Format
Items per page
Sort by

Send to:

Choose Destination

Links from PubMed

Items: 1 to 20 of 93

1.

Aquaporin 11 insufficiency modulates kidney susceptibility to oxidative stress.

Atochina-Vasserman EN, Biktasova A, Abramova E, Cheng DS, Polosukhin VV, Tanjore H, Takahashi S, Sonoda H, Foye L, Venkov C, Ryzhov SV, Novitskiy S, Shlonimskaya N, Ikeda M, Blackwell TS, Lawson WE, Gow AJ, Harris RC, Dikov MM, Tchekneva EE.

Am J Physiol Renal Physiol. 2013 May 15;304(10):F1295-307. doi: 10.1152/ajprenal.00344.2012. Epub 2013 Mar 13.

2.

Single amino acid substitution in aquaporin 11 causes renal failure.

Tchekneva EE, Khuchua Z, Davis LS, Kadkina V, Dunn SR, Bachman S, Ishibashi K, Rinchik EM, Harris RC, Dikov MM, Breyer MD.

J Am Soc Nephrol. 2008 Oct;19(10):1955-64. doi: 10.1681/ASN.2008030296. Epub 2008 Aug 13.

3.

Aberrant glycosylation and localization of polycystin-1 cause polycystic kidney in an AQP11 knockout model.

Inoue Y, Sohara E, Kobayashi K, Chiga M, Rai T, Ishibashi K, Horie S, Su X, Zhou J, Sasaki S, Uchida S.

J Am Soc Nephrol. 2014 Dec;25(12):2789-99. doi: 10.1681/ASN.2013060614. Epub 2014 May 22.

4.

Liver-specific Aquaporin 11 knockout mice show rapid vacuolization of the rough endoplasmic reticulum in periportal hepatocytes after amino acid feeding.

Rojek A, Füchtbauer EM, Füchtbauer A, Jelen S, Malmendal A, Fenton RA, Nielsen S.

Am J Physiol Gastrointest Liver Physiol. 2013 Mar 1;304(5):G501-15. doi: 10.1152/ajpgi.00208.2012. Epub 2012 Dec 28.

5.

Aquaporin-11 knockout mice and polycystic kidney disease animals share a common mechanism of cyst formation.

Okada S, Misaka T, Tanaka Y, Matsumoto I, Ishibashi K, Sasaki S, Abe K.

FASEB J. 2008 Oct;22(10):3672-84. doi: 10.1096/fj.08-111872. Epub 2008 Jul 7.

6.

Disruption of aquaporin-11 produces polycystic kidneys following vacuolization of the proximal tubule.

Morishita Y, Matsuzaki T, Hara-chikuma M, Andoo A, Shimono M, Matsuki A, Kobayashi K, Ikeda M, Yamamoto T, Verkman A, Kusano E, Ookawara S, Takata K, Sasaki S, Ishibashi K.

Mol Cell Biol. 2005 Sep;25(17):7770-9.

7.

Protective role for nitric oxide during the endoplasmic reticulum stress response in pancreatic beta-cells.

Kitiphongspattana K, Khan TA, Ishii-Schrade K, Roe MW, Philipson LH, Gaskins HR.

Am J Physiol Endocrinol Metab. 2007 Jun;292(6):E1543-54. Epub 2007 Jan 30.

8.

Reactive oxygen species promote caspase-12 expression and tubular apoptosis in diabetic nephropathy.

Brezniceanu ML, Lau CJ, Godin N, Chénier I, Duclos A, Ethier J, Filep JG, Ingelfinger JR, Zhang SL, Chan JS.

J Am Soc Nephrol. 2010 Jun;21(6):943-54. doi: 10.1681/ASN.2009030242. Epub 2010 Mar 18.

9.

The distribution and function of aquaporins in the kidney: resolved and unresolved questions.

Matsuzaki T, Yaguchi T, Shimizu K, Kita A, Ishibashi K, Takata K.

Anat Sci Int. 2016 Jan 21. [Epub ahead of print]

PMID:
26798062
10.

Kidney-specific overexpression of Sirt1 protects against acute kidney injury by retaining peroxisome function.

Hasegawa K, Wakino S, Yoshioka K, Tatematsu S, Hara Y, Minakuchi H, Sueyasu K, Washida N, Tokuyama H, Tzukerman M, Skorecki K, Hayashi K, Itoh H.

J Biol Chem. 2010 Apr 23;285(17):13045-56. doi: 10.1074/jbc.M109.067728. Epub 2010 Feb 5.

11.

Effect of BSA-induced ER stress on SGLT protein expression levels and alpha-MG uptake in renal proximal tubule cells.

Lee YJ, Suh HN, Han HJ.

Am J Physiol Renal Physiol. 2009 Jun;296(6):F1405-16. doi: 10.1152/ajprenal.90652.2008. Epub 2009 Feb 11.

12.

Berberine protects human renal proximal tubular cells from hypoxia/reoxygenation injury via inhibiting endoplasmic reticulum and mitochondrial stress pathways.

Yu W, Sheng M, Xu R, Yu J, Cui K, Tong J, Shi L, Ren H, Du H.

J Transl Med. 2013 Jan 29;11:24. doi: 10.1186/1479-5876-11-24.

13.

The NPC motif of aquaporin-11, unlike the NPA motif of known aquaporins, is essential for full expression of molecular function.

Ikeda M, Andoo A, Shimono M, Takamatsu N, Taki A, Muta K, Matsushita W, Uechi T, Matsuzaki T, Kenmochi N, Takata K, Sasaki S, Ito K, Ishibashi K.

J Biol Chem. 2011 Feb 4;286(5):3342-50. doi: 10.1074/jbc.M110.180968. Epub 2010 Nov 30.

14.

Sestrin2 decreases renal oxidative stress, lowers blood pressure, and mediates dopamine D2 receptor-induced inhibition of reactive oxygen species production.

Yang Y, Cuevas S, Yang S, Villar VA, Escano C, Asico L, Yu P, Jiang X, Weinman EJ, Armando I, Jose PA.

Hypertension. 2014 Oct;64(4):825-32. doi: 10.1161/HYPERTENSIONAHA.114.03840. Epub 2014 Jul 14.

15.

Modulation of albumin-induced endoplasmic reticulum stress in renal proximal tubule cells by upregulation of mapk phosphatase-1.

Gorostizaga A, Mori Sequeiros García MM, Acquier A, Gomez NV, Maloberti PM, Mendez CF, Paz C.

Chem Biol Interact. 2013 Oct 25;206(1):47-54. doi: 10.1016/j.cbi.2013.08.009. Epub 2013 Aug 29.

PMID:
23994741
16.

Gender differences control the susceptibility to ER stress-induced acute kidney injury.

Hodeify R, Megyesi J, Tarcsafalvi A, Mustafa HI, Hti Lar Seng NS, Price PM.

Am J Physiol Renal Physiol. 2013 Apr 1;304(7):F875-82. doi: 10.1152/ajprenal.00590.2012. Epub 2013 Jan 30.

17.

p66SHC-mediated mitochondrial dysfunction in renal proximal tubule cells during oxidative injury.

Arany I, Faisal A, Clark JS, Vera T, Baliga R, Nagamine Y.

Am J Physiol Renal Physiol. 2010 May;298(5):F1214-21. doi: 10.1152/ajprenal.00639.2009. Epub 2010 Jan 6.

18.

Downregulation of miR-205 modulates cell susceptibility to oxidative and endoplasmic reticulum stresses in renal tubular cells.

Muratsu-Ikeda S, Nangaku M, Ikeda Y, Tanaka T, Wada T, Inagi R.

PLoS One. 2012;7(7):e41462. doi: 10.1371/journal.pone.0041462. Epub 2012 Jul 30.

19.

Reactive oxygen species-mediated endoplasmic reticulum stress contributes to aldosterone-induced apoptosis in tubular epithelial cells.

Ding W, Yang L, Zhang M, Gu Y.

Biochem Biophys Res Commun. 2012 Feb 17;418(3):451-6. doi: 10.1016/j.bbrc.2012.01.037. Epub 2012 Jan 18.

PMID:
22281495
20.

Endoplasmic reticulum stress implicated in the development of renal fibrosis.

Chiang CK, Hsu SP, Wu CT, Huang JW, Cheng HT, Chang YW, Hung KY, Wu KD, Liu SH.

Mol Med. 2011;17(11-12):1295-305. doi: 10.2119/molmed.2011.00131. Epub 2011 Aug 19.

Format
Items per page
Sort by

Send to:

Choose Destination

Supplemental Content

Write to the Help Desk