Items per page
Sort by

Send to:

Choose Destination

Results: 1 to 20 of 159

Similar articles for PubMed (Select 23283344)


Phenotyping the function of TRPV1-expressing sensory neurons by targeted axonal silencing.

Brenneis C, Kistner K, Puopolo M, Segal D, Roberson D, Sisignano M, Labocha S, Ferreirós N, Strominger A, Cobos EJ, Ghasemlou N, Geisslinger G, Reeh PW, Bean BP, Woolf CJ.

J Neurosci. 2013 Jan 2;33(1):315-26. doi: 10.1523/JNEUROSCI.2804-12.2013.


Inhibition of nociceptors by TRPV1-mediated entry of impermeant sodium channel blockers.

Binshtok AM, Bean BP, Woolf CJ.

Nature. 2007 Oct 4;449(7162):607-10.


Emulsified isoflurane enhances thermal transient receptor potential vanilloid-1 channel activation-mediated sensory/nociceptive blockade by QX-314.

Zhou C, Liang P, Liu J, Zhang W, Liao D, Chen Y, Chen X, Li T.

Anesthesiology. 2014 Aug;121(2):280-9. doi: 10.1097/ALN.0000000000000236.


Acid solution is a suitable medium for introducing QX-314 into nociceptors through TRPV1 channels to produce sensory-specific analgesic effects.

Liu H, Zhang HX, Hou HY, Lu XF, Wei JQ, Wang CG, Zhang LC, Zeng YM, Wu YP, Cao JL.

PLoS One. 2011;6(12):e29395. doi: 10.1371/journal.pone.0029395. Epub 2011 Dec 28.


Expression of TRPV1 channels after nerve injury provides an essential delivery tool for neuropathic pain attenuation.

Zakir HM, Mostafeezur RM, Suzuki A, Hitomi S, Suzuki I, Maeda T, Seo K, Yamada Y, Yamamura K, Lev S, Binshtok AM, Iwata K, Kitagawa J.

PLoS One. 2012;7(9):e44023. doi: 10.1371/journal.pone.0044023. Epub 2012 Sep 4.


Bupivacaine-induced cellular entry of QX-314 and its contribution to differential nerve block.

Brenneis C, Kistner K, Puopolo M, Jo S, Roberson D, Sisignano M, Segal D, Cobos EJ, Wainger BJ, Labocha S, Ferreirós N, von Hehn C, Tran J, Geisslinger G, Reeh PW, Bean BP, Woolf CJ.

Br J Pharmacol. 2014 Jan;171(2):438-51. doi: 10.1111/bph.12466.


External QX-314 inhibits evoked cranial primary afferent synaptic transmission independent of TRPV1.

Hofmann ME, Largent-Milnes TM, Fawley JA, Andresen MC.

J Neurophysiol. 2014 Dec 1;112(11):2697-706. doi: 10.1152/jn.00316.2014. Epub 2014 Sep 3.


Permeation and block of TRPV1 channels by the cationic lidocaine derivative QX-314.

Puopolo M, Binshtok AM, Yao GL, Oh SB, Woolf CJ, Bean BP.

J Neurophysiol. 2013 Apr;109(7):1704-12. doi: 10.1152/jn.00012.2013. Epub 2013 Jan 9.


QX-314 inhibits ectopic nerve activity associated with neuropathic pain.

Omana-Zapata I, Khabbaz MA, Hunter JC, Bley KR.

Brain Res. 1997 Oct 17;771(2):228-37.


Selectively targeting pain in the trigeminal system.

Kim HY, Kim K, Li HY, Chung G, Park CK, Kim JS, Jung SJ, Lee MK, Ahn DK, Hwang SJ, Kang Y, Binshtok AM, Bean BP, Woolf CJ, Oh SB.

Pain. 2010 Jul;150(1):29-40. doi: 10.1016/j.pain.2010.02.016. Epub 2010 Mar 16.


Involvement of capsaicin-sensitive afferents and the Transient Receptor Potential Vanilloid 1 Receptor in xylene-induced nocifensive behaviour and inflammation in the mouse.

Sándor K, Helyes Z, Elekes K, Szolcsányi J.

Neurosci Lett. 2009 Feb 27;451(3):204-7. doi: 10.1016/j.neulet.2009.01.016. Epub 2009 Jan 13.


Targeting of sodium channel blockers into nociceptors to produce long-duration analgesia: a systematic study and review.

Roberson DP, Binshtok AM, Blasl F, Bean BP, Woolf CJ.

Br J Pharmacol. 2011 Sep;164(1):48-58. doi: 10.1111/j.1476-5381.2011.01391.x. Review.


Increased function of the TRPV1 channel in small sensory neurons after local inflammation or in vitro exposure to the pro-inflammatory cytokine GRO/KC.

Dong F, Du YR, Xie W, Strong JA, He XJ, Zhang JM.

Neurosci Bull. 2012 Apr;28(2):155-64. doi: 10.1007/s12264-012-1208-8.


Contributions of central and peripheral TRPV1 receptors to mechanically evoked and spontaneous firing of spinal neurons in inflamed rats.

McGaraughty S, Chu KL, Brown BS, Zhu CZ, Zhong C, Joshi SK, Honore P, Faltynek CR, Jarvis MF.

J Neurophysiol. 2008 Dec;100(6):3158-66. doi: 10.1152/jn.90768.2008. Epub 2008 Oct 1.


Nociceptor-selective peripheral nerve block induces delayed mechanical hypersensitivity and neurotoxicity in rats.

Peters CM, Ririe D, Houle TT, Aschenbrenner CA, Eisenach JC.

Anesthesiology. 2014 Apr;120(4):976-86. doi: 10.1097/ALN.0000000000000088.


Coapplication of lidocaine and the permanently charged sodium channel blocker QX-314 produces a long-lasting nociceptive blockade in rodents.

Binshtok AM, Gerner P, Oh SB, Puopolo M, Suzuki S, Roberson DP, Herbert T, Wang CF, Kim D, Chung G, Mitani AA, Wang GK, Bean BP, Woolf CJ.

Anesthesiology. 2009 Jul;111(1):127-37. doi: 10.1097/ALN.0b013e3181a915e7.


Persistent Nociception Triggered by Nerve Growth Factor (NGF) Is Mediated by TRPV1 and Oxidative Mechanisms.

Eskander MA, Ruparel S, Green DP, Chen PB, Por ED, Jeske NA, Gao X, Flores ER, Hargreaves KM.

J Neurosci. 2015 Jun 3;35(22):8593-603. doi: 10.1523/JNEUROSCI.3993-14.2015.


Peripheral nerve injury and TRPV1-expressing primary afferent C-fibers cause opening of the blood-brain barrier.

Beggs S, Liu XJ, Kwan C, Salter MW.

Mol Pain. 2010 Nov 2;6:74. doi: 10.1186/1744-8069-6-74.


Differential effects of peripheral versus central coadministration of QX-314 and capsaicin on neuropathic pain in rats.

Shen J, Fox LE, Cheng J.

Anesthesiology. 2012 Aug;117(2):365-80. doi: 10.1097/ALN.0b013e318260de41.


Investigation of the role of TRPV1 receptors in acute and chronic nociceptive processes using gene-deficient mice.

Bölcskei K, Helyes Z, Szabó A, Sándor K, Elekes K, Németh J, Almási R, Pintér E, Petho G, Szolcsányi J.

Pain. 2005 Oct;117(3):368-76.

Items per page
Sort by

Send to:

Choose Destination

Supplemental Content

Write to the Help Desk