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Items: 1 to 20 of 107

1.

Infectious arthritis and immune dysregulation: lessons from Lyme disease.

Iliopoulou BP, Huber BT.

Curr Opin Rheumatol. 2010 Jul;22(4):451-5. doi: 10.1097/BOR.0b013e328338f73f. Review.

PMID:
20375899
2.

Host-pathogen interactions and the pathogenesis of murine Lyme disease.

Weis JJ.

Curr Opin Rheumatol. 2002 Jul;14(4):399-403. Review.

PMID:
12118174
3.

HLA-DR alleles determine responsiveness to Borrelia burgdorferi antigens in a mouse model of self-perpetuating arthritis.

Iliopoulou BP, Guerau-de-Arellano M, Huber BT.

Arthritis Rheum. 2009 Dec;60(12):3831-40. doi: 10.1002/art.25005.

4.

Persistent arthritis in Borrelia burgdorferi-infected HLA-DR4-positive CD28-negative mice post-antibiotic treatment.

Iliopoulou BP, Alroy J, Huber BT.

Arthritis Rheum. 2008 Dec;58(12):3892-901. doi: 10.1002/art.24028.

5.

Gene expression profiling reveals unique pathways associated with differential severity of lyme arthritis.

Crandall H, Dunn DM, Ma Y, Wooten RM, Zachary JF, Weis JH, Weiss RB, Weis JJ.

J Immunol. 2006 Dec 1;177(11):7930-42.

7.

Toll-like receptor 2 is required for innate, but not acquired, host defense to Borrelia burgdorferi.

Wooten RM, Ma Y, Yoder RA, Brown JP, Weis JH, Zachary JF, Kirschning CJ, Weis JJ.

J Immunol. 2002 Jan 1;168(1):348-55.

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10.

Dual role for Fcγ receptors in host defense and disease in Borrelia burgdorferi-infected mice.

Belperron AA, Liu N, Booth CJ, Bockenstedt LK.

Front Cell Infect Microbiol. 2014 Jun 11;4:75. doi: 10.3389/fcimb.2014.00075. eCollection 2014.

12.

[Immunopathology of Lyme arthritis].

Pancewicz SA, Rutkowski R, Rutkowski K, Zajkowska JM, Kondrusik M.

Pol Merkur Lekarski. 2007 Aug;23(134):141-4. Review. Polish.

PMID:
18044347
13.

Mice lacking CD21 and CD35 proteins mount effective immune responses against Borrelia burgdorferi infection.

Jacobson AC, Ma Y, Zachary JF, Weis JJ, Weis JH.

Infect Immun. 2007 Apr;75(4):2075-8. Epub 2007 Feb 5.

14.

MyD88 plays a unique role in host defense but not arthritis development in Lyme disease.

Bolz DD, Sundsbak RS, Ma Y, Akira S, Kirschning CJ, Zachary JF, Weis JH, Weis JJ.

J Immunol. 2004 Aug 1;173(3):2003-10.

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Borrelia burgdorferi, an extracellular pathogen, circumvents osteopontin in inducing an inflammatory cytokine response.

Craig-Mylius K, Weber GF, Coburn J, Glickstein L.

J Leukoc Biol. 2005 May;77(5):710-8. Epub 2005 Feb 4.

18.

Distinct characteristics of resistance to Borrelia burgdorferi-induced arthritis in C57BL/6N mice.

Ma Y, Seiler KP, Eichwald EJ, Weis JH, Teuscher C, Weis JJ.

Infect Immun. 1998 Jan;66(1):161-8.

19.

Borrelia burgdorferi NapA-driven Th17 cell inflammation in lyme arthritis.

Codolo G, Amedei A, Steere AC, Papinutto E, Cappon A, Polenghi A, Benagiano M, Paccani SR, Sambri V, Del Prete G, Baldari CT, Zanotti G, Montecucco C, D'Elios MM, de Bernard M.

Arthritis Rheum. 2008 Nov;58(11):3609-17. doi: 10.1002/art.23972.

20.

Mechanisms of Borrelia burgdorferi internalization and intracellular innate immune signaling.

Petnicki-Ocwieja T, Kern A.

Front Cell Infect Microbiol. 2014 Dec 15;4:175. doi: 10.3389/fcimb.2014.00175. eCollection 2014. Review.

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