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Items: 1 to 20 of 107

1.

Histamine H4 receptor optimizes T regulatory cell frequency and facilitates anti-inflammatory responses within the central nervous system.

del Rio R, Noubade R, Saligrama N, Wall EH, Krementsov DN, Poynter ME, Zachary JF, Thurmond RL, Teuscher C.

J Immunol. 2012 Jan 15;188(2):541-7. doi: 10.4049/jimmunol.1101498. Epub 2011 Dec 5.

2.

Systemic lack of canonical histamine receptor signaling results in increased resistance to autoimmune encephalomyelitis.

Saligrama N, Case LK, del Rio R, Noubade R, Teuscher C.

J Immunol. 2013 Jul 15;191(2):614-22. doi: 10.4049/jimmunol.1203137. Epub 2013 Jun 14.

3.

CD44 deficiency contributes to enhanced experimental autoimmune encephalomyelitis: a role in immune cells and vascular cells of the blood-brain barrier.

Flynn KM, Michaud M, Madri JA.

Am J Pathol. 2013 Apr;182(4):1322-36. doi: 10.1016/j.ajpath.2013.01.003. Epub 2013 Feb 12.

4.

Combinatorial roles for histamine H1-H2 and H3-H4 receptors in autoimmune inflammatory disease of the central nervous system.

Saligrama N, Noubade R, Case LK, del Rio R, Teuscher C.

Eur J Immunol. 2012 Jun;42(6):1536-46. doi: 10.1002/eji.201141859.

5.

Deletion of the G2A receptor fails to attenuate experimental autoimmune encephalomyelitis.

Osmers I, Smith SS, Parks BW, Yu S, Srivastava R, Wohler JE, Barnum SR, Kabarowski JH.

J Neuroimmunol. 2009 Feb 15;207(1-2):18-23. doi: 10.1016/j.jneuroim.2008.11.008. Epub 2009 Jan 9.

7.

Resistance to myelin oligodendrocyte glycoprotein-induced experimental autoimmune encephalomyelitis by death receptor 6-deficient mice.

Schmidt CS, Zhao J, Chain J, Hepburn D, Gitter B, Sandusky G, Chintalacharuvu S, Glasebrook A, Na S.

J Immunol. 2005 Aug 15;175(4):2286-92.

8.

Histamine H₂ receptor signaling × environment interactions determine susceptibility to experimental allergic encephalomyelitis.

Saligrama N, Case LK, Krementsov DN, Teuscher C.

FASEB J. 2014 Apr;28(4):1898-909. doi: 10.1096/fj.13-239939. Epub 2013 Dec 26.

9.

Histamine H(3) receptor integrates peripheral inflammatory signals in the neurogenic control of immune responses and autoimmune disease susceptibility.

Krementsov DN, Wall EH, Martin RA, Subramanian M, Noubade R, Del Rio R, Mawe GM, Bond JP, Poynter ME, Blankenhorn EP, Teuscher C.

PLoS One. 2013 Jul 22;8(7):e62743. doi: 10.1371/journal.pone.0062743. Print 2013.

10.

Galectin-3 deficiency reduces the severity of experimental autoimmune encephalomyelitis.

Jiang HR, Al Rasebi Z, Mensah-Brown E, Shahin A, Xu D, Goodyear CS, Fukada SY, Liu FT, Liew FY, Lukic ML.

J Immunol. 2009 Jan 15;182(2):1167-73.

11.

Antagonism of histamine H4 receptors exacerbates clinical and pathological signs of experimental autoimmune encephalomyelitis.

Ballerini C, Aldinucci A, Luccarini I, Galante A, Manuelli C, Blandina P, Katebe M, Chazot PL, Masini E, Passani MB.

Br J Pharmacol. 2013 Sep;170(1):67-77. doi: 10.1111/bph.12263.

12.

Kit (W-sh) mice develop earlier and more severe experimental autoimmune encephalomyelitis due to absence of immune suppression.

Li H, Nourbakhsh B, Safavi F, Li K, Xu H, Cullimore M, Zhou F, Zhang G, Rostami A.

J Immunol. 2011 Jul 1;187(1):274-82. doi: 10.4049/jimmunol.1003603. Epub 2011 Jun 6.

13.

Peroxisome proliferator-activated receptor-gamma-deficient heterozygous mice develop an exacerbated neural antigen-induced Th1 response and experimental allergic encephalomyelitis.

Natarajan C, Muthian G, Barak Y, Evans RM, Bright JJ.

J Immunol. 2003 Dec 1;171(11):5743-50. Erratum in: J Immunol. 2004 Apr 15;172(8):following 5127.

14.

Immune regulatory CNS-reactive CD8+T cells in experimental autoimmune encephalomyelitis.

York NR, Mendoza JP, Ortega SB, Benagh A, Tyler AF, Firan M, Karandikar NJ.

J Autoimmun. 2010 Aug;35(1):33-44. doi: 10.1016/j.jaut.2010.01.003. Epub 2010 Feb 20.

15.

The histamine H4 receptor mediates allergic airway inflammation by regulating the activation of CD4+ T cells.

Dunford PJ, O'Donnell N, Riley JP, Williams KN, Karlsson L, Thurmond RL.

J Immunol. 2006 Jun 1;176(11):7062-70.

16.

B-cell activation influences T-cell polarization and outcome of anti-CD20 B-cell depletion in central nervous system autoimmunity.

Weber MS, Prod'homme T, Patarroyo JC, Molnarfi N, Karnezis T, Lehmann-Horn K, Danilenko DM, Eastham-Anderson J, Slavin AJ, Linington C, Bernard CC, Martin F, Zamvil SS.

Ann Neurol. 2010 Sep;68(3):369-83. doi: 10.1002/ana.22081.

18.

Central histamine H3 receptor signaling negatively regulates susceptibility to autoimmune inflammatory disease of the CNS.

Teuscher C, Subramanian M, Noubade R, Gao JF, Offner H, Zachary JF, Blankenhorn EP.

Proc Natl Acad Sci U S A. 2007 Jun 12;104(24):10146-51. Epub 2007 Jun 4.

19.

H(1)R expression by CD11B(+) cells is not required for susceptibility to experimental allergic encephalomyelitis.

Saligrama N, Noubade R, Case LK, Poynter ME, Teuscher C.

Cell Immunol. 2012 Jul-Aug;278(1-2):27-34. doi: 10.1016/j.cellimm.2012.06.012. Epub 2012 Jul 14.

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