Items per page
Sort by

Send to:

Choose Destination

Links from PubMed

Items: 1 to 20 of 104


Forkhead transcription factors establish origin timing and long-range clustering in S. cerevisiae.

Knott SR, Peace JM, Ostrow AZ, Gan Y, Rex AE, Viggiani CJ, Tavaré S, Aparicio OM.

Cell. 2012 Jan 20;148(1-2):99-111. doi: 10.1016/j.cell.2011.12.012.


Fkh1 and Fkh2 bind multiple chromosomal elements in the S. cerevisiae genome with distinct specificities and cell cycle dynamics.

Ostrow AZ, Nellimoottil T, Knott SR, Fox CA, Tavaré S, Aparicio OM.

PLoS One. 2014 Feb 4;9(2):e87647. doi: 10.1371/journal.pone.0087647. eCollection 2014.


Conversion of a replication origin to a silencer through a pathway shared by a Forkhead transcription factor and an S phase cyclin.

Casey L, Patterson EE, Müller U, Fox CA.

Mol Biol Cell. 2008 Feb;19(2):608-22. Epub 2007 Nov 28.


Origin association of Sld3, Sld7, and Cdc45 proteins is a key step for determination of origin-firing timing.

Tanaka S, Nakato R, Katou Y, Shirahige K, Araki H.

Curr Biol. 2011 Dec 20;21(24):2055-63. doi: 10.1016/j.cub.2011.11.038. Epub 2011 Dec 8.


Quantitative BrdU immunoprecipitation method demonstrates that Fkh1 and Fkh2 are rate-limiting activators of replication origins that reprogram replication timing in G1 phase.

Peace JM, Villwock SK, Zeytounian JL, Gan Y, Aparicio OM.

Genome Res. 2016 Mar;26(3):365-75. doi: 10.1101/gr.196857.115. Epub 2016 Jan 4.


Coupling phosphate homeostasis to cell cycle-specific transcription: mitotic activation of Saccharomyces cerevisiae PHO5 by Mcm1 and Forkhead proteins.

Pondugula S, Neef DW, Voth WP, Darst RP, Dhasarathy A, Reynolds MM, Takahata S, Stillman DJ, Kladde MP.

Mol Cell Biol. 2009 Sep;29(18):4891-905. doi: 10.1128/MCB.00222-09. Epub 2009 Jul 13.


The forkhead protein Fkh2 is a component of the yeast cell cycle transcription factor SFF.

Pic A, Lim FL, Ross SJ, Veal EA, Johnson AL, Sultan MR, West AG, Johnston LH, Sharrocks AD, Morgan BA.

EMBO J. 2000 Jul 17;19(14):3750-61.


Genome-wide distribution of ORC and MCM proteins in S. cerevisiae: high-resolution mapping of replication origins.

Wyrick JJ, Aparicio JG, Chen T, Barnett JD, Jennings EG, Young RA, Bell SP, Aparicio OM.

Science. 2001 Dec 14;294(5550):2357-60.


The requirement of yeast replication origins for pre-replication complex proteins is modulated by transcription.

Nieduszynski CA, Blow JJ, Donaldson AD.

Nucleic Acids Res. 2005 Apr 28;33(8):2410-20. Print 2005.


Behavior of replication origins in Eukaryota - spatio-temporal dynamics of licensing and firing.

Musiałek MW, Rybaczek D.

Cell Cycle. 2015;14(14):2251-64. doi: 10.1080/15384101.2015.1056421. Epub 2015 Jun 1. Review.


Chromatin-dependent and -independent regulation of DNA replication origin activation in budding yeast.

Lõoke M, Kristjuhan K, Värv S, Kristjuhan A.

EMBO Rep. 2013 Feb;14(2):191-8. doi: 10.1038/embor.2012.196. Epub 2012 Dec 7.


Forkhead transcription factors, Fkh1p and Fkh2p, collaborate with Mcm1p to control transcription required for M-phase.

Kumar R, Reynolds DM, Shevchenko A, Shevchenko A, Goldstone SD, Dalton S.

Curr Biol. 2000 Jul 27-Aug 10;10(15):896-906.


Two yeast forkhead genes regulate the cell cycle and pseudohyphal growth.

Zhu G, Spellman PT, Volpe T, Brown PO, Botstein D, Davis TN, Futcher B.

Nature. 2000 Jul 6;406(6791):90-4.


Rif1 regulates initiation timing of late replication origins throughout the S. cerevisiae genome.

Peace JM, Ter-Zakarian A, Aparicio OM.

PLoS One. 2014 May 30;9(5):e98501. doi: 10.1371/journal.pone.0098501. eCollection 2014.


Cell cycle dependent topological changes of chromosomal replication origins in Saccharomyces cerevisiae.

Fujita M, Hori Y, Shirahige K, Tsurimoto T, Yoshikawa H, Obuse C.

Genes Cells. 1998 Nov;3(11):737-49.


Origin single-stranded DNA releases Sld3 protein from the Mcm2-7 complex, allowing the GINS tetramer to bind the Mcm2-7 complex.

Bruck I, Kaplan DL.

J Biol Chem. 2011 May 27;286(21):18602-13. doi: 10.1074/jbc.M111.226332. Epub 2011 Apr 1.

Items per page
Sort by

Send to:

Choose Destination

Supplemental Content

Write to the Help Desk