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Similar articles for PubMed (Select 23338126)

1.

Hydrogen sulfide attenuates doxorubicin-induced cardiotoxicity by inhibition of the p38 MAPK pathway in H9c2 cells.

Guo R, Lin J, Xu W, Shen N, Mo L, Zhang C, Feng J.

Int J Mol Med. 2013 Mar;31(3):644-50. doi: 10.3892/ijmm.2013.1246. Epub 2013 Jan 15.

PMID:
23338126
2.

Exogenous hydrogen sulfide protects H9c2 cardiac cells against high glucose-induced injury by inhibiting the activities of the p38 MAPK and ERK1/2 pathways.

Xu W, Wu W, Chen J, Guo R, Lin J, Liao X, Feng J.

Int J Mol Med. 2013 Oct;32(4):917-25. doi: 10.3892/ijmm.2013.1462. Epub 2013 Aug 1.

PMID:
23912965
3.

Exogenous hydrogen sulfide protects against doxorubicin-induced inflammation and cytotoxicity by inhibiting p38MAPK/NFκB pathway in H9c2 cardiac cells.

Guo R, Wu K, Chen J, Mo L, Hua X, Zheng D, Chen P, Chen G, Xu W, Feng J.

Cell Physiol Biochem. 2013;32(6):1668-80. doi: 10.1159/000356602. Epub 2013 Dec 13.

PMID:
24356372
4.

Naringin inhibits ROS-activated MAPK pathway in high glucose-induced injuries in H9c2 cardiac cells.

Chen J, Guo R, Yan H, Tian L, You Q, Li S, Huang R, Wu K.

Basic Clin Pharmacol Toxicol. 2014 Apr;114(4):293-304. doi: 10.1111/bcpt.12153. Epub 2013 Dec 11.

PMID:
24118820
5.

Hydrogen sulfide protects H9c2 cells against doxorubicin-induced cardiotoxicity through inhibition of endoplasmic reticulum stress.

Wang XY, Yang CT, Zheng DD, Mo LQ, Lan AP, Yang ZL, Hu F, Chen PX, Liao XX, Feng JQ.

Mol Cell Biochem. 2012 Apr;363(1-2):419-26. doi: 10.1007/s11010-011-1194-6. Epub 2011 Dec 28.

PMID:
22203419
6.

Hydrogen sulfide protects against chemical hypoxia-induced injury by inhibiting ROS-activated ERK1/2 and p38MAPK signaling pathways in PC12 cells.

Lan A, Liao X, Mo L, Yang C, Yang Z, Wang X, Hu F, Chen P, Feng J, Zheng D, Xiao L.

PLoS One. 2011;6(10):e25921. doi: 10.1371/journal.pone.0025921. Epub 2011 Oct 5.

7.

Inhibition of the leptin-induced activation of the p38 MAPK pathway contributes to the protective effects of naringin against high glucose-induced injury in H9c2 cardiac cells.

Chen J, Mo H, Guo R, You Q, Huang R, Wu K.

Int J Mol Med. 2014 Mar;33(3):605-12. doi: 10.3892/ijmm.2014.1614. Epub 2014 Jan 7.

PMID:
24399619
8.

Novel insights into the role of HSP90 in cytoprotection of H2S against chemical hypoxia-induced injury in H9c2 cardiac myocytes.

Yang Z, Yang C, Xiao L, Liao X, Lan A, Wang X, Guo R, Chen P, Hu C, Feng J.

Int J Mol Med. 2011 Sep;28(3):397-403. doi: 10.3892/ijmm.2011.682. Epub 2011 Apr 20.

PMID:
21519787
9.

Activation of the p38 MAPK/NF-κB pathway contributes to doxorubicin-induced inflammation and cytotoxicity in H9c2 cardiac cells.

Guo RM, Xu WM, Lin JC, Mo LQ, Hua XX, Chen PX, Wu K, Zheng DD, Feng JQ.

Mol Med Rep. 2013 Aug;8(2):603-8. doi: 10.3892/mmr.2013.1554. Epub 2013 Jun 26.

PMID:
23807148
10.

Inhibition of ROS-activated ERK1/2 pathway contributes to the protection of H2S against chemical hypoxia-induced injury in H9c2 cells.

Dong XB, Yang CT, Zheng DD, Mo LQ, Wang XY, Lan AP, Hu F, Chen PX, Feng JQ, Zhang MF, Liao XX.

Mol Cell Biochem. 2012 Mar;362(1-2):149-57. doi: 10.1007/s11010-011-1137-2. Epub 2011 Dec 2.

PMID:
22134701
11.

Doxorubicin induces apoptosis in H9c2 cardiomyocytes: role of overexpressed eukaryotic translation initiation factor 5A.

Tan X, Wang DB, Lu X, Wei H, Zhu R, Zhu SS, Jiang H, Yang ZJ.

Biol Pharm Bull. 2010;33(10):1666-72.

12.

Exogenous H2S protects H9c2 cardiac cells against high glucose-induced injury and inflammation by inhibiting the activation of the NF-κB and IL-1β pathways.

Xu W, Chen J, Lin J, Liu D, Mo L, Pan W, Feng J, Wu W, Zheng D.

Int J Mol Med. 2015 Jan;35(1):177-86. doi: 10.3892/ijmm.2014.2007. Epub 2014 Nov 19.

PMID:
25412187
13.

Ghrelin inhibits doxorubicin cardiotoxicity by inhibiting excessive autophagy through AMPK and p38-MAPK.

Wang X, Wang XL, Chen HL, Wu D, Chen JX, Wang XX, Li RL, He JH, Mo L, Cen X, Wei YQ, Jiang W.

Biochem Pharmacol. 2014 Apr 1;88(3):334-50. doi: 10.1016/j.bcp.2014.01.040. Epub 2014 Feb 9.

PMID:
24522112
14.

Exogenous hydrogen sulfide alleviates high glucose-induced cardiotoxicity via inhibition of leptin signaling in H9c2 cells.

Zhuang XD, Hu X, Long M, Dong XB, Liu DH, Liao XX.

Mol Cell Biochem. 2014 Jun;391(1-2):147-55. doi: 10.1007/s11010-014-1997-3. Epub 2014 Apr 1.

PMID:
24687304
15.

Bupivacaine induces apoptosis via mitochondria and p38 MAPK dependent pathways.

Lu J, Xu SY, Zhang QG, Xu R, Lei HY.

Eur J Pharmacol. 2011 Apr 25;657(1-3):51-8. doi: 10.1016/j.ejphar.2011.01.055. Epub 2011 Feb 12.

PMID:
21315711
16.

Apoptosis induced by a new flavonoid in human hepatoma HepG2 cells involves reactive oxygen species-mediated mitochondrial dysfunction and MAPK activation.

Liu H, Xiao Y, Xiong C, Wei A, Ruan J.

Eur J Pharmacol. 2011 Mar 11;654(3):209-16. doi: 10.1016/j.ejphar.2010.12.036. Epub 2011 Jan 15.

PMID:
21241688
17.
18.

N-acetylcysteine amide decreases oxidative stress but not cell death induced by doxorubicin in H9c2 cardiomyocytes.

Shi R, Huang CC, Aronstam RS, Ercal N, Martin A, Huang YW.

BMC Pharmacol. 2009 Apr 15;9:7. doi: 10.1186/1471-2210-9-7.

19.

Modulation of ROS/MAPK signaling pathways by okadaic acid leads to cell death via, mitochondrial mediated caspase-dependent mechanism.

Ravindran J, Gupta N, Agrawal M, Bala Bhaskar AS, Lakshmana Rao PV.

Apoptosis. 2011 Feb;16(2):145-61. doi: 10.1007/s10495-010-0554-0.

PMID:
21082355
20.

Hydrogen sulphide protects H9c2 cells against chemical hypoxia-induced injury.

Chen SL, Yang CT, Yang ZL, Guo RX, Meng JL, Cui Y, Lan AP, Chen PX, Feng JQ.

Clin Exp Pharmacol Physiol. 2010 Mar;37(3):316-21. doi: 10.1111/j.1440-1681.2009.05289.x. Epub 2009 Sep 21.

PMID:
19769612
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