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Items: 1 to 20 of 985

1.

Viral suppression and immune restoration in the gastrointestinal mucosa of human immunodeficiency virus type 1-infected patients initiating therapy during primary or chronic infection.

Guadalupe M, Sankaran S, George MD, Reay E, Verhoeven D, Shacklett BL, Flamm J, Wegelin J, Prindiville T, Dandekar S.

J Virol. 2006 Aug;80(16):8236-47.

3.

Effective CD4+ T-cell restoration in gut-associated lymphoid tissue of HIV-infected patients is associated with enhanced Th17 cells and polyfunctional HIV-specific T-cell responses.

Macal M, Sankaran S, Chun TW, Reay E, Flamm J, Prindiville TJ, Dandekar S.

Mucosal Immunol. 2008 Nov;1(6):475-88. doi: 10.1038/mi.2008.35. Epub 2008 Sep 10.

PMID:
19079215
4.

Gut mucosal T cell responses and gene expression correlate with protection against disease in long-term HIV-1-infected nonprogressors.

Sankaran S, Guadalupe M, Reay E, George MD, Flamm J, Prindiville T, Dandekar S.

Proc Natl Acad Sci U S A. 2005 Jul 12;102(28):9860-5. Epub 2005 Jun 24.

5.

Trafficking of human immunodeficiency virus type 1-specific CD8+ T cells to gut-associated lymphoid tissue during chronic infection.

Shacklett BL, Cox CA, Sandberg JK, Stollman NH, Jacobson MA, Nixon DF.

J Virol. 2003 May;77(10):5621-31.

6.

Pathogenesis of HIV in the gastrointestinal tract.

Dandekar S.

Curr HIV/AIDS Rep. 2007 Feb;4(1):10-5. Review.

PMID:
17338855
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10.

HIV-1-infected children on HAART: immunologic features of three different levels of viral suppression.

Zaccarelli-Filho CA, Ono E, Machado DM, Brunialti M, Succi RC, Salomão R, Kallás EG, de Moraes-Pinto MI.

Cytometry B Clin Cytom. 2007 Jan 15;72(1):14-21.

12.

Early initiation of highly active antiretroviral therapy fails to reverse immunovirological abnormalities in gut-associated lymphoid tissue induced by acute HIV infection.

Tincati C, Biasin M, Bandera A, Violin M, Marchetti G, Piacentini L, Vago GL, Balotta C, Moroni M, Franzetti F, Clerici M, Gori A.

Antivir Ther. 2009;14(3):321-30.

PMID:
19474466
13.

Differential upregulation of CD38 on different T-cell subsets may influence the ability to reconstitute CD4+ T cells under successful highly active antiretroviral therapy.

Benito JM, López M, Lozano S, Ballesteros C, Martinez P, González-Lahoz J, Soriano V.

J Acquir Immune Defic Syndr. 2005 Apr 1;38(4):373-81.

PMID:
15764953
14.

CCR5 and CXCR4 expression on memory and naive T cells in HIV-1 infection and response to highly active antiretroviral therapy.

Nicholson JK, Browning SW, Hengel RL, Lew E, Gallagher LE, Rimland D, McDougal JS.

J Acquir Immune Defic Syndr. 2001 Jun 1;27(2):105-15.

PMID:
11404531
15.

Reconstitution of EBV latent but not lytic antigen-specific CD4+ and CD8+ T cells after HIV treatment with highly active antiretroviral therapy.

Piriou E, Jansen CA, van Dort K, De Cuyper I, Nanlohy NM, Lange JM, van Oers MH, Miedema F, van Baarle D.

J Immunol. 2005 Aug 1;175(3):2010-7.

16.

Age-related immune reconstitution during highly active antiretroviral therapy in human immunodeficiency virus type 1-infected children.

Hainaut M, Ducarme M, Schandene L, Peltier CA, Marissens D, Zissis G, Mascart F, Levy J.

Pediatr Infect Dis J. 2003 Jan;22(1):62-9.

PMID:
12544411
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18.

Incomplete CD4 T cell recovery in HIV-1 infection after 12 months of highly active antiretroviral therapy is associated with ongoing increased CD4 T cell activation and turnover.

Anthony KB, Yoder C, Metcalf JA, DerSimonian R, Orenstein JM, Stevens RA, Falloon J, Polis MA, Lane HC, Sereti I.

J Acquir Immune Defic Syndr. 2003 Jun 1;33(2):125-33.

PMID:
12794543
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