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Items: 1 to 20 of 104

1.

Age-dependent carcinogenic susceptibility in rat liver is related to potential of gap junctional intercellular communication.

Naiki-Ito A, Kato H, Asamoto M, Naiki T, Shirai T.

Toxicol Pathol. 2012 Jul;40(5):715-21. doi: 10.1177/0192623312441402. Epub 2012 May 8.

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The relationship between decrease in Cx32 and induction of P450 isozymes in the early phase of clofibrate hepatocarcinogenesis in the rat.

Shoda T, Mitsumori K, Onodera H, Toyoda K, Uneyama C, Imazawa T, Hirose M.

Arch Toxicol. 1999 Sep;73(7):373-80.

PMID:
10550479
6.

Low dose DDT inhibition of hepatocarcinogenesis initiated by diethylnitrosamine in male rats: possible mechanisms.

Kushida M, Sukata T, Uwagawa S, Ozaki K, Kinoshita A, Wanibuchi H, Morimura K, Okuno Y, Fukushima S.

Toxicol Appl Pharmacol. 2005 Nov 1;208(3):285-94.

PMID:
15885732
7.

Both early and late stages of hepatocarcinogenesis are enhanced in Cx32 dominant negative mutant transgenic rats with disrupted gap junctional intercellular communication.

Hokaiwado N, Asamoto M, Futakuchi M, Ogawa K, Takahashi S, Shirai T.

J Membr Biol. 2007 Aug;218(1-3):101-6. Epub 2007 Nov 3.

PMID:
17978847
8.

Multiple mechanisms are responsible for altered expression of gap junction genes during oncogenesis in rat liver.

Neveu MJ, Hully JR, Babcock KL, Hertzberg EL, Nicholson BJ, Paul DL, Pitot HC.

J Cell Sci. 1994 Jan;107 ( Pt 1):83-95.

12.

Morphology and morphometric investigation of hepatocellular preneoplastic lesions and neoplasms in connexin32-deficient mice.

Evert M, Ott T, Temme A, Willecke K, Dombrowski F.

Carcinogenesis. 2002 May;23(5):697-703.

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Colocalized alterations in connexin32 and cytochrome P450IIB1/2 by phenobarbital and related liver tumor promoters.

Neveu MJ, Babcock KL, Hertzberg EL, Paul DL, Nicholson BJ, Pitot HC.

Cancer Res. 1994 Jun 15;54(12):3145-52.

17.

Liver tumor promoting effects of fenbendazole in rats.

Shoda T, Onodera H, Takeda M, Uneyama C, Imazawa T, Takegawa K, Yasuhara K, Watanabe T, Hirose M, Mitsumori K.

Toxicol Pathol. 1999 Sep-Oct;27(5):553-62.

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Ablation of gap junctional communication in hepatocytes of transgenic mice does not lead to disrupted cellular homeostasis or increased spontaneous tumourigenesis.

Ott T, Jokwitz M, Lenhard D, Romualdi A, Dombrowski F, Ittrich C, Schwarz M, Willecke K.

Eur J Cell Biol. 2006 Aug;85(8):717-28. Epub 2006 Jun 5.

PMID:
16740338
20.

Lack of tumor-promoting effects of flavonoids: studies on rat liver preneoplastic foci and on in vivo and in vitro gap junctional intercellular communication.

Chaumontet C, Suschetet M, Honikman-Leban E, Krutovskikh VA, Berges R, Le Bon AM, Heberden C, Shahin MM, Yamasaki H, Martel P.

Nutr Cancer. 1996;26(3):251-63.

PMID:
8910908
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