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Items: 1 to 20 of 151

1.

Abrogation of IL-4 receptor-α-dependent alternatively activated macrophages is sufficient to confer resistance against pulmonary cryptococcosis despite an ongoing T(h)2 response.

Müller U, Stenzel W, Piehler D, Grahnert A, Protschka M, Köhler G, Frey O, Held J, Richter T, Eschke M, Kamradt T, Brombacher F, Alber G.

Int Immunol. 2013 Aug;25(8):459-70. doi: 10.1093/intimm/dxt003. Epub 2013 Mar 26.

2.

Lack of IL-4 receptor expression on T helper cells reduces T helper 2 cell polyfunctionality and confers resistance in allergic bronchopulmonary mycosis.

Müller U, Piehler D, Stenzel W, Köhler G, Frey O, Held J, Grahnert A, Richter T, Eschke M, Kamradt T, Brombacher F, Alber G.

Mucosal Immunol. 2012 May;5(3):299-310. doi: 10.1038/mi.2012.9. Epub 2012 Feb 15.

PMID:
22333910
3.

IL-4Ralpha-independent expression of mannose receptor and Ym1 by macrophages depends on their IL-10 responsiveness.

Dewals BG, Marillier RG, Hoving JC, Leeto M, Schwegmann A, Brombacher F.

PLoS Negl Trop Dis. 2010 May 18;4(5):e689. doi: 10.1371/journal.pntd.0000689.

4.

Allergic airway disease is unaffected by the absence of IL-4Rα-dependent alternatively activated macrophages.

Nieuwenhuizen NE, Kirstein F, Jayakumar J, Emedi B, Hurdayal R, Horsnell WG, Lopata AL, Brombacher F.

J Allergy Clin Immunol. 2012 Sep;130(3):743-750.e8. doi: 10.1016/j.jaci.2012.03.011. Epub 2012 Apr 30.

PMID:
22552110
5.

IL-13 induces disease-promoting type 2 cytokines, alternatively activated macrophages and allergic inflammation during pulmonary infection of mice with Cryptococcus neoformans.

Müller U, Stenzel W, Köhler G, Werner C, Polte T, Hansen G, Schütze N, Straubinger RK, Blessing M, McKenzie AN, Brombacher F, Alber G.

J Immunol. 2007 Oct 15;179(8):5367-77.

6.

Role of IFN-gamma in regulating T2 immunity and the development of alternatively activated macrophages during allergic bronchopulmonary mycosis.

Arora S, Hernandez Y, Erb-Downward JR, McDonald RA, Toews GB, Huffnagle GB.

J Immunol. 2005 May 15;174(10):6346-56.

7.

Deletion of IL-4 receptor alpha on dendritic cells renders BALB/c mice hypersusceptible to Leishmania major infection.

Hurdayal R, Nieuwenhuizen NE, Revaz-Breton M, Smith L, Hoving JC, Parihar SP, Reizis B, Brombacher F.

PLoS Pathog. 2013 Oct;9(10):e1003699. doi: 10.1371/journal.ppat.1003699. Epub 2013 Oct 24.

8.
9.

Deletion of IL-4Ralpha on CD4 T cells renders BALB/c mice resistant to Leishmania major infection.

Radwanska M, Cutler AJ, Hoving JC, Magez S, Holscher C, Bohms A, Arendse B, Kirsch R, Hunig T, Alexander J, Kaye P, Brombacher F.

PLoS Pathog. 2007 May 11;3(5):e68.

10.

Effect of cytokine interplay on macrophage polarization during chronic pulmonary infection with Cryptococcus neoformans.

Arora S, Olszewski MA, Tsang TM, McDonald RA, Toews GB, Huffnagle GB.

Infect Immun. 2011 May;79(5):1915-26. doi: 10.1128/IAI.01270-10. Epub 2011 Mar 7.

11.

IL-4 receptor-alpha-dependent control of Cryptococcus neoformans in the early phase of pulmonary infection.

Grahnert A, Richter T, Piehler D, Eschke M, Schulze B, Müller U, Protschka M, Köhler G, Sabat R, Brombacher F, Alber G.

PLoS One. 2014 Jan 27;9(1):e87341. doi: 10.1371/journal.pone.0087341. eCollection 2014.

12.

Pulmonary infection with an interferon-gamma-producing Cryptococcus neoformans strain results in classical macrophage activation and protection.

Hardison SE, Ravi S, Wozniak KL, Young ML, Olszewski MA, Wormley FL Jr.

Am J Pathol. 2010 Feb;176(2):774-85. doi: 10.2353/ajpath.2010.090634. Epub 2010 Jan 7.

13.

Th2 but not Th1 immune bias results in altered lung functions in a murine model of pulmonary Cryptococcus neoformans infection.

Jain AV, Zhang Y, Fields WB, McNamara DA, Choe MY, Chen GH, Erb-Downward J, Osterholzer JJ, Toews GB, Huffnagle GB, Olszewski MA.

Infect Immun. 2009 Dec;77(12):5389-99. doi: 10.1128/IAI.00809-09. Epub 2009 Sep 14.

14.

BALB/c mice deficient in CD4 T cell IL-4Rα expression control Leishmania mexicana Load although female but not male mice develop a healer phenotype.

Bryson KJ, Millington OR, Mokgethi T, McGachy HA, Brombacher F, Alexander J.

PLoS Negl Trop Dis. 2011 Jan 4;5(1):e930. doi: 10.1371/journal.pntd.0000930.

15.

Insights into the mechanisms of protective immunity against Cryptococcus neoformans infection using a mouse model of pulmonary cryptococcosis.

Wozniak KL, Ravi S, Macias S, Young ML, Olszewski MA, Steele C, Wormley FL.

PLoS One. 2009 Sep 3;4(9):e6854. doi: 10.1371/journal.pone.0006854.

16.

Adoptive transfer of IL-4Rα+ macrophages is sufficient to enhance eosinophilic inflammation in a mouse model of allergic lung inflammation.

Ford AQ, Dasgupta P, Mikhailenko I, Smith EM, Noben-Trauth N, Keegan AD.

BMC Immunol. 2012 Jan 31;13:6. doi: 10.1186/1471-2172-13-6.

17.

B cells that produce immunoglobulin E mediate colitis in BALB/c mice.

Hoving JC, Kirstein F, Nieuwenhuizen NE, Fick LC, Hobeika E, Reth M, Brombacher F.

Gastroenterology. 2012 Jan;142(1):96-108. doi: 10.1053/j.gastro.2011.09.044. Epub 2011 Oct 6.

PMID:
21983080
18.

STAT1 signaling is essential for protection against Cryptococcus neoformans infection in mice.

Leopold Wager CM, Hole CR, Wozniak KL, Olszewski MA, Wormley FL Jr.

J Immunol. 2014 Oct 15;193(8):4060-71. doi: 10.4049/jimmunol.1400318. Epub 2014 Sep 8.

19.

A gene-dosage effect for interleukin-4 receptor alpha-chain expression has an impact on Th2-mediated allergic inflammation during bronchopulmonary mycosis.

Müller U, Stenzel W, Köhler G, Polte T, Blessing M, Mann A, Piehler D, Brombacher F, Alber G.

J Infect Dis. 2008 Dec 1;198(11):1714-21. doi: 10.1086/593068.

20.

Expression of IL-4 receptor alpha on smooth muscle cells is not necessary for development of experimental allergic asthma.

Kirstein F, Horsnell WG, Kuperman DA, Huang X, Erle DJ, Lopata AL, Brombacher F.

J Allergy Clin Immunol. 2010 Aug;126(2):347-54. doi: 10.1016/j.jaci.2010.04.028. Epub 2010 Jun 25.

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