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Items: 1 to 20 of 118


Recurrent patterns of dual RB and p53 pathway inactivation in melanoma.

Yang G, Rajadurai A, Tsao H.

J Invest Dermatol. 2005 Dec;125(6):1242-51.


Simultaneous aberrations of single CDKN2A network components and a high Rb phosphorylation status can differentiate subgroups of primary cutaneous B-cell lymphomas.

Kaune KM, Neumann C, Hallermann C, Haller F, Schön MP, Middel P.

Exp Dermatol. 2011 Apr;20(4):331-5. doi: 10.1111/j.1600-0625.2010.01226.x.


Dual inactivation of RB and p53 pathways in RAS-induced melanomas.

Bardeesy N, Bastian BC, Hezel A, Pinkel D, DePinho RA, Chin L.

Mol Cell Biol. 2001 Mar;21(6):2144-53.


Chimeras of p14ARF and p16: functional hybrids with the ability to arrest growth.

Williams RT, Barnhill LM, Kuo HH, Lin WD, Batova A, Yu AL, Diccianni MB.

PLoS One. 2014 Feb 5;9(2):e88219. doi: 10.1371/journal.pone.0088219. eCollection 2014.


Loss of CDKN2A and p14ARF expression occurs frequently in human nonmelanoma skin cancers.

Pacifico A, Goldberg LH, Peris K, Chimenti S, Leone G, Ananthaswamy HN.

Br J Dermatol. 2008 Feb;158(2):291-7. Epub 2007 Dec 6. Erratum in: Br J Dermatol. 2008 Mar;158(3):657.


Inactivation of the INK4A/ARF locus frequently coexists with TP53 mutations in non-small cell lung cancer.

Sanchez-Cespedes M, Reed AL, Buta M, Wu L, Westra WH, Herman JG, Yang SC, Jen J, Sidransky D.

Oncogene. 1999 Oct 21;18(43):5843-9.


P16 UV mutations in human skin epithelial tumors.

Soufir N, Molès JP, Vilmer C, Moch C, Verola O, Rivet J, Tesniere A, Dubertret L, Basset-Seguin N.

Oncogene. 1999 Sep 23;18(39):5477-81.


Expression and significance of p53, rb, p21/waf-1, p16/ink-4a, and PTEN tumor suppressors in canine melanoma.

Koenig A, Bianco SR, Fosmire S, Wojcieszyn J, Modiano JF.

Vet Pathol. 2002 Jul;39(4):458-72.


Combined analysis of p53 and RB pathways in epithelial ovarian cancer.

Hashiguchi Y, Tsuda H, Yamamoto K, Inoue T, Ishiko O, Ogita S.

Hum Pathol. 2001 Sep;32(9):988-96.


INK4a-ARF alterations and p53 mutations in hepatocellular carcinomas.

Tannapfel A, Busse C, Weinans L, Benicke M, Katalinic A, Geissler F, Hauss J, Wittekind C.

Oncogene. 2001 Oct 25;20(48):7104-9.


CDKN2A and CDK4 mutation analysis in Italian melanoma-prone families: functional characterization of a novel CDKN2A germ line mutation.

Della Torre G, Pasini B, Frigerio S, Donghi R, Rovini D, Delia D, Peters G, Huot TJ, Bianchi-Scarra G, Lantieri F, Rodolfo M, Parmiani G, Pierotti MA.

Br J Cancer. 2001 Sep 14;85(6):836-44.


CDKN2A/p16 is inactivated in most melanoma cell lines.

Castellano M, Pollock PM, Walters MK, Sparrow LE, Down LM, Gabrielli BG, Parsons PG, Hayward NK.

Cancer Res. 1997 Nov 1;57(21):4868-75.


A melanoma-predisposing germline CDKN2A mutation with functional significance for both p16 and p14ARF.

Hashemi J, Lindström MS, Asker C, Platz A, Hansson J, Wiman KG.

Cancer Lett. 2002 Jun 28;180(2):211-21.


Deregulation of the TP53/p14ARF tumor suppressor pathway in low-grade diffuse astrocytomas and its influence on clinical course.

Watanabe T, Katayama Y, Yoshino A, Komine C, Yokoyama T.

Clin Cancer Res. 2003 Oct 15;9(13):4884-90.


CDKN2A gene deletions and loss of p16 expression occur in osteosarcomas that lack RB alterations.

Nielsen GP, Burns KL, Rosenberg AE, Louis DN.

Am J Pathol. 1998 Jul;153(1):159-63.


Use of human tissue to assess the oncogenic activity of melanoma-associated mutations.

Chudnovsky Y, Adams AE, Robbins PB, Lin Q, Khavari PA.

Nat Genet. 2005 Jul;37(7):745-9. Epub 2005 Jun 12.


p16/CDKN2 and CDK4 gene mutations in sporadic melanoma development and progression.

Piccinin S, Doglioni C, Maestro R, Vukosavljevic T, Gasparotto D, D'Orazi C, Boiocchi M.

Int J Cancer. 1997 Feb 20;74(1):26-30.


CDKN2A germ-line mutations in individuals with multiple cutaneous melanomas.

Hashemi J, Platz A, Ueno T, Stierner U, Ringborg U, Hansson J.

Cancer Res. 2000 Dec 15;60(24):6864-7.

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