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Results: 1 to 20 of 192

Similar articles for PubMed (Select 23536180)

1.

Molecular architecture of a sodium channel S6 helix: radial tuning of the voltage-gated sodium channel 1.7 activation gate.

Yang Y, Estacion M, Dib-Hajj SD, Waxman SG.

J Biol Chem. 2013 May 10;288(19):13741-7. doi: 10.1074/jbc.M113.462366. Epub 2013 Mar 27.

2.

Deletion mutation of sodium channel Na(V)1.7 in inherited erythromelalgia: enhanced slow inactivation modulates dorsal root ganglion neuron hyperexcitability.

Cheng X, Dib-Hajj SD, Tyrrell L, Te Morsche RH, Drenth JP, Waxman SG.

Brain. 2011 Jul;134(Pt 7):1972-86. doi: 10.1093/brain/awr143.

3.

Structural basis of ion permeation gating in Slo2.1 K+ channels.

Garg P, Gardner A, Garg V, Sanguinetti MC.

J Gen Physiol. 2013 Nov;142(5):523-42. doi: 10.1085/jgp.201311064.

4.

A pore-blocking hydrophobic motif at the cytoplasmic aperture of the closed-state Nav1.7 channel is disrupted by the erythromelalgia-associated F1449V mutation.

Lampert A, O'Reilly AO, Dib-Hajj SD, Tyrrell L, Wallace BA, Waxman SG.

J Biol Chem. 2008 Aug 29;283(35):24118-27. doi: 10.1074/jbc.M802900200. Epub 2008 Jun 12.

5.

Erythromelalgia mutation Q875E Stabilizes the activated state of sodium channel Nav1.7.

Stadler T, O'Reilly AO, Lampert A.

J Biol Chem. 2015 Mar 6;290(10):6316-25. doi: 10.1074/jbc.M114.605899. Epub 2015 Jan 9.

PMID:
25575597
6.

Novel mutations mapping to the fourth sodium channel domain of Nav1.7 result in variable clinical manifestations of primary erythromelalgia.

Cregg R, Laguda B, Werdehausen R, Cox JJ, Linley JE, Ramirez JD, Bodi I, Markiewicz M, Howell KJ, Chen YC, Agnew K, Houlden H, Lunn MP, Bennett DL, Wood JN, Kinali M.

Neuromolecular Med. 2013 Jun;15(2):265-78. doi: 10.1007/s12017-012-8216-8. Epub 2013 Jan 6.

7.

Erythromelalgia mutation L823R shifts activation and inactivation of threshold sodium channel Nav1.7 to hyperpolarized potentials.

Lampert A, Dib-Hajj SD, Eastman EM, Tyrrell L, Lin Z, Yang Y, Waxman SG.

Biochem Biophys Res Commun. 2009 Dec 11;390(2):319-24. doi: 10.1016/j.bbrc.2009.09.121. Epub 2009 Oct 1.

PMID:
19800314
8.

Reversed voltage-dependent gating of a bacterial sodium channel with proline substitutions in the S6 transmembrane segment.

Zhao Y, Scheuer T, Catterall WA.

Proc Natl Acad Sci U S A. 2004 Dec 21;101(51):17873-8. Epub 2004 Dec 6.

9.

Dynamic-clamp analysis of wild-type human Nav1.7 and erythromelalgia mutant channel L858H.

Vasylyev DV, Han C, Zhao P, Dib-Hajj S, Waxman SG.

J Neurophysiol. 2014 Apr;111(7):1429-43. doi: 10.1152/jn.00763.2013. Epub 2014 Jan 8.

10.
11.

Arrangement and mobility of the voltage sensor domain in prokaryotic voltage-gated sodium channels.

Shimomura T, Irie K, Nagura H, Imai T, Fujiyoshi Y.

J Biol Chem. 2011 Mar 4;286(9):7409-17. doi: 10.1074/jbc.M110.186510. Epub 2010 Dec 22.

12.

Hinge-bending motions in the pore domain of a bacterial voltage-gated sodium channel.

Barber AF, Carnevale V, Raju SG, Amaral C, Treptow W, Klein ML.

Biochim Biophys Acta. 2012 Sep;1818(9):2120-5. doi: 10.1016/j.bbamem.2012.05.002. Epub 2012 May 9.

13.

A novel SCN9A mutation responsible for primary erythromelalgia and is resistant to the treatment of sodium channel blockers.

Wu MT, Huang PY, Yen CT, Chen CC, Lee MJ.

PLoS One. 2013;8(1):e55212. doi: 10.1371/journal.pone.0055212. Epub 2013 Jan 31.

14.

Subtype specificity of scorpion beta-toxin Tz1 interaction with voltage-gated sodium channels is determined by the pore loop of domain 3.

Leipold E, Hansel A, Borges A, Heinemann SH.

Mol Pharmacol. 2006 Jul;70(1):340-7. Epub 2006 Apr 25.

15.

Inherited pain: sodium channel Nav1.7 A1632T mutation causes erythromelalgia due to a shift of fast inactivation.

Eberhardt M, Nakajima J, Klinger AB, Neacsu C, Hühne K, O'Reilly AO, Kist AM, Lampe AK, Fischer K, Gibson J, Nau C, Winterpacht A, Lampert A.

J Biol Chem. 2014 Jan 24;289(4):1971-80. doi: 10.1074/jbc.M113.502211. Epub 2013 Dec 5.

16.

Mechanisms of a human skeletal myotonia produced by mutation in the C-terminus of NaV1.4: is Ca2+ regulation defective?

Biswas S, DiSilvestre DA, Dong P, Tomaselli GF.

PLoS One. 2013 Dec 6;8(12):e81063. doi: 10.1371/journal.pone.0081063. eCollection 2013.

17.

CRMP2 protein SUMOylation modulates NaV1.7 channel trafficking.

Dustrude ET, Wilson SM, Ju W, Xiao Y, Khanna R.

J Biol Chem. 2013 Aug 23;288(34):24316-31. doi: 10.1074/jbc.M113.474924. Epub 2013 Jul 8.

18.

Role of arginine residues on the S4 segment of the Bacillus halodurans Na+ channel in voltage-sensing.

Chahine M, Pilote S, Pouliot V, Takami H, Sato C.

J Membr Biol. 2004 Sep 1;201(1):9-24.

PMID:
15635808
19.

Differential effect of D623N variant and wild-type Na(v)1.7 sodium channels on resting potential and interspike membrane potential of dorsal root ganglion neurons.

Ahn HS, Vasylyev DV, Estacion M, Macala LJ, Shah P, Faber CG, Merkies IS, Dib-Hajj SD, Waxman SG.

Brain Res. 2013 Sep 5;1529:165-77. doi: 10.1016/j.brainres.2013.07.005. Epub 2013 Jul 11.

PMID:
23850641
20.

The region adjacent to the C-end of the inner gate in transient receptor potential melastatin 8 (TRPM8) channels plays a central role in allosteric channel activation.

Taberner FJ, López-Córdoba A, Fernández-Ballester G, Korchev Y, Ferrer-Montiel A.

J Biol Chem. 2014 Oct 10;289(41):28579-94. doi: 10.1074/jbc.M114.577478. Epub 2014 Aug 25.

PMID:
25157108
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