Similar articles for PMID: 18393801

Year	Number of Results
1998	1
1999	2
2000	1
2001	2
2002	1
2003	2
2004	4
2005	3
2006	3
2007	8
2008	3
2009	5
2010	3
2011	11
2012	13
2013	13
2014	10
2015	4
2016	5
2017	8
2018	6
2019	8
2020	3
2021	2
2022	7
2023	3
2024	0

1

Epsilon-secretase: reduction of amyloid precursor protein epsilon-site cleavage in Alzheimer's disease.

Kametani F. Kametani F. Curr Alzheimer Res. 2008 Apr;5(2):165-71. doi: 10.2174/156720508783954776. Curr Alzheimer Res. 2008. PMID: 18393801 Review.

2

Abeta 11-40/42 production without gamma-secretase epsilon-site cleavage.

Kume H, Kametani F. Kume H, et al. Biochem Biophys Res Commun. 2006 Nov 3;349(4):1356-60. doi: 10.1016/j.bbrc.2006.08.181. Epub 2006 Sep 11. Biochem Biophys Res Commun. 2006. PMID: 16979587

3

Intracellular domain generation of amyloid precursor protein by epsilon-cleavage depends on C-terminal fragment by alpha-secretase cleavage.

Kume H, Maruyama K, Kametani F. Kume H, et al. Int J Mol Med. 2004 Jan;13(1):121-5. Int J Mol Med. 2004. PMID: 14654982

4

Amyloid-β protein (Aβ) Glu11 is the major β-secretase site of β-site amyloid-β precursor protein-cleaving enzyme 1(BACE1), and shifting the cleavage site to Aβ Asp1 contributes to Alzheimer pathogenesis.

Deng Y, Wang Z, Wang R, Zhang X, Zhang S, Wu Y, Staufenbiel M, Cai F, Song W. Deng Y, et al. Eur J Neurosci. 2013 Jun;37(12):1962-9. doi: 10.1111/ejn.12235. Eur J Neurosci. 2013. PMID: 23773065

5

Lysine 624 of the amyloid precursor protein (APP) is a critical determinant of amyloid β peptide length: support for a sequential model of γ-secretase intramembrane proteolysis and regulation by the amyloid β precursor protein (APP) juxtamembrane region.

Kukar TL, Ladd TB, Robertson P, Pintchovski SA, Moore B, Bann MA, Ren Z, Jansen-West K, Malphrus K, Eggert S, Maruyama H, Cottrell BA, Das P, Basi GS, Koo EH, Golde TE. Kukar TL, et al. J Biol Chem. 2011 Nov 18;286(46):39804-12. doi: 10.1074/jbc.M111.274696. Epub 2011 Aug 25. J Biol Chem. 2011. PMID: 21868378 Free PMC article.

6

Autosomal-dominant Alzheimer's disease mutations at the same codon of amyloid precursor protein differentially alter Aβ production.

Suárez-Calvet M, Belbin O, Pera M, Badiola N, Magrané J, Guardia-Laguarta C, Muñoz L, Colom-Cadena M, Clarimón J, Lleó A. Suárez-Calvet M, et al. J Neurochem. 2014 Jan;128(2):330-9. doi: 10.1111/jnc.12466. Epub 2013 Oct 24. J Neurochem. 2014. PMID: 24117942 Free article.

7

The C-terminal fragment of the Alzheimer's disease amyloid protein precursor is degraded by a proteasome-dependent mechanism distinct from gamma-secretase.

Nunan J, Shearman MS, Checler F, Cappai R, Evin G, Beyreuther K, Masters CL, Small DH. Nunan J, et al. Eur J Biochem. 2001 Oct;268(20):5329-36. doi: 10.1046/j.0014-2956.2001.02465.x. Eur J Biochem. 2001. PMID: 11606195 Free article.

8

The physical approximation of APP and BACE-1: A key event in alzheimer's disease pathogenesis.

Sun J, Roy S. Sun J, et al. Dev Neurobiol. 2018 Mar;78(3):340-347. doi: 10.1002/dneu.22556. Epub 2017 Nov 13. Dev Neurobiol. 2018. PMID: 29106038 Free PMC article. Review.

9

Proteolytic processing of the Alzheimer's disease amyloid precursor protein in brain and platelets.

Evin G, Zhu A, Holsinger RM, Masters CL, Li QX. Evin G, et al. J Neurosci Res. 2003 Nov 1;74(3):386-92. doi: 10.1002/jnr.10745. J Neurosci Res. 2003. PMID: 14598315

10

gamma-Secretase can cleave amyloid precursor protein fragments independent of alpha- and beta-secretase pre-cutting.

Kume H, Sekijima Y, Maruyama K, Kametani F. Kume H, et al. Int J Mol Med. 2003 Jul;12(1):57-60. Int J Mol Med. 2003. PMID: 12792809

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