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Results: 1 to 20 of 208

Similar articles for PubMed (Select 19838693)

1.

Genome-wide association analysis of canine atopic dermatitis and identification of disease related SNPs.

Wood SH, Ke X, Nuttall T, McEwan N, Ollier WE, Carter SD.

Immunogenetics. 2009 Dec;61(11-12):765-72. doi: 10.1007/s00251-009-0402-y. Epub 2010 Jan 5.

PMID:
19838693
2.

Atopic dermatitis in West Highland white terriers is associated with a 1.3-Mb region on CFA 17.

Roque JB, O'Leary CA, Duffy DL, Kyaw-Tanner M, Gharahkhani P, Vogelnest L, Mason K, Shipstone M, Latter M.

Immunogenetics. 2012 Mar;64(3):209-17. doi: 10.1007/s00251-011-0577-x. Epub 2011 Oct 12.

PMID:
21989516
3.

Genome-wide analysis in German shepherd dogs reveals association of a locus on CFA 27 with atopic dermatitis.

Tengvall K, Kierczak M, Bergvall K, Olsson M, Frankowiack M, Farias FH, Pielberg G, Carlborg Ö, Leeb T, Andersson G, Hammarström L, Hedhammar Å, Lindblad-Toh K.

PLoS Genet. 2013 May;9(5):e1003475. doi: 10.1371/journal.pgen.1003475. Epub 2013 May 9. Erratum in: PLoS Genet. 2013 May;9(5). doi:10.1371/annotation/fd6f4425-3d84-4017-a012-a5df6ddee13a.

4.

Despite identifying some shared gene associations with human atopic dermatitis the use of multiple dog breeds from various locations limits detection of gene associations in canine atopic dermatitis.

Wood SH, Ollier WE, Nuttall T, McEwan NA, Carter SD.

Vet Immunol Immunopathol. 2010 Dec 1;138(3):193-7. doi: 10.1016/j.vetimm.2010.07.020. Epub 2010 Aug 6.

PMID:
20728225
5.

The pursuit of genome-wide association studies: where are we now?

Ku CS, Loy EY, Pawitan Y, Chia KS.

J Hum Genet. 2010 Apr;55(4):195-206. doi: 10.1038/jhg.2010.19. Epub 2010 Mar 19. Review.

PMID:
20300123
6.

Identification of susceptibility genes for complex diseases using pooling-based genome-wide association scans.

Bossé Y, Bacot F, Montpetit A, Rung J, Qu HQ, Engert JC, Polychronakos C, Hudson TJ, Froguel P, Sladek R, Desrosiers M.

Hum Genet. 2009 Apr;125(3):305-18. doi: 10.1007/s00439-009-0626-9. Epub 2009 Jan 29.

PMID:
19184112
7.

The 9p21 susceptibility locus for coronary artery disease and the severity of coronary atherosclerosis.

Chen SN, Ballantyne CM, Gotto AM Jr, Marian AJ.

BMC Cardiovasc Disord. 2009 Jan 27;9:3. doi: 10.1186/1471-2261-9-3.

8.

Accuracy of genome-wide imputation of untyped markers and impacts on statistical power for association studies.

Hao K, Chudin E, McElwee J, Schadt EE.

BMC Genet. 2009 Jun 16;10:27. doi: 10.1186/1471-2156-10-27.

9.

Haplotype sharing excludes canine orthologous Filaggrin locus in atopy in West Highland White Terriers.

Barros Roque J, O'Leary CA, Kyaw-Tanner M, Latter M, Mason K, Shipstone M, Vogelnest L, Duffy DL.

Anim Genet. 2009 Oct;40(5):793-4. doi: 10.1111/j.1365-2052.2009.01915.x. Epub 2009 May 20. No abstract available.

PMID:
19466940
10.

CTLA4 promoter polymorphisms are associated with canine diabetes mellitus.

Short AD, Saleh NM, Catchpole B, Kennedy LJ, Barnes A, Jones CA, Fretwell N, Ollier WE.

Tissue Antigens. 2010 Mar;75(3):242-52. doi: 10.1111/j.1399-0039.2009.01434.x.

PMID:
20196832
11.

Meta-analysis in genome-wide association datasets: strategies and application in Parkinson disease.

Evangelou E, Maraganore DM, Ioannidis JP.

PLoS One. 2007 Feb 7;2(2):e196.

12.

A case-control study of risk factors for canine atopic dermatitis among boxer, bullterrier and West Highland white terrier dogs in Sweden.

Nødtvedt A, Bergvall K, Sallander M, Egenvall A, Emanuelson U, Hedhammar A.

Vet Dermatol. 2007 Oct;18(5):309-15.

PMID:
17845618
13.

Estimation of heritability of atopic dermatitis in Labrador and Golden Retrievers.

Shaw SC, Wood JL, Freeman J, Littlewood JD, Hannant D.

Am J Vet Res. 2004 Jul;65(7):1014-20.

PMID:
15281664
14.

Genome-wide association study identifies a novel canine glaucoma locus.

Ahonen SJ, Pietilä E, Mellersh CS, Tiira K, Hansen L, Johnson GS, Lohi H.

PLoS One. 2013 Aug 7;8(8):e70903. doi: 10.1371/journal.pone.0070903. eCollection 2013.

15.

Examination of the current top candidate genes for AD in a genome-wide association study.

Feulner TM, Laws SM, Friedrich P, Wagenpfeil S, Wurst SH, Riehle C, Kuhn KA, Krawczak M, Schreiber S, Nikolaus S, Förstl H, Kurz A, Riemenschneider M.

Mol Psychiatry. 2010 Jul;15(7):756-66. doi: 10.1038/mp.2008.141. Epub 2009 Jan 6.

PMID:
19125160
16.

Four SNPs on chromosome 9p21 in a South Korean population implicate a genetic locus that confers high cross-race risk for development of coronary artery disease.

Shen GQ, Li L, Rao S, Abdullah KG, Ban JM, Lee BS, Park JE, Wang QK.

Arterioscler Thromb Vasc Biol. 2008 Feb;28(2):360-5. Epub 2007 Nov 29.

17.

Genome-wide association study identifies a new locus for coronary artery disease on chromosome 10p11.23.

Erdmann J, Willenborg C, Nahrstaedt J, Preuss M, König IR, Baumert J, Linsel-Nitschke P, Gieger C, Tennstedt S, Belcredi P, Aherrahrou Z, Klopp N, Loley C, Stark K, Hengstenberg C, Bruse P, Freyer J, Wagner AK, Medack A, Lieb W, Grosshennig A, Sager HB, Reinhardt A, Schäfer A, Schreiber S, El Mokhtari NE, Raaz-Schrauder D, Illig T, Garlichs CD, Ekici AB, Reis A, Schrezenmeir J, Rubin D, Ziegler A, Wichmann HE, Doering A, Meisinger C, Meitinger T, Peters A, Schunkert H.

Eur Heart J. 2011 Jan;32(2):158-68. doi: 10.1093/eurheartj/ehq405. Epub 2010 Nov 18.

18.

Assessing batch effects of genotype calling algorithm BRLMM for the Affymetrix GeneChip Human Mapping 500 K array set using 270 HapMap samples.

Hong H, Su Z, Ge W, Shi L, Perkins R, Fang H, Xu J, Chen JJ, Han T, Kaput J, Fuscoe JC, Tong W.

BMC Bioinformatics. 2008 Aug 12;9 Suppl 9:S17. doi: 10.1186/1471-2105-9-S9-S17.

19.

Evaluating variations of genotype calling: a potential source of spurious associations in genome-wide association studies.

Hong H, Su Z, Ge W, Shi L, Perkins R, Fang H, Mendrick D, Tong W.

J Genet. 2010 Apr;89(1):55-64.

20.

Genome-wide association study identifies single-nucleotide polymorphism in KCNB1 associated with left ventricular mass in humans: the HyperGEN Study.

Arnett DK, Li N, Tang W, Rao DC, Devereux RB, Claas SA, Kraemer R, Broeckel U.

BMC Med Genet. 2009 May 19;10:43. doi: 10.1186/1471-2350-10-43.

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