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Results: 1 to 20 of 109

Similar articles for PubMed (Select 20661276)

1.

Gene network disruptions and neurogenesis defects in the adult Ts1Cje mouse model of Down syndrome.

Hewitt CA, Ling KH, Merson TD, Simpson KM, Ritchie ME, King SL, Pritchard MA, Smyth GK, Thomas T, Scott HS, Voss AK.

PLoS One. 2010 Jul 16;5(7):e11561. doi: 10.1371/journal.pone.0011561.

2.

The fetal brain transcriptome and neonatal behavioral phenotype in the Ts1Cje mouse model of Down syndrome.

Guedj F, Pennings JL, Ferres MA, Graham LC, Wick HC, Miczek KA, Slonim DK, Bianchi DW.

Am J Med Genet A. 2015 May 14. doi: 10.1002/ajmg.a.37156. [Epub ahead of print]

PMID:
25975229
3.

Brief report: isogenic induced pluripotent stem cell lines from an adult with mosaic down syndrome model accelerated neuronal ageing and neurodegeneration.

Murray A, Letourneau A, Canzonetta C, Stathaki E, Gimelli S, Sloan-Bena F, Abrehart R, Goh P, Lim S, Baldo C, Dagna-Bricarelli F, Hannan S, Mortensen M, Ballard D, Syndercombe Court D, Fusaki N, Hasegawa M, Smart TG, Bishop C, Antonarakis SE, Groet J, Nizetic D.

Stem Cells. 2015 Jun;33(6):2077-84. doi: 10.1002/stem.1968.

PMID:
25694335
4.

Stem and progenitor cell dysfunction in human trisomies.

Liu B, Filippi S, Roy A, Roberts I.

EMBO Rep. 2015 Jan;16(1):44-62. doi: 10.15252/embr.201439583. Epub 2014 Dec 17.

PMID:
25520324
5.

Comparative proteomic profiling reveals aberrant cell proliferation in the brain of embryonic Ts1Cje, a mouse model of Down syndrome.

Ishihara K, Kanai S, Sago H, Yamakawa K, Akiba S.

Neuroscience. 2014 Sep 28;281C:1-15. doi: 10.1016/j.neuroscience.2014.09.039. [Epub ahead of print]

PMID:
25261685
6.

Functional transcriptome analysis of the postnatal brain of the Ts1Cje mouse model for Down syndrome reveals global disruption of interferon-related molecular networks.

Ling KH, Hewitt CA, Tan KL, Cheah PS, Vidyadaran S, Lai MI, Lee HC, Simpson K, Hyde L, Pritchard MA, Smyth GK, Thomas T, Scott HS.

BMC Genomics. 2014 Jul 22;15:624. doi: 10.1186/1471-2164-15-624.

7.

[Mechanisms for the differentiation of postnatal and adult neural stem cells: new insights and pathological issues based on the analysis of Girdin].

Enomoto A, Asai N, Takahashi M.

Nihon Yakurigaku Zasshi. 2014 Jun;143(6):289-94. Review. Japanese. No abstract available.

PMID:
24919555
8.

Analysis of adult cerebral cortex and hippocampus transcriptomes reveals unique molecular changes in the Ts1Cje mouse model of down syndrome.

Guedj F, Pennings JL, Wick HC, Bianchi DW.

Brain Pathol. 2015 Jan;25(1):11-23. doi: 10.1111/bpa.12151. Epub 2014 Jul 25.

PMID:
24916381
9.

Prenatal treatment of Down syndrome: a reality?

Guedj F, Bianchi DW, Delabar JM.

Curr Opin Obstet Gynecol. 2014 Apr;26(2):92-103. doi: 10.1097/GCO.0000000000000056. Review.

PMID:
24573065
10.

Modelling and rescuing neurodevelopmental defect of Down syndrome using induced pluripotent stem cells from monozygotic twins discordant for trisomy 21.

Hibaoui Y, Grad I, Letourneau A, Sailani MR, Dahoun S, Santoni FA, Gimelli S, Guipponi M, Pelte MF, Béna F, Antonarakis SE, Feki A.

EMBO Mol Med. 2014 Feb;6(2):259-77. doi: 10.1002/emmm.201302848. Epub 2013 Dec 27.

11.

Increased dosage of DYRK1A and DSCR1 delays neuronal differentiation in neocortical progenitor cells.

Kurabayashi N, Sanada K.

Genes Dev. 2013 Dec 15;27(24):2708-21. doi: 10.1101/gad.226381.113.

12.

Usp16: key controller of stem cells in Down syndrome.

Xu JC, Dawson VL, Dawson TM.

EMBO J. 2013 Oct 30;32(21):2788-9. doi: 10.1038/emboj.2013.220. Epub 2013 Sep 27. No abstract available.

13.

Surveying the Down syndrome mouse model resource identifies critical regions responsible for chronic otitis media.

Bhutta MF, Cheeseman MT, Herault Y, Yu YE, Brown SD.

Mamm Genome. 2013 Dec;24(11-12):439-45. doi: 10.1007/s00335-013-9475-x. Epub 2013 Sep 26.

14.

Usp16 contributes to somatic stem-cell defects in Down's syndrome.

Adorno M, Sikandar S, Mitra SS, Kuo A, Nicolis Di Robilant B, Haro-Acosta V, Ouadah Y, Quarta M, Rodriguez J, Qian D, Reddy VM, Cheshier S, Garner CC, Clarke MF.

Nature. 2013 Sep 19;501(7467):380-4. doi: 10.1038/nature12530. Epub 2013 Sep 11.

15.
16.

Non-trisomic homeobox gene expression during craniofacial development in the Ts65Dn mouse model of Down syndrome.

Billingsley CN, Allen JR, Baumann DD, Deitz SL, Blazek JD, Newbauer A, Darrah A, Long BC, Young B, Clement M, Doerge RW, Roper RJ.

Am J Med Genet A. 2013 Aug;161A(8):1866-74. doi: 10.1002/ajmg.a.36006. Epub 2013 Jul 10.

17.

The amyloid precursor protein (APP) triplicated gene impairs neuronal precursor differentiation and neurite development through two different domains in the Ts65Dn mouse model for Down syndrome.

Trazzi S, Fuchs C, Valli E, Perini G, Bartesaghi R, Ciani E.

J Biol Chem. 2013 Jul 19;288(29):20817-29. doi: 10.1074/jbc.M113.451088. Epub 2013 Jun 5.

18.

Deficits in human trisomy 21 iPSCs and neurons.

Weick JP, Held DL, Bonadurer GF 3rd, Doers ME, Liu Y, Maguire C, Clark A, Knackert JA, Molinarolo K, Musser M, Yao L, Yin Y, Lu J, Zhang X, Zhang SC, Bhattacharyya A.

Proc Natl Acad Sci U S A. 2013 Jun 11;110(24):9962-7. doi: 10.1073/pnas.1216575110. Epub 2013 May 28.

19.

Commonality in Down and fetal alcohol syndromes.

Solzak JP, Liang Y, Zhou FC, Roper RJ.

Birth Defects Res A Clin Mol Teratol. 2013 Apr;97(4):187-97. doi: 10.1002/bdra.23129. Epub 2013 Apr 3.

20.
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