williams DW, Byrd D, Rubin LH, Anastos K, Morgello S, Berman JW.

Neurol Neuroimmunol Neuroinflamm. 2014 Oct 9;1(3):e36. doi: 10.1212/NXI.0000000000000036. eCollection 2014 Oct.

10.

Shedding of PECAM-1 during HIV infection: a potential role for soluble PECAM-1 in the pathogenesis of NeuroAIDS.

Eugenin EA, Gamss R, Buckner C, Buono D, Klein RS, Schoenbaum EE, Calderon TM, Berman JW.

J Leukoc Biol. 2006 Mar;79(3):444-52. Epub 2006 Jan 13.

11.

Selective expansion of pro-inflammatory chemokine CCL2-loaded CD14+CD16+ monocytes subset in HIV-infected therapy naïve individuals.

Ansari AW, Meyer-Olson D, Schmidt RE.

J Clin Immunol. 2013 Jan;33(1):302-6. doi: 10.1007/s10875-012-9790-0. Epub 2012 Sep 8.

PMID:
22961048
12.

CXCL12-induced monocyte-endothelial interactions promote lymphocyte transmigration across an in vitro blood-brain barrier.

Man S, Tucky B, Cotleur A, Drazba J, Takeshita Y, Ransohoff RM.

Sci Transl Med. 2012 Feb 1;4(119):119ra14. doi: 10.1126/scitranslmed.3003197.

13.

CD14(high)CD16(+) rather than CD14(low)CD16(+) monocytes correlate with disease progression in chronic HIV-infected patients.

Han J, Wang B, Han N, Zhao Y, Song C, Feng X, Mao Y, Zhang F, Zhao H, Zeng H.

J Acquir Immune Defic Syndr. 2009 Dec;52(5):553-9.

PMID:
19950429
14.

Differential expression of CD163 on monocyte subsets in healthy and HIV-1 infected individuals.

Tippett E, Cheng WJ, Westhorpe C, Cameron PU, Brew BJ, Lewin SR, Jaworowski A, Crowe SM.

PLoS One. 2011;6(5):e19968. doi: 10.1371/journal.pone.0019968. Epub 2011 May 20.

15.
16.

Elevated plasma soluble CD14 and skewed CD16+ monocyte distribution persist despite normalisation of soluble CD163 and CXCL10 by effective HIV therapy: a changing paradigm for routine HIV laboratory monitoring?

Castley A, Berry C, French M, Fernandez S, Krueger R, Nolan D.

PLoS One. 2014 Dec 29;9(12):e115226. doi: 10.1371/journal.pone.0115226. eCollection 2014.

17.

Cocaine hijacks σ1 receptor to initiate induction of activated leukocyte cell adhesion molecule: implication for increased monocyte adhesion and migration in the CNS.

Yao H, Kim K, Duan M, Hayashi T, Guo M, Morgello S, Prat A, Wang J, Su TP, Buch S.

J Neurosci. 2011 Apr 20;31(16):5942-55. doi: 10.1523/JNEUROSCI.5618-10.2011.

18.

The CD16+ monocyte subset is more permissive to infection and preferentially harbors HIV-1 in vivo.

Ellery PJ, Tippett E, Chiu YL, Paukovics G, Cameron PU, Solomon A, Lewin SR, Gorry PR, Jaworowski A, Greene WC, Sonza S, Crowe SM.

J Immunol. 2007 May 15;178(10):6581-9.

19.

Glucocorticoid treatment at moderate doses of SIVmac251-infected rhesus macaques decreases the frequency of circulating CD14+CD16++ monocytes but does not alter the tissue virus reservoir.

Moniuszko M, Liyanage NP, Doster MN, Parks RW, Grubczak K, Lipinska D, McKinnon K, Brown C, Hirsch V, Vaccari M, Gordon S, Pegu P, Fenizia C, Flisiak R, Grzeszczuk A, Dabrowska M, Robert-Guroff M, Silvestri G, Stevenson M, McCune J, Franchini G.

AIDS Res Hum Retroviruses. 2015 Jan;31(1):115-26. doi: 10.1089/AID.2013.0220.

20.

CNS invasion by CD14+/CD16+ peripheral blood-derived monocytes in HIV dementia: perivascular accumulation and reservoir of HIV infection.

Fischer-Smith T, Croul S, Sverstiuk AE, Capini C, L'Heureux D, Régulier EG, Richardson MW, Amini S, Morgello S, Khalili K, Rappaport J.

J Neurovirol. 2001 Dec;7(6):528-41.

PMID:
11704885
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