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Results: 1 to 20 of 117

1.

Neonatal neuronal circuitry shows hyperexcitable disturbance in a mouse model of the adult-onset neurodegenerative disease amyotrophic lateral sclerosis.

van Zundert B, Peuscher MH, Hynynen M, Chen A, Neve RL, Brown RH Jr, Constantine-Paton M, Bellingham MC.

J Neurosci. 2008 Oct 22;28(43):10864-74. doi: 10.1523/JNEUROSCI.1340-08.2008.

PMID:
18945894
[PubMed - indexed for MEDLINE]
Free PMC Article
2.

Embryonic alteration of motoneuronal morphology induces hyperexcitability in the mouse model of amyotrophic lateral sclerosis.

Martin E, Cazenave W, Cattaert D, Branchereau P.

Neurobiol Dis. 2013 Jun;54:116-26. doi: 10.1016/j.nbd.2013.02.011. Epub 2013 Mar 4. Erratum in: Neurobiol Dis. 2013 Jul;55:63.

PMID:
23466698
[PubMed - indexed for MEDLINE]
3.

Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.

Jaarsma D, Haasdijk ED, Grashorn JA, Hawkins R, van Duijn W, Verspaget HW, London J, Holstege JC.

Neurobiol Dis. 2000 Dec;7(6 Pt B):623-43.

PMID:
11114261
[PubMed - indexed for MEDLINE]
4.

Inhibitory synaptic regulation of motoneurons: a new target of disease mechanisms in amyotrophic lateral sclerosis.

Martin LJ, Chang Q.

Mol Neurobiol. 2012 Feb;45(1):30-42. doi: 10.1007/s12035-011-8217-x. Epub 2011 Nov 10. Review.

PMID:
22072396
[PubMed - indexed for MEDLINE]
Free PMC Article
5.

Increased persistent sodium current determines cortical hyperexcitability in a genetic model of amyotrophic lateral sclerosis.

Pieri M, Carunchio I, Curcio L, Mercuri NB, Zona C.

Exp Neurol. 2009 Feb;215(2):368-79. doi: 10.1016/j.expneurol.2008.11.002. Epub 2008 Nov 21.

PMID:
19071115
[PubMed - indexed for MEDLINE]
6.

eGFP expression under UCHL1 promoter genetically labels corticospinal motor neurons and a subpopulation of degeneration-resistant spinal motor neurons in an ALS mouse model.

Yasvoina MV, Genç B, Jara JH, Sheets PL, Quinlan KA, Milosevic A, Shepherd GM, Heckman CJ, Özdinler PH.

J Neurosci. 2013 May 1;33(18):7890-904. doi: 10.1523/JNEUROSCI.2787-12.2013.

PMID:
23637180
[PubMed - indexed for MEDLINE]
Free PMC Article
7.

Early excitability changes in lumbar motoneurons of transgenic SOD1G85R and SOD1G(93A-Low) mice.

Pambo-Pambo A, Durand J, Gueritaud JP.

J Neurophysiol. 2009 Dec;102(6):3627-42. doi: 10.1152/jn.00482.2009. Epub 2009 Oct 14.

PMID:
19828728
[PubMed - indexed for MEDLINE]
Free Article
8.

Progressive changes in synaptic inputs to motoneurons in adult sacral spinal cord of a mouse model of amyotrophic lateral sclerosis.

Jiang M, Schuster JE, Fu R, Siddique T, Heckman CJ.

J Neurosci. 2009 Dec 2;29(48):15031-8. doi: 10.1523/JNEUROSCI.0574-09.2009.

PMID:
19955354
[PubMed - indexed for MEDLINE]
Free PMC Article
9.

Over-expression of N-type calcium channels in cortical neurons from a mouse model of Amyotrophic Lateral Sclerosis.

Pieri M, Caioli S, Canu N, Mercuri NB, Guatteo E, Zona C.

Exp Neurol. 2013 Sep;247:349-58. doi: 10.1016/j.expneurol.2012.11.002. Epub 2012 Nov 8.

PMID:
23142186
[PubMed - indexed for MEDLINE]
10.

Hyperexcitability of cultured spinal motoneurons from presymptomatic ALS mice.

Kuo JJ, Schonewille M, Siddique T, Schults AN, Fu R, Bär PR, Anelli R, Heckman CJ, Kroese AB.

J Neurophysiol. 2004 Jan;91(1):571-5. Epub 2003 Oct 1.

PMID:
14523070
[PubMed - indexed for MEDLINE]
Free Article
11.

Transient recovery in a rat model of familial amyotrophic lateral sclerosis after transplantation of motor neurons derived from mouse embryonic stem cells.

López-González R, Kunckles P, Velasco I.

Cell Transplant. 2009;18(10):1171-81. doi: 10.3727/096368909X12483162197123. Epub 2009 Aug 5.

PMID:
19660174
[PubMed - indexed for MEDLINE]
12.

Early electrophysiological abnormalities in lumbar motoneurons in a transgenic mouse model of amyotrophic lateral sclerosis.

Bories C, Amendola J, Lamotte d'Incamps B, Durand J.

Eur J Neurosci. 2007 Jan;25(2):451-9.

PMID:
17284186
[PubMed - indexed for MEDLINE]
13.

Glycine receptor channels in spinal motoneurons are abnormal in a transgenic mouse model of amyotrophic lateral sclerosis.

Chang Q, Martin LJ.

J Neurosci. 2011 Feb 23;31(8):2815-27. doi: 10.1523/JNEUROSCI.2475-10.2011.

PMID:
21414903
[PubMed - indexed for MEDLINE]
Free PMC Article
14.

GAB(A) receptors present higher affinity and modified subunit composition in spinal motor neurons from a genetic model of amyotrophic lateral sclerosis.

Carunchio I, Mollinari C, Pieri M, Merlo D, Zona C.

Eur J Neurosci. 2008 Oct;28(7):1275-85. doi: 10.1111/j.1460-9568.2008.06436.x.

PMID:
18973555
[PubMed - indexed for MEDLINE]
15.

Early signs of motoneuron vulnerability in a disease model system: Characterization of transverse slice cultures of spinal cord isolated from embryonic ALS mice.

Avossa D, Grandolfo M, Mazzarol F, Zatta M, Ballerini L.

Neuroscience. 2006;138(4):1179-94. Epub 2006 Jan 25.

PMID:
16442737
[PubMed - indexed for MEDLINE]
16.

Overexpression of human mutated G93A SOD1 changes dynamics of the ER mitochondria calcium cycle specifically in mouse embryonic motor neurons.

Lautenschläger J, Prell T, Ruhmer J, Weidemann L, Witte OW, Grosskreutz J.

Exp Neurol. 2013 Sep;247:91-100. doi: 10.1016/j.expneurol.2013.03.027. Epub 2013 Apr 8.

PMID:
23578819
[PubMed - indexed for MEDLINE]
17.

Nuclear localization of human SOD1 and mutant SOD1-specific disruption of survival motor neuron protein complex in transgenic amyotrophic lateral sclerosis mice.

Gertz B, Wong M, Martin LJ.

J Neuropathol Exp Neurol. 2012 Feb;71(2):162-77. doi: 10.1097/NEN.0b013e318244b635.

PMID:
22249462
[PubMed - indexed for MEDLINE]
Free PMC Article
18.

Early pathogenesis in the adult-onset neurodegenerative disease amyotrophic lateral sclerosis.

van Zundert B, Izaurieta P, Fritz E, Alvarez FJ.

J Cell Biochem. 2012 Nov;113(11):3301-12. doi: 10.1002/jcb.24234.

PMID:
22740507
[PubMed - indexed for MEDLINE]
Free PMC Article
19.

Autophagy activation and neuroprotection by progesterone in the G93A-SOD1 transgenic mouse model of amyotrophic lateral sclerosis.

Kim J, Kim TY, Cho KS, Kim HN, Koh JY.

Neurobiol Dis. 2013 Nov;59:80-5. doi: 10.1016/j.nbd.2013.07.011. Epub 2013 Jul 26.

PMID:
23891729
[PubMed - indexed for MEDLINE]
20.

Zinc pre-treatment enhances NMDAR-mediated excitotoxicity in cultured cortical neurons from SOD1(G93A) mouse, a model of amyotrophic lateral sclerosis.

Nutini M, Frazzini V, Marini C, Spalloni A, Sensi SL, Longone P.

Neuropharmacology. 2011 Jun;60(7-8):1200-8. doi: 10.1016/j.neuropharm.2010.11.001. Epub 2010 Nov 5.

PMID:
21056589
[PubMed - indexed for MEDLINE]

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