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Items: 1 to 20 of 189

1.

Spontaneous skin erosions and reduced skin and corneal wound healing characterize CLIC4(NULL) mice.

Padmakumar VC, Speer K, Pal-Ghosh S, Masiuk KE, Ryscavage A, Dengler SL, Hwang S, Edwards JC, Coppola V, Tessarollo L, Stepp MA, Yuspa SH.

Am J Pathol. 2012 Jul;181(1):74-84. doi: 10.1016/j.ajpath.2012.03.025. Epub 2012 May 18.

2.

Myostatin-null mice exhibit delayed skin wound healing through the blockade of transforming growth factor-β signaling by decorin.

Zhang C, Tan CK, McFarlane C, Sharma M, Tan NS, Kambadur R.

Am J Physiol Cell Physiol. 2012 Apr 15;302(8):C1213-25. doi: 10.1152/ajpcell.00179.2011. Epub 2012 Jan 25. Erratum in: Am J Physiol Cell Physiol. 2014 Dec 15;307(12):C1153.

3.

Alteration of skin wound healing in keratinocyte-specific mediator complex subunit 1 null mice.

Noguchi F, Nakajima T, Inui S, Reddy JK, Itami S.

PLoS One. 2014 Aug 14;9(8):e102271. doi: 10.1371/journal.pone.0102271. eCollection 2014.

4.

Transforming growth factor-beta and its effect on reepithelialization of partial-thickness ear wounds in transgenic mice.

Tredget EB, Demare J, Chandran G, Tredget EE, Yang L, Ghahary A.

Wound Repair Regen. 2005 Jan-Feb;13(1):61-7.

PMID:
15659037
5.

Mice that lack matrix metalloproteinase-9 display delayed wound healing associated with delayed reepithelization and disordered collagen fibrillogenesis.

Kyriakides TR, Wulsin D, Skokos EA, Fleckman P, Pirrone A, Shipley JM, Senior RM, Bornstein P.

Matrix Biol. 2009 Mar;28(2):65-73. doi: 10.1016/j.matbio.2009.01.001. Epub 2009 Jan 20.

6.

Cutaneous wound reepithelialization is compromised in mice lacking functional Slug (Snai2).

Hudson LG, Newkirk KM, Chandler HL, Choi C, Fossey SL, Parent AE, Kusewitt DF.

J Dermatol Sci. 2009 Oct;56(1):19-26. doi: 10.1016/j.jdermsci.2009.06.009. Epub 2009 Jul 29.

7.

Fibromodulin-deficiency alters temporospatial expression patterns of transforming growth factor-β ligands and receptors during adult mouse skin wound healing.

Zheng Z, Lee KS, Zhang X, Nguyen C, Hsu C, Wang JZ, Rackohn TM, Enjamuri DR, Murphy M, Ting K, Soo C.

PLoS One. 2014 Mar 6;9(6):e90817. doi: 10.1371/journal.pone.0090817. eCollection 2014.

8.

BALB/c and C57BL6 mouse strains vary in their ability to heal corneal epithelial debridement wounds.

Pal-Ghosh S, Tadvalkar G, Jurjus RA, Zieske JD, Stepp MA.

Exp Eye Res. 2008 Nov;87(5):478-86. doi: 10.1016/j.exer.2008.08.013. Epub 2008 Sep 6.

9.

TGF-beta signalling is regulated by Schnurri-2-dependent nuclear translocation of CLIC4 and consequent stabilization of phospho-Smad2 and 3.

Shukla A, Malik M, Cataisson C, Ho Y, Friesen T, Suh KS, Yuspa SH.

Nat Cell Biol. 2009 Jun;11(6):777-84. doi: 10.1038/ncb1885. Epub 2009 May 17.

10.

Mice lacking Smad3 show accelerated wound healing and an impaired local inflammatory response.

Ashcroft GS, Yang X, Glick AB, Weinstein M, Letterio JL, Mizel DE, Anzano M, Greenwell-Wild T, Wahl SM, Deng C, Roberts AB.

Nat Cell Biol. 1999 Sep;1(5):260-6.

PMID:
10559937
11.

Activation of the δ-opioid receptor promotes cutaneous wound healing by affecting keratinocyte intercellular adhesion and migration.

Bigliardi PL, Neumann C, Teo YL, Pant A, Bigliardi-Qi M.

Br J Pharmacol. 2015 Jan;172(2):501-14. doi: 10.1111/bph.12687. Epub 2014 Jul 1.

12.

CLIC4 is a tumor suppressor for cutaneous squamous cell cancer.

Suh KS, Malik M, Shukla A, Ryscavage A, Wright L, Jividen K, Crutchley JM, Dumont RA, Fernandez-Salas E, Webster JD, Simpson RM, Yuspa SH.

Carcinogenesis. 2012 May;33(5):986-95. doi: 10.1093/carcin/bgs115. Epub 2012 Mar 1.

13.

Functional significance of Smad2 in regulating basal keratinocyte migration during wound healing.

Hosokawa R, Urata MM, Ito Y, Bringas P Jr, Chai Y.

J Invest Dermatol. 2005 Dec;125(6):1302-9.

14.

Reduced migration, altered matrix and enhanced TGFbeta1 signaling are signatures of mouse keratinocytes lacking Sdc1.

Stepp MA, Liu Y, Pal-Ghosh S, Jurjus RA, Tadvalkar G, Sekaran A, Losicco K, Jiang L, Larsen M, Li L, Yuspa SH.

J Cell Sci. 2007 Aug 15;120(Pt 16):2851-63. Epub 2007 Jul 31.

15.

Interference with transforming growth factor-beta/ Smad3 signaling results in accelerated healing of wounds in previously irradiated skin.

Flanders KC, Major CD, Arabshahi A, Aburime EE, Okada MH, Fujii M, Blalock TD, Schultz GS, Sowers A, Anzano MA, Mitchell JB, Russo A, Roberts AB.

Am J Pathol. 2003 Dec;163(6):2247-57.

16.

A novel small compound accelerates dermal wound healing by modifying infiltration, proliferation and migration of distinct cellular components in mice.

Yamaoka H, Sumiyoshi H, Higashi K, Nakao S, Minakawa K, Sumida K, Saito K, Ikoma N, Mabuchi T, Ozawa A, Inagaki Y.

J Dermatol Sci. 2014 Jun;74(3):204-13. doi: 10.1016/j.jdermsci.2014.03.002. Epub 2014 Mar 18.

PMID:
24702853
17.

Topical androgen antagonism promotes cutaneous wound healing without systemic androgen deprivation by blocking β-catenin nuclear translocation and cross-talk with TGF-β signaling in keratinocytes.

Toraldo G, Bhasin S, Bakhit M, Guo W, Serra C, Safer JD, Bhawan J, Jasuja R.

Wound Repair Regen. 2012 Jan-Feb;20(1):61-73. doi: 10.1111/j.1524-475X.2011.00757.x.

PMID:
22276587
18.

Beta-catenin regulates wound size and mediates the effect of TGF-beta in cutaneous healing.

Cheon SS, Wei Q, Gurung A, Youn A, Bright T, Poon R, Whetstone H, Guha A, Alman BA.

FASEB J. 2006 Apr;20(6):692-701.

19.

Stress-mediated increases in systemic and local epinephrine impair skin wound healing: potential new indication for beta blockers.

Sivamani RK, Pullar CE, Manabat-Hidalgo CG, Rocke DM, Carlsen RC, Greenhalgh DG, Isseroff RR.

PLoS Med. 2009 Jan 13;6(1):e12. doi: 10.1371/journal.pmed.1000012.

20.

EGFR enhances early healing after cutaneous incisional wounding.

Repertinger SK, Campagnaro E, Fuhrman J, El-Abaseri T, Yuspa SH, Hansen LA.

J Invest Dermatol. 2004 Nov;123(5):982-9.

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