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Items: 1 to 20 of 104


Salmonella uses energy taxis to benefit from intestinal inflammation.

Rivera-Chávez F, Winter SE, Lopez CA, Xavier MN, Winter MG, Nuccio SP, Russell JM, Laughlin RC, Lawhon SD, Sterzenbach T, Bevins CL, Tsolis RM, Harshey R, Adams LG, Bäumler AJ.

PLoS Pathog. 2013;9(4):e1003267. doi: 10.1371/journal.ppat.1003267. Epub 2013 Apr 18.


Phage-mediated acquisition of a type III secreted effector protein boosts growth of salmonella by nitrate respiration.

Lopez CA, Winter SE, Rivera-Chávez F, Xavier MN, Poon V, Nuccio SP, Tsolis RM, Bäumler AJ.

MBio. 2012 Jun 12;3(3). pii: e00143-12. doi: 10.1128/mBio.00143-12. Print 2012.


Gut inflammation provides a respiratory electron acceptor for Salmonella.

Winter SE, Thiennimitr P, Winter MG, Butler BP, Huseby DL, Crawford RW, Russell JM, Bevins CL, Adams LG, Tsolis RM, Roth JR, Bäumler AJ.

Nature. 2010 Sep 23;467(7314):426-9. doi: 10.1038/nature09415.


Salmonella "sops" up a preferred electron receptor in the inflamed intestine.

Bliska JB, van der Velden AW.

MBio. 2012 Aug 14;3(4):e00226-12. doi: 10.1128/mBio.00226-12. Print 2012.


Very long O-antigen chains enhance fitness during Salmonella-induced colitis by increasing bile resistance.

Crawford RW, Keestra AM, Winter SE, Xavier MN, Tsolis RM, Tolstikov V, Bäumler AJ.

PLoS Pathog. 2012 Sep;8(9):e1002918. doi: 10.1371/journal.ppat.1002918. Epub 2012 Sep 20.


Intestinal inflammation allows Salmonella to use ethanolamine to compete with the microbiota.

Thiennimitr P, Winter SE, Winter MG, Xavier MN, Tolstikov V, Huseby DL, Sterzenbach T, Tsolis RM, Roth JR, Bäumler AJ.

Proc Natl Acad Sci U S A. 2011 Oct 18;108(42):17480-5. doi: 10.1073/pnas.1107857108. Epub 2011 Oct 3.


Limited role for ASC and NLRP3 during in vivo Salmonella Typhimurium infection.

De Jong HK, Koh GC, van Lieshout MH, Roelofs JJ, van Dissel JT, van der Poll T, Wiersinga WJ.

BMC Immunol. 2014 Aug 13;15:30. doi: 10.1186/s12865-014-0030-7.


Aer and Tsr guide Escherichia coli in spatial gradients of oxidizable substrates.

Greer-Phillips SE, Alexandre G, Taylor BL, Zhulin IB.

Microbiology. 2003 Sep;149(Pt 9):2661-7.


Protective role of Akt2 in Salmonella enterica serovar typhimurium-induced gastroenterocolitis.

Kum WW, Lo BC, Yu HB, Finlay BB.

Infect Immun. 2011 Jul;79(7):2554-66. doi: 10.1128/IAI.01235-10. Epub 2011 May 9.


Granulocytes impose a tight bottleneck upon the gut luminal pathogen population during Salmonella typhimurium colitis.

Maier L, Diard M, Sellin ME, Chouffane ES, Trautwein-Weidner K, Periaswamy B, Slack E, Dolowschiak T, Stecher B, Loverdo C, Regoes RR, Hardt WD.

PLoS Pathog. 2014 Dec 18;10(12):e1004557. doi: 10.1371/journal.ppat.1004557. eCollection 2014 Dec. Erratum in: PLoS Pathog. 2015 Jul;11(7):e1005047.


Electron acceptor taxis and blue light effect on bacterial chemotaxis.

Taylor BL, Miller JB, Warrick HM, Koshland DE Jr.

J Bacteriol. 1979 Nov;140(2):567-73.


Aerotaxis in Salmonella typhimurium: role of electron transport.

Laszlo DJ, Taylor BL.

J Bacteriol. 1981 Feb;145(2):990-1001.


Early MyD88-dependent induction of interleukin-17A expression during Salmonella colitis.

Keestra AM, Godinez I, Xavier MN, Winter MG, Winter SE, Tsolis RM, Bäumler AJ.

Infect Immun. 2011 Aug;79(8):3131-40. doi: 10.1128/IAI.00018-11. Epub 2011 May 16.


Coinfection with an intestinal helminth impairs host innate immunity against Salmonella enterica serovar Typhimurium and exacerbates intestinal inflammation in mice.

Su L, Su CW, Qi Y, Yang G, Zhang M, Cherayil BJ, Zhang X, Shi HN.

Infect Immun. 2014 Sep;82(9):3855-66. doi: 10.1128/IAI.02023-14. Epub 2014 Jun 30.


A breathtaking feat: to compete with the gut microbiota, Salmonella drives its host to provide a respiratory electron acceptor.

Winter SE, Bäumler AJ.

Gut Microbes. 2011 Jan-Feb;2(1):58-60. doi: 10.4161/gmic.2.1.14911.


Salmonella enterica serotype Typhimurium MisL is an intestinal colonization factor that binds fibronectin.

Dorsey CW, Laarakker MC, Humphries AD, Weening EH, Bäumler AJ.

Mol Microbiol. 2005 Jul;57(1):196-211.


Flagella and chemotaxis are required for efficient induction of Salmonella enterica serovar Typhimurium colitis in streptomycin-pretreated mice.

Stecher B, Hapfelmeier S, Müller C, Kremer M, Stallmach T, Hardt WD.

Infect Immun. 2004 Jul;72(7):4138-50.


The Salmonella pathogenicity island (SPI)-2 and SPI-1 type III secretion systems allow Salmonella serovar typhimurium to trigger colitis via MyD88-dependent and MyD88-independent mechanisms.

Hapfelmeier S, Stecher B, Barthel M, Kremer M, Müller AJ, Heikenwalder M, Stallmach T, Hensel M, Pfeffer K, Akira S, Hardt WD.

J Immunol. 2005 Feb 1;174(3):1675-85.


Intestinal invasion of Salmonella enterica serovar Typhimurium in the avian host is dose dependent and does not depend on motility and chemotaxis.

Olsen JE, Hoegh-Andersen KH, Rosenkrantz JT, Schroll C, Casadesús J, Aabo S, Christensen JP.

Vet Microbiol. 2013 Aug 30;165(3-4):373-7. doi: 10.1016/j.vetmic.2013.04.008. Epub 2013 Apr 12.


Genome expression analysis of nonproliferating intracellular Salmonella enterica serovar Typhimurium unravels an acid pH-dependent PhoP-PhoQ response essential for dormancy.

Núñez-Hernández C, Tierrez A, Ortega AD, Pucciarelli MG, Godoy M, Eisman B, Casadesús J, García-del Portillo F.

Infect Immun. 2013 Jan;81(1):154-65. doi: 10.1128/IAI.01080-12. Epub 2012 Oct 22.

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