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Items: 1 to 20 of 84

1.

Subunits Rip1p and Cox9p of the respiratory chain contribute to diclofenac-induced mitochondrial dysfunction.

van Leeuwen JS, Orij R, Luttik MA, Smits GJ, Vermeulen NP, Vos JC.

Microbiology. 2011 Mar;157(Pt 3):685-94. doi: 10.1099/mic.0.044578-0. Epub 2010 Dec 9.

PMID:
21148204
2.

Metabolism related toxicity of diclofenac in yeast as model system.

van Leeuwen JS, Vredenburg G, Dragovic S, Tjong TF, Vos JC, Vermeulen NP.

Toxicol Lett. 2011 Feb 5;200(3):162-8. doi: 10.1016/j.toxlet.2010.11.010. Epub 2010 Nov 24.

PMID:
21111035
6.

Cisplatin-induced nephrotoxicity in porcine proximal tubular cells: mitochondrial dysfunction by inhibition of complexes I to IV of the respiratory chain.

Kruidering M, Van de Water B, de Heer E, Mulder GJ, Nagelkerke JF.

J Pharmacol Exp Ther. 1997 Feb;280(2):638-49.

7.

Differential involvement of mitochondrial dysfunction, cytochrome P450 activity, and active transport in the toxicity of structurally related NSAIDs.

van Leeuwen JS, Unlü B, Vermeulen NP, Vos JC.

Toxicol In Vitro. 2012 Mar;26(2):197-205. doi: 10.1016/j.tiv.2011.11.013. Epub 2011 Nov 26.

PMID:
22138569
8.

Diclofenac toxicity to hepatocytes: a role for drug metabolism in cell toxicity.

Bort R, Ponsoda X, Jover R, Gómez-Lechón MJ, Castell JV.

J Pharmacol Exp Ther. 1999 Jan;288(1):65-72.

9.
10.

The novel equisetin-like compound, TA-289, causes aberrant mitochondrial morphology which is independent of the production of reactive oxygen species in Saccharomyces cerevisiae.

Quek NC, Matthews JH, Bloor SJ, Jones DA, Bircham PW, Heathcott RW, Atkinson PH.

Mol Biosyst. 2013 Aug;9(8):2125-33. doi: 10.1039/c3mb70056a. Epub 2013 May 28.

PMID:
23715404
11.

Production of reactive oxygen species and loss of viability in yeast mitochondrial mutants: protective effect of Bcl-xL.

Trancíková A, Weisová P, Kissová I, Zeman I, Kolarov J.

FEMS Yeast Res. 2004 Nov;5(2):149-56.

12.

Combined effects of aging and in vitro non-steroid anti-inflammatory drugs on kidney and liver mitochondrial physiology.

Rocha-Rodrigues S, Santos-Alves E, Coxito PM, Marques-Aleixo I, Passos E, Guimarães JT, Martins MJ, Oliveira PJ, Magalhães J, Ascensão A.

Life Sci. 2013 Sep 3;93(8):329-37. doi: 10.1016/j.lfs.2013.07.004. Epub 2013 Jul 17.

PMID:
23872100
13.

Inactivation of the 20S proteasome maturase, Ump1p, leads to the instability of mtDNA in Saccharomyces cerevisiae.

Malc E, Dzierzbicki P, Kaniak A, Skoneczna A, Ciesla Z.

Mutat Res. 2009 Oct 2;669(1-2):95-103. doi: 10.1016/j.mrfmmm.2009.05.008. Epub 2009 May 23.

PMID:
19467248
14.
15.

A chemical genetic screen for modulators of asymmetrical 2,2'-dimeric naphthoquinones cytotoxicity in yeast.

Emadi A, Ross AE, Cowan KM, Fortenberry YM, Vuica-Ross M.

PLoS One. 2010 May 26;5(5):e10846. doi: 10.1371/journal.pone.0010846.

16.

Exercise mitigates diclofenac-induced liver mitochondrial dysfunction.

Santos-Alves E, Marques-Aleixo I, Coxito P, Balça MM, Rizo-Roca D, Rocha-Rodrigues S, Martins S, Torrella JR, Oliveira PJ, Moreno AJ, Magalhães J, Ascensão A.

Eur J Clin Invest. 2014 Jul;44(7):668-77. doi: 10.1111/eci.12285.

PMID:
24889192
17.

Diclofenac-induced liver injury: a paradigm of idiosyncratic drug toxicity.

Boelsterli UA.

Toxicol Appl Pharmacol. 2003 Nov 1;192(3):307-22. Review.

PMID:
14575648
18.

Involvement of the pleiotropic drug resistance response, protein kinase C signaling, and altered zinc homeostasis in resistance of Saccharomyces cerevisiae to diclofenac.

van Leeuwen JS, Vermeulen NP, Vos JC.

Appl Environ Microbiol. 2011 Sep;77(17):5973-80. doi: 10.1128/AEM.00253-11. Epub 2011 Jul 1.

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