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Items: 1 to 20 of 81

1.

VE-PTP controls blood vessel development by balancing Tie-2 activity.

Winderlich M, Keller L, Cagna G, Broermann A, Kamenyeva O, Kiefer F, Deutsch U, Nottebaum AF, Vestweber D.

J Cell Biol. 2009 May 18;185(4):657-71. doi: 10.1083/jcb.200811159.

2.

Vascular endothelial cell-specific phosphotyrosine phosphatase (VE-PTP) activity is required for blood vessel development.

Bäumer S, Keller L, Holtmann A, Funke R, August B, Gamp A, Wolburg H, Wolburg-Buchholz K, Deutsch U, Vestweber D.

Blood. 2006 Jun 15;107(12):4754-62. Epub 2006 Mar 2.

3.
4.

VE-PTP and VE-cadherin ectodomains interact to facilitate regulation of phosphorylation and cell contacts.

Nawroth R, Poell G, Ranft A, Kloep S, Samulowitz U, Fachinger G, Golding M, Shima DT, Deutsch U, Vestweber D.

EMBO J. 2002 Sep 16;21(18):4885-95. Erratum in: EMBO J. 2005 Sep 7;24(17):3158.

5.

How T cells trigger the dissociation of the endothelial receptor phosphatase VE-PTP from VE-cadherin.

Vockel M, Vestweber D.

Blood. 2013 Oct 3;122(14):2512-22. doi: 10.1182/blood-2013-04-499228. Epub 2013 Aug 1.

6.

VE-PTP regulates VEGFR2 activity in stalk cells to establish endothelial cell polarity and lumen formation.

Hayashi M, Majumdar A, Li X, Adler J, Sun Z, Vertuani S, Hellberg C, Mellberg S, Koch S, Dimberg A, Koh GY, Dejana E, Belting HG, Affolter M, Thurston G, Holmgren L, Vestweber D, Claesson-Welsh L.

Nat Commun. 2013;4:1672. doi: 10.1038/ncomms2683.

7.

Contact inhibition of VEGF-induced proliferation requires vascular endothelial cadherin, beta-catenin, and the phosphatase DEP-1/CD148.

Grazia Lampugnani M, Zanetti A, Corada M, Takahashi T, Balconi G, Breviario F, Orsenigo F, Cattelino A, Kemler R, Daniel TO, Dejana E.

J Cell Biol. 2003 May 26;161(4):793-804.

8.

Inhibition of hypoxia-inducible factor activity in endothelial cells disrupts embryonic cardiovascular development.

Licht AH, Müller-Holtkamp F, Flamme I, Breier G.

Blood. 2006 Jan 15;107(2):584-90. Epub 2005 Sep 27.

9.

VE-PTP maintains the endothelial barrier via plakoglobin and becomes dissociated from VE-cadherin by leukocytes and by VEGF.

Nottebaum AF, Cagna G, Winderlich M, Gamp AC, Linnepe R, Polaschegg C, Filippova K, Lyck R, Engelhardt B, Kamenyeva O, Bixel MG, Butz S, Vestweber D.

J Exp Med. 2008 Nov 24;205(12):2929-45. doi: 10.1084/jem.20080406. Epub 2008 Nov 17.

10.

Dissociation of VE-PTP from VE-cadherin is required for leukocyte extravasation and for VEGF-induced vascular permeability in vivo.

Broermann A, Winderlich M, Block H, Frye M, Rossaint J, Zarbock A, Cagna G, Linnepe R, Schulte D, Nottebaum AF, Vestweber D.

J Exp Med. 2011 Nov 21;208(12):2393-401. doi: 10.1084/jem.20110525. Epub 2011 Oct 24.

11.

Promotion of cell spreading and migration by vascular endothelial-protein tyrosine phosphatase (VE-PTP) in cooperation with integrins.

Mori M, Murata Y, Kotani T, Kusakari S, Ohnishi H, Saito Y, Okazawa H, Ishizuka T, Mori M, Matozaki T.

J Cell Physiol. 2010 Jul;224(1):195-204. doi: 10.1002/jcp.22122.

PMID:
20301196
12.

Tyrosine phosphatase inhibition augments collateral blood flow in a rat model of peripheral vascular disease.

Carr AN, Davis MG, Eby-Wilkens E, Howard BW, Towne BA, Dufresne TE, Peters KG.

Am J Physiol Heart Circ Physiol. 2004 Jul;287(1):H268-76. Epub 2004 Feb 26.

13.

Targeted null-mutation in the vascular endothelial-cadherin gene impairs the organization of vascular-like structures in embryoid bodies.

Vittet D, Buchou T, Schweitzer A, Dejana E, Huber P.

Proc Natl Acad Sci U S A. 1997 Jun 10;94(12):6273-8.

14.

N-cadherin acts upstream of VE-cadherin in controlling vascular morphogenesis.

Luo Y, Radice GL.

J Cell Biol. 2005 Apr 11;169(1):29-34. Epub 2005 Apr 4.

15.

Zebrafish Tie-2 shares a redundant role with Tie-1 in heart development and regulates vessel integrity.

Gjini E, Hekking LH, Küchler A, Saharinen P, Wienholds E, Post JA, Alitalo K, Schulte-Merker S.

Dis Model Mech. 2011 Jan;4(1):57-66. doi: 10.1242/dmm.005033. Epub 2010 Nov 2.

16.

VE-cadherin is not required for the formation of nascent blood vessels but acts to prevent their disassembly.

Crosby CV, Fleming PA, Argraves WS, Corada M, Zanetta L, Dejana E, Drake CJ.

Blood. 2005 Apr 1;105(7):2771-6. Epub 2004 Dec 16.

17.

Vascular endothelial tyrosine phosphatase (VE-PTP)-null mice undergo vasculogenesis but die embryonically because of defects in angiogenesis.

Dominguez MG, Hughes VC, Pan L, Simmons M, Daly C, Anderson K, Noguera-Troise I, Murphy AJ, Valenzuela DM, Davis S, Thurston G, Yancopoulos GD, Gale NW.

Proc Natl Acad Sci U S A. 2007 Feb 27;104(9):3243-8. Epub 2007 Feb 21.

18.

Src kinase phosphorylates vascular endothelial-cadherin in response to vascular endothelial growth factor: identification of tyrosine 685 as the unique target site.

Wallez Y, Cand F, Cruzalegui F, Wernstedt C, Souchelnytskyi S, Vilgrain I, Huber P.

Oncogene. 2007 Feb 15;26(7):1067-77. Epub 2006 Aug 14.

PMID:
16909109
19.

Molecular cloning and expression of murine vascular endothelial-cadherin in early stage development of cardiovascular system.

Breier G, Breviario F, Caveda L, Berthier R, Schnürch H, Gotsch U, Vestweber D, Risau W, Dejana E.

Blood. 1996 Jan 15;87(2):630-41.

20.

Transcriptional profiling reveals a critical role for tyrosine phosphatase VE-PTP in regulation of VEGFR2 activity and endothelial cell morphogenesis.

Mellberg S, Dimberg A, Bahram F, Hayashi M, Rennel E, Ameur A, Westholm JO, Larsson E, Lindahl P, Cross MJ, Claesson-Welsh L.

FASEB J. 2009 May;23(5):1490-502. doi: 10.1096/fj.08-123810. Epub 2009 Jan 9.

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