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Items: 1 to 20 of 90

1.

[Lead exposure markers and related susceptibility genes based on different people].

Chen H, Liu L, Cao Z, Qu W.

Wei Sheng Yan Jiu. 2010 Sep;39(5):643-6. Review. Chinese.

PMID:
21033449
2.

The relevance of the individual genetic background for the toxicokinetics of two significant neurodevelopmental toxicants: mercury and lead.

Gundacker C, Gencik M, Hengstschläger M.

Mutat Res. 2010 Oct;705(2):130-40. doi: 10.1016/j.mrrev.2010.06.003. Epub 2010 Jun 30. Review.

PMID:
20601101
3.

Genetic susceptibility to lead poisoning.

Onalaja AO, Claudio L.

Environ Health Perspect. 2000 Mar;108 Suppl 1:23-8. Review.

4.

[Genetic aspects of hypertensive effect of lead].

Skoczyńska A.

Med Pr. 2008;59(4):325-32. Review. Polish.

PMID:
19143224
5.

[Molecular genetic susceptibility to lead poisoning].

Wu S, Yan C, Shen X.

Wei Sheng Yan Jiu. 2004 Mar;33(2):226-8, 232. Review. Chinese.

PMID:
15209014
6.

Genotoxic effects of occupational exposure to lead and influence of polymorphisms in genes involved in lead toxicokinetics and in DNA repair.

García-Lestón J, Roma-Torres J, Vilares M, Pinto R, Prista J, Teixeira JP, Mayan O, Conde J, Pingarilho M, Gaspar JF, Pásaro E, Méndez J, Laffon B.

Environ Int. 2012 Aug;43:29-36. doi: 10.1016/j.envint.2012.03.001. Epub 2012 Mar 30.

PMID:
22466227
7.

Modification by the genes ALAD and VDR of lead-induced cognitive effects in children.

Pawlas N, Broberg K, Olewińska E, Prokopowicz A, Skerfving S, Pawlas K.

Neurotoxicology. 2012 Jan;33(1):37-43. doi: 10.1016/j.neuro.2011.10.012. Epub 2011 Nov 9.

PMID:
22101007
8.

Lead exposure suppressed ALAD transcription by increasing methylation level of the promoter CpG islands.

Li C, Xu M, Wang S, Yang X, Zhou S, Zhang J, Liu Q, Sun Y.

Toxicol Lett. 2011 May 30;203(1):48-53. doi: 10.1016/j.toxlet.2011.03.002. Epub 2011 Mar 9.

PMID:
21396434
9.

Environmental and occupational exposure to lead as a potential risk factor for cardiovascular disease.

Poręba R, Gać P, Poręba M, Andrzejak R.

Environ Toxicol Pharmacol. 2011 Mar;31(2):267-77. doi: 10.1016/j.etap.2010.12.002. Epub 2010 Dec 17. Review.

PMID:
21787694
10.

Assessment of how pregnancy modifies plasma lead and plasma/whole blood lead ratio in ALAD 1-1 genotype women.

Montenegro MF, Barbosa F Jr, Tanus-Santos JE.

Basic Clin Pharmacol Toxicol. 2008 Apr;102(4):347-51. doi: 10.1111/j.1742-7843.2007.00205.x. Epub 2008 Feb 12.

11.

Delta-aminolevulinic acid dehydratase genotype and lead toxicity: a HuGE review.

Kelada SN, Shelton E, Kaufmann RB, Khoury MJ.

Am J Epidemiol. 2001 Jul 1;154(1):1-13. Review.

12.

Co-exposure to environmental lead and manganese affects the intelligence of school-aged children.

Kim Y, Kim BN, Hong YC, Shin MS, Yoo HJ, Kim JW, Bhang SY, Cho SC.

Neurotoxicology. 2009 Jul;30(4):564-71. doi: 10.1016/j.neuro.2009.03.012. Epub 2009 Apr 14.

PMID:
19635390
13.

[Study of ALAD and VDR gene polymorphisms associated with lead nephrotoxicity susceptibility].

Wang A, Wang Q, Song Q, Xu J.

Wei Sheng Yan Jiu. 2009 May;38(3):326-9. Chinese.

PMID:
19548578
14.

The present status of biological effects of toxic metals in the environment: lead, cadmium, and manganese.

Shukla GS, Singhal RL.

Can J Physiol Pharmacol. 1984 Aug;62(8):1015-31. Review.

PMID:
6149004
15.

Lead contamination in Uruguay: the "La Teja" neighborhood case.

Mañay N, Cousillas AZ, Alvarez C, Heller T.

Rev Environ Contam Toxicol. 2008;195:93-115. Review.

PMID:
18418955
16.

Assessment of immunotoxicity parameters in individuals occupationally exposed to lead.

García-Lestón J, Roma-Torres J, Mayan O, Schroecksnadel S, Fuchs D, Moreira AO, Pásaro E, Méndez J, Teixeira JP, Laffon B.

J Toxicol Environ Health A. 2012;75(13-15):807-18. doi: 10.1080/15287394.2012.690327.

PMID:
22788368
17.

Genetic background of lead and mercury metabolism in a group of medical students in Austria.

Gundacker C, Wittmann KJ, Kukuckova M, Komarnicki G, Hikkel I, Gencik M.

Environ Res. 2009 Aug;109(6):786-96. doi: 10.1016/j.envres.2009.05.003. Epub 2009 Jun 9.

PMID:
19515364
18.

Effect modification by delta-aminolevulinic acid dehydratase, vitamin D receptor, and nitric oxide synthase gene polymorphisms on associations between patella lead and renal function in lead workers.

Weaver VM, Lee BK, Todd AC, Ahn KD, Shi W, Jaar BG, Kelsey KT, Lustberg ME, Silbergeld EK, Parsons PJ, Wen J, Schwartz BS.

Environ Res. 2006 Sep;102(1):61-9. Epub 2006 Feb 17.

PMID:
16487505
19.

A polymorphism in the delta-aminolevulinic acid dehydratase gene modifies plasma/whole blood lead ratio.

Montenegro MF, Barbosa F Jr, Sandrim VC, Gerlach RF, Tanus-Santos JE.

Arch Toxicol. 2006 Jul;80(7):394-8. Epub 2005 Dec 9.

PMID:
16341691
20.

Transient loss of the Y-chromosome in an elderly man with anemia and lead poisoning: chance occurrence or a clonal marker of the underlying hematological abnormality?

Mantadakis E, Boula AM, Girakis G, Xilouri IM, Foudoulakis AM, Samonis G.

Haematologica. 2006 Aug;91(8 Suppl):ECR37.

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