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Results: 1 to 20 of 166

Similar articles for PubMed (Select 23751316)

1.

Clinical and morphological characteristics of sporadic genetically determined pancreatitis as compared to idiopathic pancreatitis: higher risk of pancreatic cancer in CFTR variants.

Hamoir C, Pepermans X, Piessevaux H, Jouret-Mourin A, Weynand B, Habyalimana JB, Leal T, Geubel A, Gigot JF, Deprez PH.

Digestion. 2013;87(4):229-39. doi: 10.1159/000348439. Epub 2013 Jun 4.

PMID:
23751316
2.

Genetic prevalence and characteristics in children with recurrent pancreatitis.

Sultan M, Werlin S, Venkatasubramani N.

J Pediatr Gastroenterol Nutr. 2012 May;54(5):645-50. doi: 10.1097/MPG.0b013e31823f0269.

PMID:
22094894
3.

CFTR, SPINK1, PRSS1, and CTRC mutations are not associated with pancreatic cancer in German patients.

Schubert S, Traub F, Brakensiek K, von Kopylow K, Marohn B, Maelzer M, Gaedcke J, Kreipe H, Stuhrmann M.

Pancreas. 2014 Oct;43(7):1078-82. doi: 10.1097/MPA.0000000000000166.

PMID:
25003218
4.

Combined bicarbonate conductance-impairing variants in CFTR and SPINK1 variants are associated with chronic pancreatitis in patients without cystic fibrosis.

Schneider A, Larusch J, Sun X, Aloe A, Lamb J, Hawes R, Cotton P, Brand RE, Anderson MA, Money ME, Banks PA, Lewis MD, Baillie J, Sherman S, Disario J, Burton FR, Gardner TB, Amann ST, Gelrud A, George R, Rockacy MJ, Kassabian S, Martinson J, Slivka A, Yadav D, Oruc N, Barmada MM, Frizzell R, Whitcomb DC.

Gastroenterology. 2011 Jan;140(1):162-71. doi: 10.1053/j.gastro.2010.10.045. Epub 2010 Oct 25.

5.

Contribution of the CFTR gene, the pancreatic secretory trypsin inhibitor gene (SPINK1) and the cationic trypsinogen gene (PRSS1) to the etiology of recurrent pancreatitis.

Tzetis M, Kaliakatsos M, Fotoulaki M, Papatheodorou A, Doudounakis S, Tsezou A, Makrythanasis P, Kanavakis E, Nousia-Arvanitakis S.

Clin Genet. 2007 May;71(5):451-7.

PMID:
17489851
6.

Association of SPINK1 gene mutation and CFTR gene polymorphisms in patients with pancreas divisum presenting with idiopathic pancreatitis.

Garg PK, Khajuria R, Kabra M, Shastri SS.

J Clin Gastroenterol. 2009 Oct;43(9):848-52. doi: 10.1097/MCG.0b013e3181a4e772.

PMID:
19593166
7.

Hereditary pancreatitis in children: surgical implications with special regard to genetic background.

Schmitt F, Le Henaff G, Piloquet H, Leclair MD, David A, Heloury Y, Podevin G.

J Pediatr Surg. 2009 Nov;44(11):2078-82. doi: 10.1016/j.jpedsurg.2009.05.023.

PMID:
19944211
8.

Identification of CFTR, PRSS1, and SPINK1 mutations in 381 patients with pancreatitis.

Keiles S, Kammesheidt A.

Pancreas. 2006 Oct;33(3):221-7.

PMID:
17003641
9.

The variable phenotype of the p.A16V mutation of cationic trypsinogen (PRSS1) in pancreatitis families.

Grocock CJ, Rebours V, Delhaye MN, Andrén-Sandberg A, Weiss FU, Mountford R, Harcus MJ, Niemczyck E, Vitone LJ, Dodd S, Jørgensen MT, Ammann RW, Schaffalitzky de Muckadell O, Butler JV, Burgess P, Kerr B, Charnley R, Sutton R, Raraty MG, Devière J, Whitcomb DC, Neoptolemos JP, Lévy P, Lerch MM, Greenhalf W; European Registry of Hereditary Pancreatitis and Pancreatic Cancer.

Gut. 2010 Mar;59(3):357-63. doi: 10.1136/gut.2009.186817. Epub 2009 Dec 1.

PMID:
19951905
10.

Polymorphisms of SPINK1 N34S and CFTR in patients with sporadic and familial pancreatic cancer.

Matsubayashi H, Fukushima N, Sato N, Brune K, Canto M, Yeo CJ, Hruban RH, Kern SE, Goggins M.

Cancer Biol Ther. 2003 Nov-Dec;2(6):652-5.

11.

Complete cystic fibrosis transmembrane conductance regulator gene sequencing in patients with idiopathic chronic pancreatitis and controls.

Weiss FU, Simon P, Bogdanova N, Mayerle J, Dworniczak B, Horst J, Lerch MM.

Gut. 2005 Oct;54(10):1456-60. Epub 2005 Jun 29.

12.

CFTR, PRSS1 and SPINK1 mutations in the development of pancreatitis in Brazilian patients.

Bernardino AL, Guarita DR, Mott CB, Pedroso MR, Machado MC, Laudanna AA, Tani CM, Almeida FL, Zatz M.

JOP. 2003 Sep;4(5):169-77.

13.

Hereditary chronic pancreatitis.

Rosendahl J, Bödeker H, Mössner J, Teich N.

Orphanet J Rare Dis. 2007 Jan 4;2:1. Review.

14.

CFTR, SPINK1, CTRC and PRSS1 variants in chronic pancreatitis: is the role of mutated CFTR overestimated?

Rosendahl J, Landt O, Bernadova J, Kovacs P, Teich N, Bödeker H, Keim V, Ruffert C, Mössner J, Kage A, Stumvoll M, Groneberg D, Krüger R, Luck W, Treiber M, Becker M, Witt H.

Gut. 2013 Apr;62(4):582-92. doi: 10.1136/gutjnl-2011-300645. Epub 2012 Mar 17.

PMID:
22427236
15.

Genetic, epidemiological, and clinical aspects of hereditary pancreatitis: a population-based cohort study in Denmark.

Joergensen MT, Brusgaard K, Crüger DG, Gerdes AM, Schaffalitzky de Muckadell OB.

Am J Gastroenterol. 2010 Aug;105(8):1876-83. doi: 10.1038/ajg.2010.193. Epub 2010 May 25.

PMID:
20502448
16.

Cystic fibrosis transmembrane conductance regulator (CFTR) gene mutations and risk for pancreatic adenocarcinoma.

McWilliams RR, Petersen GM, Rabe KG, Holtegaard LM, Lynch PJ, Bishop MD, Highsmith WE Jr.

Cancer. 2010 Jan 1;116(1):203-9. doi: 10.1002/cncr.24697.

17.

Idiopathic chronic pancreatitis in India: phenotypic characterisation and strong genetic susceptibility due to SPINK1 and CFTR gene mutations.

Midha S, Khajuria R, Shastri S, Kabra M, Garg PK.

Gut. 2010 Jun;59(6):800-7. doi: 10.1136/gut.2009.191239.

PMID:
20551465
19.

Analysis of CFTR, SPINK1, PRSS1 and AAT mutations in children with acute or chronic pancreatitis.

Sobczyńska-Tomaszewska A, Bak D, Oralewska B, Oracz G, Norek A, Czerska K, Mazurczak T, Teisseyre M, Socha J, Zagulski M, Bal J.

J Pediatr Gastroenterol Nutr. 2006 Sep;43(3):299-306.

PMID:
16954950
20.

Pancreas divisum is not a cause of pancreatitis by itself but acts as a partner of genetic mutations.

Bertin C, Pelletier AL, Vullierme MP, Bienvenu T, Rebours V, Hentic O, Maire F, Hammel P, Vilgrain V, Ruszniewski P, Lévy P.

Am J Gastroenterol. 2012 Feb;107(2):311-7. doi: 10.1038/ajg.2011.424. Epub 2011 Dec 13.

PMID:
22158025
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