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Results: 1 to 20 of 85

Similar articles for PubMed (Select 23449591)

1.

Interferon-ε protects the female reproductive tract from viral and bacterial infection.

Fung KY, Mangan NE, Cumming H, Horvat JC, Mayall JR, Stifter SA, De Weerd N, Roisman LC, Rossjohn J, Robertson SA, Schjenken JE, Parker B, Gargett CE, Nguyen HP, Carr DJ, Hansbro PM, Hertzog PJ.

Science. 2013 Mar 1;339(6123):1088-92. doi: 10.1126/science.1233321.

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IFN-ε is constitutively expressed by cells of the reproductive tract and is inefficiently secreted by fibroblasts and cell lines.

Hermant P, Francius C, Clotman F, Michiels T.

PLoS One. 2013 Aug 9;8(8):e71320. doi: 10.1371/journal.pone.0071320. eCollection 2013.

5.

Expression of type III interferon (IFN) in the vaginal mucosa is mediated primarily by dendritic cells and displays stronger dependence on NF-kappaB than type I IFNs.

Iversen MB, Ank N, Melchjorsen J, Paludan SR.

J Virol. 2010 May;84(9):4579-86. doi: 10.1128/JVI.02591-09. Epub 2010 Feb 24.

6.

A critical role for IL-15 in TLR-mediated innate antiviral immunity against genital HSV-2 infection.

Thatte A, DeWitte-Orr SJ, Lichty B, Mossman KL, Ashkar AA.

Immunol Cell Biol. 2011 Aug;89(6):663-9. doi: 10.1038/icb.2011.7. Epub 2011 Feb 22.

PMID:
21339766
7.

The Chlamydia muridarum-induced IFN-β response is TLR3-dependent in murine oviduct epithelial cells.

Derbigny WA, Johnson RM, Toomey KS, Ofner S, Jayarapu K.

J Immunol. 2010 Dec 1;185(11):6689-97. doi: 10.4049/jimmunol.1001548. Epub 2010 Oct 25.

8.

The role of toll-like receptor ligands/agonists in protection against genital HSV-2 infection.

Gill N, Davies EJ, Ashkar AA.

Am J Reprod Immunol. 2008 Jan;59(1):35-43. Review.

PMID:
18154594
9.

Identifying a role for Toll-like receptor 3 in the innate immune response to Chlamydia muridarum infection in murine oviduct epithelial cells.

Derbigny WA, Shobe LR, Kamran JC, Toomey KS, Ofner S.

Infect Immun. 2012 Jan;80(1):254-65. doi: 10.1128/IAI.05549-11. Epub 2011 Oct 17.

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Toll-like receptor (TLR)-3, but not TLR4, agonist protects against genital herpes infection in the absence of inflammation seen with CpG DNA.

Ashkar AA, Yao XD, Gill N, Sajic D, Patrick AJ, Rosenthal KL.

J Infect Dis. 2004 Nov 15;190(10):1841-9. Epub 2004 Oct 18.

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Antigen-specific CD4+ T cells produce sufficient IFN-gamma to mediate robust protective immunity against genital Chlamydia muridarum infection.

Li W, Murthy AK, Guentzel MN, Seshu J, Forsthuber TG, Zhong G, Arulanandam BP.

J Immunol. 2008 Mar 1;180(5):3375-82.

14.

An important role for type III interferon (IFN-lambda/IL-28) in TLR-induced antiviral activity.

Ank N, Iversen MB, Bartholdy C, Staeheli P, Hartmann R, Jensen UB, Dagnaes-Hansen F, Thomsen AR, Chen Z, Haugen H, Klucher K, Paludan SR.

J Immunol. 2008 Feb 15;180(4):2474-85.

15.

Herpes simplex virus 2-induced activation in vaginal cells involves Toll-like receptors 2 and 9 and DNA sensors DAI and IFI16.

Triantafilou K, Eryilmazlar D, Triantafilou M.

Am J Obstet Gynecol. 2014 Feb;210(2):122.e1-122.e10. doi: 10.1016/j.ajog.2013.09.034. Epub 2013 Sep 28.

PMID:
24080302
16.

Chlamydia muridarum-specific CD4 T-cell clones recognize infected reproductive tract epithelial cells in an interferon-dependent fashion.

Jayarapu K, Kerr MS, Katschke A, Johnson RM.

Infect Immun. 2009 Oct;77(10):4469-79. doi: 10.1128/IAI.00491-09. Epub 2009 Aug 10.

17.

Induction and regulation of IFNs during viral infections.

Malmgaard L.

J Interferon Cytokine Res. 2004 Aug;24(8):439-54. Review.

PMID:
15320958
20.

FimH adhesin of type 1 fimbriae is a potent inducer of innate antimicrobial responses which requires TLR4 and type 1 interferon signalling.

Ashkar AA, Mossman KL, Coombes BK, Gyles CL, Mackenzie R.

PLoS Pathog. 2008 Dec;4(12):e1000233. doi: 10.1371/journal.ppat.1000233. Epub 2008 Dec 5.

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