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Items: 1 to 20 of 95

1.

Glucose-induced gradual phenotypic modulation of cultured human glomerular epithelial cells may be independent of Wilms' tumor 1 (WT1).

Tsotakos NE, Sagnou M, Kotsopoulou ES, Tsilibary EC, Drossopoulou GI.

BMC Cell Biol. 2013 Jun 14;14:28. doi: 10.1186/1471-2121-14-28.

2.

Vitamin D receptor activators upregulate and rescue podocalyxin expression in high glucose-treated human podocytes.

Verouti SN, Tsilibary EC, Fragopoulou E, Iatrou C, Demopoulos CA, Charonis AS, Charonis SA, Drossopoulou GI.

Nephron Exp Nephrol. 2012;122(1-2):36-50. doi: 10.1159/000346562. Epub 2013 Mar 14.

PMID:
23548800
3.

Impaired transcription factor interplay in addition to advanced glycation end products suppress podocalyxin expression in high glucose-treated human podocytes.

Drossopoulou GI, Tsotakos NE, Tsilibary EC.

Am J Physiol Renal Physiol. 2009 Sep;297(3):F594-603. doi: 10.1152/ajprenal.00203.2009. Epub 2009 Jul 15.

4.

PINCH1 is transcriptional regulator in podocytes that interacts with WT1 and represses podocalyxin expression.

Wang D, Li Y, Wu C, Liu Y.

PLoS One. 2011 Feb 24;6(2):e17048. doi: 10.1371/journal.pone.0017048.

5.

Variable expression of podocyte-related markers in the glomeruloid bodies in Wilms tumor.

Kanemoto K, Takahashi S, Shu Y, Usui J, Tomari S, Yan K, Hamazaki Y, Nagata M.

Pathol Int. 2003 Sep;53(9):596-601.

PMID:
14507316
6.

Cell differentiation in Wilms' tumor (nephroblastoma): an immunohistochemical study.

Droz D, Rousseau-Merck MF, Jaubert F, Diebold N, Nezelof C, Adafer E, Mouly H.

Hum Pathol. 1990 May;21(5):536-44.

PMID:
1692563
7.

Murine Denys-Drash syndrome: evidence of podocyte de-differentiation and systemic mediation of glomerulosclerosis.

Patek CE, Fleming S, Miles CG, Bellamy CO, Ladomery M, Spraggon L, Mullins J, Hastie ND, Hooper ML.

Hum Mol Genet. 2003 Sep 15;12(18):2379-94. Epub 2003 Jul 22.

8.

[High glucose downregulates the expression of podocalyxin protein in glomerular podocytes of mice].

Qi J, Xiao YF, Zhang DJ, Yang GR, Huang HC.

Beijing Da Xue Xue Bao. 2007 Apr 18;39(2):167-70. Chinese.

9.

Intrauterine growth restriction leads to a dysregulation of Wilms' tumour supressor gene 1 (WT1) and to early podocyte alterations.

Menendez-Castro C, Hilgers KF, Amann K, Daniel C, Cordasic N, Wachtveitl R, Fahlbusch F, Plank C, Dötsch J, Rascher W, Hartner A.

Nephrol Dial Transplant. 2013 Jun;28(6):1407-17. doi: 10.1093/ndt/gfs517. Epub 2012 Dec 9.

10.

Fluvastatin prevents podocyte injury in a murine model of HIV-associated nephropathy.

Sakurai N, Kuroiwa T, Ikeuchi H, Hiramatsu N, Takeuchi S, Tomioka M, Shigehara T, Maeshima A, Kaneko Y, Hiromura K, Kopp JB, Nojima Y.

Nephrol Dial Transplant. 2009 Aug;24(8):2378-83. doi: 10.1093/ndt/gfp012. Epub 2009 Feb 2.

11.

WT1 regulates the expression of the major glomerular podocyte membrane protein Podocalyxin.

Palmer RE, Kotsianti A, Cadman B, Boyd T, Gerald W, Haber DA.

Curr Biol. 2001 Nov 13;11(22):1805-9.

12.

Mutual antagonism of Wilms' tumor 1 and β-catenin dictates podocyte health and disease.

Zhou L, Li Y, He W, Zhou D, Tan RJ, Nie J, Hou FF, Liu Y.

J Am Soc Nephrol. 2015 Mar;26(3):677-91. doi: 10.1681/ASN.2013101067. Epub 2014 Jul 28.

13.

Stretch reduces nephrin expression via an angiotensin II-AT(1)-dependent mechanism in human podocytes: effect of rosiglitazone.

Miceli I, Burt D, Tarabra E, Camussi G, Perin PC, Gruden G.

Am J Physiol Renal Physiol. 2010 Feb;298(2):F381-90. doi: 10.1152/ajprenal.90423.2008. Epub 2009 Nov 11.

14.

Severe intraglomerular detachment of podocytes in a Gitelman syndrome patient.

Takahashi N, Kimura H, Mizuno S, Hara M, Hirayama Y, Kurosawa H, Tsutaya S, Yasujima M, Yokoi S, Mikami D, Kasuno K, Naiki H, Iwano M, Yoshida H.

Clin Exp Nephrol. 2012 Jun;16(3):495-500. doi: 10.1007/s10157-012-0624-4. Epub 2012 Apr 7.

PMID:
22484642
15.

The major podocyte protein nephrin is transcriptionally activated by the Wilms' tumor suppressor WT1.

Wagner N, Wagner KD, Xing Y, Scholz H, Schedl A.

J Am Soc Nephrol. 2004 Dec;15(12):3044-51.

16.

Enhanced podocalyxin expression alters the structure of podocyte basal surface.

Economou CG, Kitsiou PV, Tzinia AK, Panagopoulou E, Marinos E, Kershaw DB, Kerjaschki D, Tsilibary EC.

J Cell Sci. 2004 Jul 1;117(Pt 15):3281-94.

17.

Parietal podocytes in normal human glomeruli.

Bariety J, Mandet C, Hill GS, Bruneval P.

J Am Soc Nephrol. 2006 Oct;17(10):2770-80. Epub 2006 Aug 30.

18.

novH: differential expression in developing kidney and Wilm's tumors.

Chevalier G, Yeger H, Martinerie C, Laurent M, Alami J, Schofield PN, Perbal B.

Am J Pathol. 1998 Jun;152(6):1563-75.

19.

Marker expression, behaviors, and responses vary in different lines of conditionally immortalized cultured podocytes.

Chittiprol S, Chen P, Petrovic-Djergovic D, Eichler T, Ransom RF.

Am J Physiol Renal Physiol. 2011 Sep;301(3):F660-71. doi: 10.1152/ajprenal.00234.2011. Epub 2011 Jun 1.

20.

Transcription of nephrin-Neph3 gene pair is synergistically activated by WT1 and NF-κB and silenced by DNA methylation.

Ristola M, Arpiainen S, Saleem MA, Holthöfer H, Lehtonen S.

Nephrol Dial Transplant. 2012 May;27(5):1737-45. doi: 10.1093/ndt/gfr576. Epub 2011 Oct 6.

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