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Items: 1 to 20 of 169

1.

Gallstones containing bacteria are biofilms: bacterial slime production and ability to form pigment solids determines infection severity and bacteremia.

Stewart L, Griffiss JM, Jarvis GA, Way LW.

J Gastrointest Surg. 2007 Aug;11(8):977-83; discussion 983-4.

PMID:
17546479
2.

Biliary bacterial factors determine the path of gallstone formation.

Stewart L, Grifiss JM, Jarvis GA, Way LW.

Am J Surg. 2006 Nov;192(5):598-603.

PMID:
17071191
3.

Pigment gallstone pathogenesis: slime production by biliary bacteria is more important than beta-glucuronidase production.

Stewart L, Ponce R, Oesterle AL, Griffiss JM, Way LW.

J Gastrointest Surg. 2000 Sep-Oct;4(5):547-53.

PMID:
11077333
4.

Pathogenesis of pigment gallstones in Western societies: the central role of bacteria.

Stewart L, Oesterle AL, Erdan I, Griffiss JM, Way LW.

J Gastrointest Surg. 2002 Nov-Dec;6(6):891-903; discussion 903-4.

PMID:
12504229
5.

Spectrum of gallstone disease in the veterans population.

Stewart L, Griffiss JM, Way LW.

Am J Surg. 2005 Nov;190(5):746-51.

PMID:
16226952
6.
7.

The association between body mass index and severe biliary infections: a multivariate analysis.

Stewart L, Griffiss JM, Jarvis GA, Way LW.

Am J Surg. 2012 Nov;204(5):574-9. doi: 10.1016/j.amjsurg.2012.07.002. Epub 2012 Aug 11.

PMID:
22892201
8.

Gram-negative bacteria killed by complement are associated with more severe biliary infections and produce more tumor necrosis factor-alpha in sera.

Stewart L, Oesterle AL, Griffiss JM, Jarvis GA, Aagaard B, Way LW.

Surgery. 2002 Aug;132(2):408-14.

PMID:
12219042
9.

Cholangitis: bacterial virulence factors that facilitate cholangiovenous reflux and tumor necrosis factor-alpha production.

Stewart L, Oesterle AL, Grifiss JM, Jarvis GA, Way LW.

J Gastrointest Surg. 2003 Feb;7(2):191-8; discussion 198-9.

PMID:
12600443
10.

Elderly patients have more severe biliary infections: influence of complement-killing and induction of TNFalpha production.

Stewart L, Grifiss JM, Jarvis GA, Way LW.

Surgery. 2008 Jan;143(1):103-12. Epub 2007 Dec 3.

PMID:
18154938
11.

Pigment gallstones form as a composite of bacterial microcolonies and pigment solids.

Stewart L, Smith AL, Pellegrini CA, Motson RW, Way LW.

Ann Surg. 1987 Sep;206(3):242-50.

12.
13.

[Relationship between pigment gallstone and intestinal barrier function: experiment with guinea pigs and clinical observations].

Wu SD, Su Y, Fan Y, Jin JZ, Zhang ZH.

Zhonghua Yi Xue Za Zhi. 2008 Jun 3;88(21):1498-502. Chinese.

PMID:
18953859
14.

Fewer infectious manifestations are induced by bacteria entrapped in cholesterol stones than by bacteria in brown pigment gallstone.

Cetta F, Dhamo A, Malagnino G, Cisternino F, AzzarĂ  A.

J Gastrointest Surg. 2008 May;12(5):988-90. doi: 10.1007/s11605-008-0466-2. Epub 2008 Mar 7. No abstract available.

PMID:
18324446
15.

Risk assessment of Pseudomonas aeruginosa in water.

Mena KD, Gerba CP.

Rev Environ Contam Toxicol. 2009;201:71-115. doi: 10.1007/978-1-4419-0032-6_3. Review.

PMID:
19484589
16.
17.

Beta-glucuronidase activity related to bacterial growth in common bile duct bile in gallstone patients.

Skar V, Skar AG, Strømme JH.

Scand J Gastroenterol. 1988 Jan;23(1):83-90.

PMID:
3344403
18.

Possible relationship between intestinal barrier function and formation of pigment gallstones in hamsters.

Fan Y, Wu SD, Sun L, Fu BB, Su Y.

Hepatobiliary Pancreat Dis Int. 2008 Oct;7(5):529-32.

19.

Impact of slime dispersants and anti-adhesives on in vitro biofilm formation of Staphylococcus epidermidis on intraocular lenses and on antibiotic activities.

Kadry AA, Fouda SI, Shibl AM, Abu El-Asrar AA.

J Antimicrob Chemother. 2009 Mar;63(3):480-4. doi: 10.1093/jac/dkn533. Epub 2009 Jan 14.

20.

Bile anaerobic bacteria detection and antibiotic susceptibility in patients with gallstone.

Lu Y, Xiang TH, Shi JS, Zhang BY.

Hepatobiliary Pancreat Dis Int. 2003 Aug;2(3):431-4.

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