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Items: 1 to 20 of 281

1.

Antioxidant vitamin therapy alters sepsis-related apoptotic myocardial activity and inflammatory responses.

Carlson D, Maass DL, White DJ, Tan J, Horton JW.

Am J Physiol Heart Circ Physiol. 2006 Dec;291(6):H2779-89. Epub 2006 Jul 14.

2.

Antioxidant vitamin therapy alters burn trauma-mediated cardiac NF-kappaB activation and cardiomyocyte cytokine secretion.

Horton JW, White DJ, Maass DL, Hybki DP, Haudek S, Giroir B.

J Trauma. 2001 Mar;50(3):397-406; discussion 407-8.

PMID:
11265018
3.

Selective decontamination of the digestive tract attenuated the myocardial inflammation and dysfunction that occur with burn injury.

Horton JW, Tan J, White DJ, Maass DL, Thomas JA.

Am J Physiol Heart Circ Physiol. 2004 Nov;287(5):H2241-51. Epub 2004 Jul 15.

4.

Sodium/hydrogen exchange activity in sepsis and in sepsis complicated by previous injury: 31P and 23Na NMR study.

Sikes PJ, Zhao P, Maass DL, White J, Horton JW.

Crit Care Med. 2005 Mar;33(3):605-15.

PMID:
15753754
5.
6.

Cardiac effects of burn injury complicated by aspiration pneumonia-induced sepsis.

White J, Thomas J, Maass DL, Horton JW.

Am J Physiol Heart Circ Physiol. 2003 Jul;285(1):H47-58. Epub 2003 Mar 13.

8.

Cardiac mitochondrial damage and inflammation responses in sepsis.

Zang Q, Maass DL, Tsai SJ, Horton JW.

Surg Infect (Larchmt). 2007 Feb;8(1):41-54.

PMID:
17381396
9.

Role of cytosolic vs. mitochondrial Ca2+ accumulation in burn injury-related myocardial inflammation and function.

Maass DL, White J, Sanders B, Horton JW.

Am J Physiol Heart Circ Physiol. 2005 Feb;288(2):H744-51. Epub 2004 Sep 23.

10.
11.

Specific inhibition of mitochondrial oxidative stress suppresses inflammation and improves cardiac function in a rat pneumonia-related sepsis model.

Zang QS, Sadek H, Maass DL, Martinez B, Ma L, Kilgore JA, Williams NS, Frantz DE, Wigginton JG, Nwariaku FE, Wolf SE, Minei JP.

Am J Physiol Heart Circ Physiol. 2012 May 1;302(9):H1847-59. doi: 10.1152/ajpheart.00203.2011. Epub 2012 Mar 9.

12.

The effects of early excision and grafting on myocardial inflammation and function after burn injury.

Horton JW, Sanders B, White DJ, Maass DL.

J Trauma. 2006 Nov;61(5):1069-77.

PMID:
17099511
13.

Rho-associated kinase modulates myocardial inflammatory cytokine responses.

Horton JW, Maass DL, Ballard-Croft C.

Shock. 2005 Jul;24(1):53-8.

PMID:
15988321
14.

Acetaldehyde-induced cardiac contractile dysfunction may be alleviated by vitamin B1 but not by vitamins B6 or B12.

Aberle NS 2nd, Burd L, Zhao BH, Ren J.

Alcohol Alcohol. 2004 Sep-Oct;39(5):450-4. Epub 2004 Aug 10.

15.

Molecular or pharmacologic inhibition of the CD14 signaling pathway protects against burn-related myocardial inflammation and dysfunction.

Barber RC, Maass DL, White DJ, Chang LY, Horton JW.

Shock. 2008 Dec;30(6):705-13. doi: 10.1097/SHK.0b013e31816f6caa.

PMID:
18461018
16.

Mitochondrial ROS Induces Cardiac Inflammation via a Pathway through mtDNA Damage in a Pneumonia-Related Sepsis Model.

Yao X, Carlson D, Sun Y, Ma L, Wolf SE, Minei JP, Zang QS.

PLoS One. 2015 Oct 8;10(10):e0139416. doi: 10.1371/journal.pone.0139416. eCollection 2015.

17.

Caspase inhibition reduces cardiac myocyte dyshomeostasis and improves cardiac contractile function after major burn injury.

Carlson DL, Maass DL, White J, Sikes P, Horton JW.

J Appl Physiol (1985). 2007 Jul;103(1):323-30. Epub 2007 Apr 12.

18.

Aspiration pneumonia-induced sepsis increases cardiac dysfunction after burn trauma.

Sheeran PW, Maass DL, White DJ, Turbeville TD, Giroir BP, Horton JW.

J Surg Res. 1998 May;76(2):192-9.

PMID:
9698522
19.

Cardiomyocyte intracellular calcium and cardiac dysfunction after burn trauma.

White DJ, Maass DL, Sanders B, Horton JW.

Crit Care Med. 2002 Jan;30(1):14-22.

PMID:
11902254
20.

Nitric oxide donors alter cardiomyocyte cytokine secretion and cardiac function.

Maass DL, White J, Horton JW.

Crit Care Med. 2005 Dec;33(12):2794-803.

PMID:
16352962
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