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Items: 1 to 20 of 156

1.

The Na+-dependent chloride-bicarbonate exchanger SLC4A8 mediates an electroneutral Na+ reabsorption process in the renal cortical collecting ducts of mice.

Leviel F, Hübner CA, Houillier P, Morla L, El Moghrabi S, Brideau G, Hassan H, Parker MD, Kurth I, Kougioumtzes A, Sinning A, Pech V, Riemondy KA, Miller RL, Hummler E, Shull GE, Aronson PS, Doucet A, Wall SM, Chambrey R, Eladari D.

J Clin Invest. 2010 May;120(5):1627-35. doi: 10.1172/JCI40145. Epub 2010 Apr 12. Erratum in: J Clin Invest. 2011 Apr 1;121(4):1668. Hatim, Hassan [corrected to Hassan, Hatim].

2.

Nitric oxide reduces Cl⁻ absorption in the mouse cortical collecting duct through an ENaC-dependent mechanism.

Pech V, Thumova M, Dikalov SI, Hummler E, Rossier BC, Harrison DG, Wall SM.

Am J Physiol Renal Physiol. 2013 Jun 1;304(11):F1390-7. doi: 10.1152/ajprenal.00292.2012. Epub 2013 Mar 20.

3.

Novel mechanisms for NaCl reabsorption in the collecting duct.

Eladari D, Hübner CA.

Curr Opin Nephrol Hypertens. 2011 Sep;20(5):506-11. doi: 10.1097/MNH.0b013e3283486c4a. Review.

PMID:
21610493
4.

ENaC inhibition stimulates Cl- secretion in the mouse cortical collecting duct through an NKCC1-dependent mechanism.

Pech V, Thumova M, Kim YH, Agazatian D, Hummler E, Rossier BC, Weinstein AM, Nanami M, Wall SM.

Am J Physiol Renal Physiol. 2012 Jul 1;303(1):F45-55. doi: 10.1152/ajprenal.00030.2012. Epub 2012 Apr 11.

5.

Deletion of the anion exchanger Slc26a4 (pendrin) decreases apical Cl(-)/HCO3(-) exchanger activity and impairs bicarbonate secretion in kidney collecting duct.

Amlal H, Petrovic S, Xu J, Wang Z, Sun X, Barone S, Soleimani M.

Am J Physiol Cell Physiol. 2010 Jul;299(1):C33-41. doi: 10.1152/ajpcell.00033.2010. Epub 2010 Apr 7.

6.

Acute inhibition of NCC does not activate distal electrogenic Na+ reabsorption or kaliuresis.

Hunter RW, Craigie E, Homer NZ, Mullins JJ, Bailey MA.

Am J Physiol Renal Physiol. 2014 Feb 15;306(4):F457-67. doi: 10.1152/ajprenal.00339.2013. Epub 2014 Jan 8.

7.

Cortical distal nephron Cl(-) transport in volume homeostasis and blood pressure regulation.

Wall SM, Weinstein AM.

Am J Physiol Renal Physiol. 2013 Aug 15;305(4):F427-38. doi: 10.1152/ajprenal.00022.2013. Epub 2013 May 1. Review.

8.

Double knockout of carbonic anhydrase II (CAII) and Na(+)-Cl(-) cotransporter (NCC) causes salt wasting and volume depletion.

Xu J, Barone S, Brooks MB, Soleimani M.

Cell Physiol Biochem. 2013;32(7):173-83. doi: 10.1159/000356637. Epub 2013 Dec 18.

PMID:
24429824
9.

Pendrin regulation in mouse kidney primarily is chloride-dependent.

Vallet M, Picard N, Loffing-Cueni D, Fysekidis M, Bloch-Faure M, Deschênes G, Breton S, Meneton P, Loffing J, Aronson PS, Chambrey R, Eladari D.

J Am Soc Nephrol. 2006 Aug;17(8):2153-63. Epub 2006 Jul 6.

10.

Angiotensin II increases chloride absorption in the cortical collecting duct in mice through a pendrin-dependent mechanism.

Pech V, Kim YH, Weinstein AM, Everett LA, Pham TD, Wall SM.

Am J Physiol Renal Physiol. 2007 Mar;292(3):F914-20. Epub 2006 Oct 31.

11.
12.

Thiazide-sensitive NaCl absorption in rat cortical collecting duct.

Terada Y, Knepper MA.

Am J Physiol. 1990 Sep;259(3 Pt 2):F519-28.

PMID:
2396677
13.

Reduced ENaC protein abundance contributes to the lower blood pressure observed in pendrin-null mice.

Kim YH, Pech V, Spencer KB, Beierwaltes WH, Everett LA, Green ED, Shin W, Verlander JW, Sutliff RL, Wall SM.

Am J Physiol Renal Physiol. 2007 Oct;293(4):F1314-24. Epub 2007 Aug 8.

14.

Expression of the thiazide-sensitive Na-Cl cotransporter by rabbit distal convoluted tubule cells.

Bachmann S, Velázquez H, Obermüller N, Reilly RF, Moser D, Ellison DH.

J Clin Invest. 1995 Nov;96(5):2510-4.

15.

Increased Epithelial Sodium Channel Activity Contributes to Hypertension Caused by Na+-HCO3- Cotransporter Electrogenic 2 Deficiency.

Wen D, Yuan Y, Warner PC, Wang B, Cornelius RJ, Wang-France J, Li H, Boettger T, Sansom SC.

Hypertension. 2015 Jul;66(1):68-74. doi: 10.1161/HYPERTENSIONAHA.115.05394. Epub 2015 May 4.

16.

Double Knockout of the Na+-Driven Cl-/HCO3- Exchanger and Na+/Cl- Cotransporter Induces Hypokalemia and Volume Depletion.

Sinning A, Radionov N, Trepiccione F, López-Cayuqueo KI, Jayat M, Baron S, Cornière N, Alexander RT, Hadchouel J, Eladari D, Hübner CA, Chambrey R.

J Am Soc Nephrol. 2016 May 5. pii: ASN.2015070734. [Epub ahead of print]

PMID:
27151921
17.

The role of pendrin in renal physiology.

Wall SM, Lazo-Fernandez Y.

Annu Rev Physiol. 2015;77:363-78. doi: 10.1146/annurev-physiol-021014-071854. Review.

PMID:
25668022
18.

Upregulation of Na+ transporter abundances in response to chronic thiazide or loop diuretic treatment in rats.

Na KY, Oh YK, Han JS, Joo KW, Lee JS, Earm JH, Knepper MA, Kim GH.

Am J Physiol Renal Physiol. 2003 Jan;284(1):F133-43. Epub 2002 Sep 24.

19.
20.

Enhanced passive Ca2+ reabsorption and reduced Mg2+ channel abundance explains thiazide-induced hypocalciuria and hypomagnesemia.

Nijenhuis T, Vallon V, van der Kemp AW, Loffing J, Hoenderop JG, Bindels RJ.

J Clin Invest. 2005 Jun;115(6):1651-8. Epub 2005 May 12.

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