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Items: 1 to 20 of 433

1.

Competing roles of DNA end resection and non-homologous end joining functions in the repair of replication-born double-strand breaks by sister-chromatid recombination.

Muñoz-Galván S, López-Saavedra A, Jackson SP, Huertas P, Cortés-Ledesma F, Aguilera A.

Nucleic Acids Res. 2013 Feb 1;41(3):1669-83. doi: 10.1093/nar/gks1274. Epub 2012 Dec 18.

2.

The MRX complex plays multiple functions in resection of Yku- and Rif2-protected DNA ends.

Bonetti D, Clerici M, Manfrini N, Lucchini G, Longhese MP.

PLoS One. 2010 Nov 30;5(11):e14142. doi: 10.1371/journal.pone.0014142.

3.

Functional interplay between the 53BP1-ortholog Rad9 and the Mre11 complex regulates resection, end-tethering and repair of a double-strand break.

Ferrari M, Dibitetto D, De Gregorio G, Eapen VV, Rawal CC, Lazzaro F, Tsabar M, Marini F, Haber JE, Pellicioli A.

PLoS Genet. 2015 Jan 8;11(1):e1004928. doi: 10.1371/journal.pgen.1004928. eCollection 2015 Jan.

4.

Coincident resection at both ends of random, γ-induced double-strand breaks requires MRX (MRN), Sae2 (Ctp1), and Mre11-nuclease.

Westmoreland JW, Resnick MA.

PLoS Genet. 2013 Mar;9(3):e1003420. doi: 10.1371/journal.pgen.1003420. Epub 2013 Mar 28.

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Release of Ku and MRN from DNA ends by Mre11 nuclease activity and Ctp1 is required for homologous recombination repair of double-strand breaks.

Langerak P, Mejia-Ramirez E, Limbo O, Russell P.

PLoS Genet. 2011 Sep;7(9):e1002271. doi: 10.1371/journal.pgen.1002271. Epub 2011 Sep 8.

7.

Ku prevents Exo1 and Sgs1-dependent resection of DNA ends in the absence of a functional MRX complex or Sae2.

Mimitou EP, Symington LS.

EMBO J. 2010 Oct 6;29(19):3358-69. doi: 10.1038/emboj.2010.193. Epub 2010 Aug 20.

8.

Functional interplay of the Mre11 nuclease and Ku in the response to replication-associated DNA damage.

Foster SS, Balestrini A, Petrini JH.

Mol Cell Biol. 2011 Nov;31(21):4379-89. doi: 10.1128/MCB.05854-11. Epub 2011 Aug 29.

9.

Sae2, Exo1 and Sgs1 collaborate in DNA double-strand break processing.

Mimitou EP, Symington LS.

Nature. 2008 Oct 9;455(7214):770-4. doi: 10.1038/nature07312. Epub 2008 Sep 21.

10.

Sgs1 helicase and two nucleases Dna2 and Exo1 resect DNA double-strand break ends.

Zhu Z, Chung WH, Shim EY, Lee SE, Ira G.

Cell. 2008 Sep 19;134(6):981-94. doi: 10.1016/j.cell.2008.08.037.

11.

Saccharomyces cerevisiae Mre11/Rad50/Xrs2 and Ku proteins regulate association of Exo1 and Dna2 with DNA breaks.

Shim EY, Chung WH, Nicolette ML, Zhang Y, Davis M, Zhu Z, Paull TT, Ira G, Lee SE.

EMBO J. 2010 Oct 6;29(19):3370-80. doi: 10.1038/emboj.2010.219. Epub 2010 Sep 10.

12.

Microhomology-mediated End Joining and Homologous Recombination share the initial end resection step to repair DNA double-strand breaks in mammalian cells.

Truong LN, Li Y, Shi LZ, Hwang PY, He J, Wang H, Razavian N, Berns MW, Wu X.

Proc Natl Acad Sci U S A. 2013 May 7;110(19):7720-5. doi: 10.1073/pnas.1213431110. Epub 2013 Apr 22.

13.

CDK targets Sae2 to control DNA-end resection and homologous recombination.

Huertas P, Cortés-Ledesma F, Sartori AA, Aguilera A, Jackson SP.

Nature. 2008 Oct 2;455(7213):689-92. doi: 10.1038/nature07215. Epub 2008 Aug 20.

14.

Mre11-Rad50-Xrs2 and Sae2 promote 5' strand resection of DNA double-strand breaks.

Nicolette ML, Lee K, Guo Z, Rani M, Chow JM, Lee SE, Paull TT.

Nat Struct Mol Biol. 2010 Dec;17(12):1478-85. doi: 10.1038/nsmb.1957. Epub 2010 Nov 21.

15.

The yeast Fun30 and human SMARCAD1 chromatin remodellers promote DNA end resection.

Costelloe T, Louge R, Tomimatsu N, Mukherjee B, Martini E, Khadaroo B, Dubois K, Wiegant WW, Thierry A, Burma S, van Attikum H, Llorente B.

Nature. 2012 Sep 27;489(7417):581-4. doi: 10.1038/nature11353. Epub 2012 Sep 9.

16.

Resection activity of the Sgs1 helicase alters the affinity of DNA ends for homologous recombination proteins in Saccharomyces cerevisiae.

Bernstein KA, Mimitou EP, Mihalevic MJ, Chen H, Sunjaveric I, Symington LS, Rothstein R.

Genetics. 2013 Dec;195(4):1241-51. doi: 10.1534/genetics.113.157370. Epub 2013 Oct 4.

17.

DNA end resection by Dna2-Sgs1-RPA and its stimulation by Top3-Rmi1 and Mre11-Rad50-Xrs2.

Cejka P, Cannavo E, Polaczek P, Masuda-Sasa T, Pokharel S, Campbell JL, Kowalczykowski SC.

Nature. 2010 Sep 2;467(7311):112-6. doi: 10.1038/nature09355.

18.

Escape of Sgs1 from Rad9 inhibition reduces the requirement for Sae2 and functional MRX in DNA end resection.

Bonetti D, Villa M, Gobbini E, Cassani C, Tedeschi G, Longhese MP.

EMBO Rep. 2015 Mar;16(3):351-61. doi: 10.15252/embr.201439764. Epub 2015 Jan 30.

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