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Results: 1 to 20 of 99

1.

Mitochondrial superoxide contributes to blood flow and axonal transport deficits in the Tg2576 mouse model of Alzheimer's disease.

Massaad CA, Amin SK, Hu L, Mei Y, Klann E, Pautler RG.

PLoS One. 2010 May 10;5(5):e10561. doi: 10.1371/journal.pone.0010561.

PMID:
20479943
[PubMed - indexed for MEDLINE]
Free PMC Article
2.
3.

Overexpression of SOD-2 reduces hippocampal superoxide and prevents memory deficits in a mouse model of Alzheimer's disease.

Massaad CA, Washington TM, Pautler RG, Klann E.

Proc Natl Acad Sci U S A. 2009 Aug 11;106(32):13576-81. doi: 10.1073/pnas.0902714106. Epub 2009 Aug 3.

PMID:
19666610
[PubMed - indexed for MEDLINE]
Free PMC Article
4.

Abeta exacerbates the neuronal dysfunction caused by human tau expression in a Drosophila model of Alzheimer's disease.

Folwell J, Cowan CM, Ubhi KK, Shiabh H, Newman TA, Shepherd D, Mudher A.

Exp Neurol. 2010 Jun;223(2):401-9. doi: 10.1016/j.expneurol.2009.09.014. Epub 2009 Sep 24.

PMID:
19782075
[PubMed - indexed for MEDLINE]
5.

Axonal transport, tau protein, and neurodegeneration in Alzheimer's disease.

Terwel D, Dewachter I, Van Leuven F.

Neuromolecular Med. 2002;2(2):151-65. Review.

PMID:
12428809
[PubMed - indexed for MEDLINE]
6.

Current advances on different kinases involved in tau phosphorylation, and implications in Alzheimer's disease and tauopathies.

Ferrer I, Gomez-Isla T, Puig B, Freixes M, Ribé E, Dalfó E, Avila J.

Curr Alzheimer Res. 2005 Jan;2(1):3-18. Review.

PMID:
15977985
[PubMed - indexed for MEDLINE]
7.

A novel glycogen synthase kinase-3 inhibitor 2-methyl-5-(3-{4-[(S )-methylsulfinyl]phenyl}-1-benzofuran-5-yl)-1,3,4-oxadiazole decreases tau phosphorylation and ameliorates cognitive deficits in a transgenic model of Alzheimer's disease.

Onishi T, Iwashita H, Uno Y, Kunitomo J, Saitoh M, Kimura E, Fujita H, Uchiyama N, Kori M, Takizawa M.

J Neurochem. 2011 Dec;119(6):1330-40. doi: 10.1111/j.1471-4159.2011.07532.x. Epub 2011 Nov 2.

PMID:
21992552
[PubMed - indexed for MEDLINE]
8.

Amyloid reduction by amyloid-beta vaccination also reduces mouse tau pathology and protects from neuron loss in two mouse models of Alzheimer's disease.

Wilcock DM, Gharkholonarehe N, Van Nostrand WE, Davis J, Vitek MP, Colton CA.

J Neurosci. 2009 Jun 24;29(25):7957-65. doi: 10.1523/JNEUROSCI.1339-09.2009. Erratum in: J Neurosci. 2010 Jan 20;30(3):1197-8.

PMID:
19553436
[PubMed - indexed for MEDLINE]
Free PMC Article
9.

Abnormal tau, mitochondrial dysfunction, impaired axonal transport of mitochondria, and synaptic deprivation in Alzheimer's disease.

Reddy PH.

Brain Res. 2011 Sep 30;1415:136-48. doi: 10.1016/j.brainres.2011.07.052. Epub 2011 Jul 31. Review.

PMID:
21872849
[PubMed - indexed for MEDLINE]
Free PMC Article
10.

Mitochondria and vascular lesions as a central target for the development of Alzheimer's disease and Alzheimer disease-like pathology in transgenic mice.

Aliev G, Seyidova D, Lamb BT, Obrenovich ME, Siedlak SL, Vinters HV, Friedland RP, LaManna JC, Smith MA, Perry G.

Neurol Res. 2003 Sep;25(6):665-74.

PMID:
14503022
[PubMed - indexed for MEDLINE]
11.

β-Amyloid, cholinergic transmission, and cerebrovascular system -- a developmental study in a mouse model of Alzheimer's disease.

Kuznetsova E, Schliebs R.

Curr Pharm Des. 2013;19(38):6749-65.

PMID:
23530514
[PubMed - indexed for MEDLINE]
12.

Convergence of amyloid-beta and tau pathologies on mitochondria in vivo.

Eckert A, Schulz KL, Rhein V, Götz J.

Mol Neurobiol. 2010 Jun;41(2-3):107-14. doi: 10.1007/s12035-010-8109-5. Epub 2010 Mar 9. Review.

PMID:
20217279
[PubMed - indexed for MEDLINE]
Free PMC Article
13.

Transgenic mice overexpressing APP and transforming growth factor-beta1 feature cognitive and vascular hallmarks of Alzheimer's disease.

Ongali B, Nicolakakis N, Lecrux C, Aboulkassim T, Rosa-Neto P, Papadopoulos P, Tong XK, Hamel E.

Am J Pathol. 2010 Dec;177(6):3071-80. doi: 10.2353/ajpath.2010.100339. Epub 2010 Nov 18.

PMID:
21088218
[PubMed - indexed for MEDLINE]
Free PMC Article
14.

Potassium 2-(1-hydroxypentyl)-benzoate improves memory deficits and attenuates amyloid and τ pathologies in a mouse model of Alzheimer's disease.

Peng Y, Hu Y, Xu S, Rong X, Li J, Li P, Wang L, Yang J, Wang X.

J Pharmacol Exp Ther. 2014 Aug;350(2):361-74. doi: 10.1124/jpet.114.213140. Epub 2014 Jun 3.

PMID:
24893984
[PubMed - indexed for MEDLINE]
15.

APP/PS1 mice overexpressing SREBP-2 exhibit combined Aβ accumulation and tau pathology underlying Alzheimer's disease.

Barbero-Camps E, Fernández A, Martínez L, Fernández-Checa JC, Colell A.

Hum Mol Genet. 2013 Sep 1;22(17):3460-76. doi: 10.1093/hmg/ddt201. Epub 2013 May 6.

PMID:
23648430
[PubMed - indexed for MEDLINE]
Free PMC Article
16.

Quantitative in vivo measurement of early axonal transport deficits in a triple transgenic mouse model of Alzheimer's disease using manganese-enhanced MRI.

Kim J, Choi IY, Michaelis ML, Lee P.

Neuroimage. 2011 Jun 1;56(3):1286-92. doi: 10.1016/j.neuroimage.2011.02.039. Epub 2011 Feb 19.

PMID:
21338698
[PubMed - indexed for MEDLINE]
Free PMC Article
17.

Amyloid-beta and tau synergistically impair the oxidative phosphorylation system in triple transgenic Alzheimer's disease mice.

Rhein V, Song X, Wiesner A, Ittner LM, Baysang G, Meier F, Ozmen L, Bluethmann H, Dröse S, Brandt U, Savaskan E, Czech C, Götz J, Eckert A.

Proc Natl Acad Sci U S A. 2009 Nov 24;106(47):20057-62. doi: 10.1073/pnas.0905529106. Epub 2009 Nov 6.

PMID:
19897719
[PubMed - indexed for MEDLINE]
Free PMC Article
18.

Mitochondrial oxidative stress causes hyperphosphorylation of tau.

Melov S, Adlard PA, Morten K, Johnson F, Golden TR, Hinerfeld D, Schilling B, Mavros C, Masters CL, Volitakis I, Li QX, Laughton K, Hubbard A, Cherny RA, Gibson B, Bush AI.

PLoS One. 2007 Jun 20;2(6):e536.

PMID:
17579710
[PubMed - indexed for MEDLINE]
Free PMC Article
19.

Amyloid β-induced impairments in hippocampal synaptic plasticity are rescued by decreasing mitochondrial superoxide.

Ma T, Hoeffer CA, Wong H, Massaad CA, Zhou P, Iadecola C, Murphy MP, Pautler RG, Klann E.

J Neurosci. 2011 Apr 13;31(15):5589-95. doi: 10.1523/JNEUROSCI.6566-10.2011.

PMID:
21490199
[PubMed - indexed for MEDLINE]
Free PMC Article
20.

Abnormal interaction between the mitochondrial fission protein Drp1 and hyperphosphorylated tau in Alzheimer's disease neurons: implications for mitochondrial dysfunction and neuronal damage.

Manczak M, Reddy PH.

Hum Mol Genet. 2012 Jun 1;21(11):2538-47. doi: 10.1093/hmg/dds072. Epub 2012 Feb 24.

PMID:
22367970
[PubMed - indexed for MEDLINE]
Free PMC Article

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