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Items: 1 to 20 of 116

1.

Arsenic-transformed malignant prostate epithelia can convert noncontiguous normal stem cells into an oncogenic phenotype.

Xu Y, Tokar EJ, Sun Y, Waalkes MP.

Environ Health Perspect. 2012 Jun;120(6):865-71. doi: 10.1289/ehp.1204987. Epub 2012 Apr 4.

2.

Recruitment of normal stem cells to an oncogenic phenotype by noncontiguous carcinogen-transformed epithelia depends on the transforming carcinogen.

Xu Y, Tokar EJ, Person RJ, Orihuela RG, Ngalame NN, Waalkes MP.

Environ Health Perspect. 2013 Aug;121(8):944-50. doi: 10.1289/ehp.1306714. Epub 2013 May 17.

3.

Arsenic exposure transforms human epithelial stem/progenitor cells into a cancer stem-like phenotype.

Tokar EJ, Diwan BA, Waalkes MP.

Environ Health Perspect. 2010 Jan;118(1):108-15. doi: 10.1289/ehp.0901059.

4.

Arsenic-specific stem cell selection during malignant transformation.

Tokar EJ, Qu W, Liu J, Liu W, Webber MM, Phang JM, Waalkes MP.

J Natl Cancer Inst. 2010 May 5;102(9):638-49. doi: 10.1093/jnci/djq093. Epub 2010 Mar 25.

5.

Silencing KRAS overexpression in arsenic-transformed prostate epithelial and stem cells partially mitigates malignant phenotype.

Ngalame NN, Tokar EJ, Person RJ, Waalkes MP.

Toxicol Sci. 2014 Dec;142(2):489-96. doi: 10.1093/toxsci/kfu201. Epub 2014 Sep 30.

6.

Overabundance of putative cancer stem cells in human skin keratinocyte cells malignantly transformed by arsenic.

Sun Y, Tokar EJ, Waalkes MP.

Toxicol Sci. 2012 Jan;125(1):20-9. doi: 10.1093/toxsci/kfr282. Epub 2011 Oct 19.

7.

Aberrant microRNA expression likely controls RAS oncogene activation during malignant transformation of human prostate epithelial and stem cells by arsenic.

Ngalame NN, Tokar EJ, Person RJ, Xu Y, Waalkes MP.

Toxicol Sci. 2014 Apr;138(2):268-77. doi: 10.1093/toxsci/kfu002. Epub 2014 Jan 15.

8.

Chronic exposure of renal stem cells to inorganic arsenic induces a cancer phenotype.

Tokar EJ, Person RJ, Sun Y, Perantoni AO, Waalkes MP.

Chem Res Toxicol. 2013 Jan 18;26(1):96-105. doi: 10.1021/tx3004054. Epub 2012 Nov 19.

9.

Bad neighbors: arsenic-induced tumor cells convert normal stem cells into a cancerous phenotype.

Barrett JR.

Environ Health Perspect. 2012 Jun;120(6):A244. doi: 10.1289/ehp.120-a244a. No abstract available.

10.

Arsenic-induced cancer cell phenotype in human breast epithelia is estrogen receptor-independent but involves aromatase activation.

Xu Y, Tokar EJ, Waalkes MP.

Arch Toxicol. 2014 Feb;88(2):263-74. doi: 10.1007/s00204-013-1131-4. Epub 2013 Sep 26.

11.

Chronic cadmium exposure in vitro causes acquisition of multiple tumor cell characteristics in human pancreatic epithelial cells.

Qu W, Tokar EJ, Kim AJ, Bell MW, Waalkes MP.

Environ Health Perspect. 2012 Sep;120(9):1265-71. doi: 10.1289/ehp.1205082. Epub 2012 May 24.

12.

Mechanisms of acquired androgen independence during arsenic-induced malignant transformation of human prostate epithelial cells.

Benbrahim-Tallaa L, Webber MM, Waalkes MP.

Environ Health Perspect. 2007 Feb;115(2):243-7. Epub 2006 Nov 20.

13.

Epithelial-mesenchymal transition and cancer stem cells, mediated by a long non-coding RNA, HOTAIR, are involved in cell malignant transformation induced by cigarette smoke extract.

Liu Y, Luo F, Xu Y, Wang B, Zhao Y, Xu W, Shi L, Lu X, Liu Q.

Toxicol Appl Pharmacol. 2015 Jan 1;282(1):9-19. doi: 10.1016/j.taap.2014.10.022. Epub 2014 Nov 8.

PMID:
25447409
14.

Acquisition of androgen independence by human prostate epithelial cells during arsenic-induced malignant transformation.

Benbrahim-Tallaa L, Webber MM, Waalkes MP.

Environ Health Perspect. 2005 Sep;113(9):1134-9.

15.

Aberrant cytokeratin expression during arsenic-induced acquired malignant phenotype in human HaCaT keratinocytes consistent with epidermal carcinogenesis.

Sun Y, Pi J, Wang X, Tokar EJ, Liu J, Waalkes MP.

Toxicology. 2009 Aug 3;262(2):162-70. doi: 10.1016/j.tox.2009.06.003. Epub 2009 Jun 12.

16.

Epithelial to mesenchymal transition in arsenic-transformed cells promotes angiogenesis through activating β-catenin-vascular endothelial growth factor pathway.

Wang Z, Humphries B, Xiao H, Jiang Y, Yang C.

Toxicol Appl Pharmacol. 2013 Aug 15;271(1):20-9. doi: 10.1016/j.taap.2013.04.018. Epub 2013 Apr 30.

17.

Mitigation of arsenic-induced acquired cancer phenotype in prostate cancer stem cells by miR-143 restoration.

Ngalame NN, Makia NL, Waalkes MP, Tokar EJ.

Toxicol Appl Pharmacol. 2015 Dec 22. pii: S0041-008X(15)30159-9. doi: 10.1016/j.taap.2015.12.013. [Epub ahead of print]

PMID:
26721309
18.

Chronic inorganic arsenic exposure in vitro induces a cancer cell phenotype in human peripheral lung epithelial cells.

Person RJ, Ngalame NN, Makia NL, Bell MW, Waalkes MP, Tokar EJ.

Toxicol Appl Pharmacol. 2015 Jul 1;286(1):36-43. doi: 10.1016/j.taap.2015.03.014. Epub 2015 Mar 21.

PMID:
25804888
19.

Reversal and prevention of arsenic-induced human bronchial epithelial cell malignant transformation by microRNA-200b.

Wang Z, Zhao Y, Smith E, Goodall GJ, Drew PA, Brabletz T, Yang C.

Toxicol Sci. 2011 May;121(1):110-22. doi: 10.1093/toxsci/kfr029. Epub 2011 Feb 2.

20.

Targeting of α(v)-integrins in stem/progenitor cells and supportive microenvironment impairs bone metastasis in human prostate cancer.

van der Horst G, van den Hoogen C, Buijs JT, Cheung H, Bloys H, Pelger RC, Lorenzon G, Heckmann B, Feyen J, Pujuguet P, Blanque R, Clément-Lacroix P, van der Pluijm G.

Neoplasia. 2011 Jun;13(6):516-25.

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