Format
Sort by

Send to

Choose Destination

Links from PubMed

Items: 1 to 20 of 131

1.

Bee venom effects on ubiquitin proteasome system in hSOD1(G85R)-expressing NSC34 motor neuron cells.

Kim SH, Jung SY, Lee KW, Lee SH, Cai M, Choi SM, Yang EJ.

BMC Complement Altern Med. 2013 Jul 18;13:179. doi: 10.1186/1472-6882-13-179.

2.

Functional alterations of the ubiquitin-proteasome system in motor neurons of a mouse model of familial amyotrophic lateral sclerosis.

Cheroni C, Marino M, Tortarolo M, Veglianese P, De Biasi S, Fontana E, Zuccarello LV, Maynard CJ, Dantuma NP, Bendotti C.

Hum Mol Genet. 2009 Jan 1;18(1):82-96. doi: 10.1093/hmg/ddn319. Epub 2008 Sep 29.

3.
4.

Dysfunction of constitutive and inducible ubiquitin-proteasome system in amyotrophic lateral sclerosis: implication for protein aggregation and immune response.

Bendotti C, Marino M, Cheroni C, Fontana E, Crippa V, Poletti A, De Biasi S.

Prog Neurobiol. 2012 May;97(2):101-26. doi: 10.1016/j.pneurobio.2011.10.001. Epub 2011 Oct 20. Review.

PMID:
22033150
5.

Hypoxia causes autophagic stress and derangement of metabolic adaptation in a cell model of amyotrophic lateral sclerosis.

Cimini S, Rizzardini M, Biella G, Cantoni L.

J Neurochem. 2014 May;129(3):413-25. doi: 10.1111/jnc.12642. Epub 2014 Jan 22.

6.

The small heat shock protein B8 (HspB8) promotes autophagic removal of misfolded proteins involved in amyotrophic lateral sclerosis (ALS).

Crippa V, Sau D, Rusmini P, Boncoraglio A, Onesto E, Bolzoni E, Galbiati M, Fontana E, Marino M, Carra S, Bendotti C, De Biasi S, Poletti A.

Hum Mol Genet. 2010 Sep 1;19(17):3440-56. doi: 10.1093/hmg/ddq257. Epub 2010 Jun 22.

7.

Bee venom attenuates neuroinflammatory events and extends survival in amyotrophic lateral sclerosis models.

Yang EJ, Jiang JH, Lee SM, Yang SC, Hwang HS, Lee MS, Choi SM.

J Neuroinflammation. 2010 Oct 15;7:69. doi: 10.1186/1742-2094-7-69.

8.

CHIP promotes proteasomal degradation of familial ALS-linked mutant SOD1 by ubiquitinating Hsp/Hsc70.

Urushitani M, Kurisu J, Tateno M, Hatakeyama S, Nakayama K, Kato S, Takahashi R.

J Neurochem. 2004 Jul;90(1):231-44.

9.

Histological evidence of protein aggregation in mutant SOD1 transgenic mice and in amyotrophic lateral sclerosis neural tissues.

Watanabe M, Dykes-Hoberg M, Culotta VC, Price DL, Wong PC, Rothstein JD.

Neurobiol Dis. 2001 Dec;8(6):933-41.

PMID:
11741389
10.

Mitochondrial dysfunction in a cell culture model of familial amyotrophic lateral sclerosis.

Menzies FM, Cookson MR, Taylor RW, Turnbull DM, Chrzanowska-Lightowlers ZM, Dong L, Figlewicz DA, Shaw PJ.

Brain. 2002 Jul;125(Pt 7):1522-33.

11.

Muscle cells and motoneurons differentially remove mutant SOD1 causing familial amyotrophic lateral sclerosis.

Onesto E, Rusmini P, Crippa V, Ferri N, Zito A, Galbiati M, Poletti A.

J Neurochem. 2011 Jul;118(2):266-80. doi: 10.1111/j.1471-4159.2011.07298.x. Epub 2011 Jun 2.

12.

TDP-43 is consistently co-localized with ubiquitinated inclusions in sporadic and Guam amyotrophic lateral sclerosis but not in familial amyotrophic lateral sclerosis with and without SOD1 mutations.

Maekawa S, Leigh PN, King A, Jones E, Steele JC, Bodi I, Shaw CE, Hortobagyi T, Al-Sarraj S.

Neuropathology. 2009 Dec;29(6):672-83. doi: 10.1111/j.1440-1789.2009.01029.x. Epub 2009 Jun 3.

PMID:
19496940
13.

A role of small heat shock protein B8 (HspB8) in the autophagic removal of misfolded proteins responsible for neurodegenerative diseases.

Crippa V, Carra S, Rusmini P, Sau D, Bolzoni E, Bendotti C, De Biasi S, Poletti A.

Autophagy. 2010 Oct;6(7):958-60. doi: 10.4161/auto.6.7.13042. Epub 2010 Oct 16.

PMID:
20699640
14.

α -Synuclein Modification in an ALS Animal Model.

Yang EJ, Choi SM.

Evid Based Complement Alternat Med. 2013;2013:259381. doi: 10.1155/2013/259381. Epub 2013 May 12.

15.

Aberrant localization of FUS and TDP43 is associated with misfolding of SOD1 in amyotrophic lateral sclerosis.

Pokrishevsky E, Grad LI, Yousefi M, Wang J, Mackenzie IR, Cashman NR.

PLoS One. 2012;7(4):e35050. doi: 10.1371/journal.pone.0035050. Epub 2012 Apr 6.

16.
18.
19.

Absence of lipofuscin in motor neurons of SOD1-linked ALS mice.

Bandyopadhyay U, Nagy M, Fenton WA, Horwich AL.

Proc Natl Acad Sci U S A. 2014 Jul 29;111(30):11055-60. doi: 10.1073/pnas.1409314111. Epub 2014 Jul 14.

20.

VEGF-induced activation of the PI3-K/Akt pathway reduces mutant SOD1-mediated motor neuron cell death.

Li B, Xu W, Luo C, Gozal D, Liu R.

Brain Res Mol Brain Res. 2003 Mar 17;111(1-2):155-64.

PMID:
12654515
Items per page

Supplemental Content

Write to the Help Desk