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Results: 1 to 20 of 85

Similar articles for PubMed (Select 22433842)

1.

Loss of mitochondrial protease OMA1 alters processing of the GTPase OPA1 and causes obesity and defective thermogenesis in mice.

Quirós PM, Ramsay AJ, Sala D, Fernández-Vizarra E, Rodríguez F, Peinado JR, Fernández-García MS, Vega JA, Enríquez JA, Zorzano A, López-Otín C.

EMBO J. 2012 May 2;31(9):2117-33. doi: 10.1038/emboj.2012.70. Epub 2012 Mar 20.

2.

YME1L degradation reduces mitochondrial proteolytic capacity during oxidative stress.

Rainbolt TK, Saunders JM, Wiseman RL.

EMBO Rep. 2015 Jan;16(1):97-106. doi: 10.15252/embr.201438976. Epub 2014 Nov 27.

PMID:
25433032
3.

A Mitofusin-2-dependent inactivating cleavage of Opa1 links changes in mitochondria cristae and ER contacts in the postprandial liver.

Sood A, Jeyaraju DV, Prudent J, Caron A, Lemieux P, McBride HM, Laplante M, Tóth K, Pellegrini L.

Proc Natl Acad Sci U S A. 2014 Nov 11;111(45):16017-22. doi: 10.1073/pnas.1408061111. Epub 2014 Oct 28.

4.

OPA1-dependent cristae modulation is essential for cellular adaptation to metabolic demand.

Patten DA, Wong J, Khacho M, Soubannier V, Mailloux RJ, Pilon-Larose K, MacLaurin JG, Park DS, McBride HM, Trinkle-Mulcahy L, Harper ME, Germain M, Slack RS.

EMBO J. 2014 Nov 18;33(22):2676-91. doi: 10.15252/embj.201488349. Epub 2014 Oct 8.

PMID:
25298396
5.

p53 is required for cisplatin-induced processing of the mitochondrial fusion protein L-Opa1 that is mediated by the mitochondrial metallopeptidase Oma1 in gynecologic cancers.

Kong B, Wang Q, Fung E, Xue K, Tsang BK.

J Biol Chem. 2014 Sep 26;289(39):27134-45. doi: 10.1074/jbc.M114.594812. Epub 2014 Aug 11.

PMID:
25112877
6.

Membrane depolarization activates the mitochondrial protease OMA1 by stimulating self-cleavage.

Zhang K, Li H, Song Z.

EMBO Rep. 2014 May;15(5):576-85. doi: 10.1002/embr.201338240. Epub 2014 Apr 9.

7.

Loss of the m-AAA protease subunit AFG₃L₂ causes mitochondrial transport defects and tau hyperphosphorylation.

Kondadi AK, Wang S, Montagner S, Kladt N, Korwitz A, Martinelli P, Herholz D, Baker MJ, Schauss AC, Langer T, Rugarli EI.

EMBO J. 2014 May 2;33(9):1011-26. doi: 10.1002/embj.201387009. Epub 2014 Mar 28.

8.

OMA1 mediates OPA1 proteolysis and mitochondrial fragmentation in experimental models of ischemic kidney injury.

Xiao X, Hu Y, Quirós PM, Wei Q, López-Otín C, Dong Z.

Am J Physiol Renal Physiol. 2014 Jun 1;306(11):F1318-26. doi: 10.1152/ajprenal.00036.2014. Epub 2014 Mar 26.

9.

Stress-triggered activation of the metalloprotease Oma1 involves its C-terminal region and is important for mitochondrial stress protection in yeast.

Bohovych I, Donaldson G, Christianson S, Zahayko N, Khalimonchuk O.

J Biol Chem. 2014 May 9;289(19):13259-72. doi: 10.1074/jbc.M113.542910. Epub 2014 Mar 19.

10.
11.

Transient contraction of mitochondria induces depolarization through the inner membrane dynamin OPA1 protein.

Lee H, Yoon Y.

J Biol Chem. 2014 Apr 25;289(17):11862-72. doi: 10.1074/jbc.M113.533299. Epub 2014 Mar 13.

12.

The i-AAA protease YME1L and OMA1 cleave OPA1 to balance mitochondrial fusion and fission.

Anand R, Wai T, Baker MJ, Kladt N, Schauss AC, Rugarli E, Langer T.

J Cell Biol. 2014 Mar 17;204(6):919-29. doi: 10.1083/jcb.201308006. Epub 2014 Mar 10.

13.

Stress-induced OMA1 activation and autocatalytic turnover regulate OPA1-dependent mitochondrial dynamics.

Baker MJ, Lampe PA, Stojanovski D, Korwitz A, Anand R, Tatsuta T, Langer T.

EMBO J. 2014 Mar 18;33(6):578-93. doi: 10.1002/embj.201386474. Epub 2014 Feb 18.

14.

Hormone-induced mitochondrial fission is utilized by brown adipocytes as an amplification pathway for energy expenditure.

Wikstrom JD, Mahdaviani K, Liesa M, Sereda SB, Si Y, Las G, Twig G, Petrovic N, Zingaretti C, Graham A, Cinti S, Corkey BE, Cannon B, Nedergaard J, Shirihai OS.

EMBO J. 2014 Mar 3;33(5):418-36. doi: 10.1002/embj.201385014. Epub 2014 Jan 15.

15.

SIRT3 deacetylates and activates OPA1 to regulate mitochondrial dynamics during stress.

Samant SA, Zhang HJ, Hong Z, Pillai VB, Sundaresan NR, Wolfgeher D, Archer SL, Chan DC, Gupta MP.

Mol Cell Biol. 2014 Mar;34(5):807-19. doi: 10.1128/MCB.01483-13. Epub 2013 Dec 16.

16.

Neuroendocrinology: Mitofusins and energy balance.

Koch L.

Nat Rev Endocrinol. 2013 Dec;9(12):691. doi: 10.1038/nrendo.2013.202. Epub 2013 Oct 15. No abstract available.

PMID:
24126482
17.

Mitochondrial dynamics controlled by mitofusins regulate Agrp neuronal activity and diet-induced obesity.

Dietrich MO, Liu ZW, Horvath TL.

Cell. 2013 Sep 26;155(1):188-99. doi: 10.1016/j.cell.2013.09.004.

18.

Mitofusins: mighty regulators of metabolism.

Ozcan U.

Cell. 2013 Sep 26;155(1):17-8. doi: 10.1016/j.cell.2013.09.013.

19.

Chronic sympathoexcitation through loss of Vav3, a Rac1 activator, results in divergent effects on metabolic syndrome and obesity depending on diet.

Menacho-Márquez M, Nogueiras R, Fabbiano S, Sauzeau V, Al-Massadi O, Diéguez C, Bustelo XR.

Cell Metab. 2013 Aug 6;18(2):199-211. doi: 10.1016/j.cmet.2013.07.001.

20.

OPA1 promotes pH flashes that spread between contiguous mitochondria without matrix protein exchange.

Santo-Domingo J, Giacomello M, Poburko D, Scorrano L, Demaurex N.

EMBO J. 2013 Jul 3;32(13):1927-40. doi: 10.1038/emboj.2013.124. Epub 2013 May 28.

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